P2X7-mediated increased intracellular calcium causes functional derangement in Schwann cells from rats with CMT1A neuropathy.
about
Neuronal and glial purinergic receptors functions in neuron development and brain diseaseHyccin, the molecule mutated in the leukodystrophy hypomyelination and congenital cataract (HCC), is a neuronal proteinCFTR-deficient pigs display peripheral nervous system defects at birth.The role of oxidative stress in degeneration of the neuromuscular junction in amyotrophic lateral sclerosisThe PMP22 gene and its related diseases.Specific marker expression and cell state of Schwann cells during culture in vitro.Up-regulation of P2X7 receptors mediating proliferation of Schwann cells after sciatic nerve injury.Dysregulated expression of death, stress and mitochondrion related genes in the sciatic nerve of presymptomatic SOD1(G93A) mouse model of Amyotrophic Lateral Sclerosis.Identification of drug modulators targeting gene-dosage disease CMT1A.Characterization of the structure and intermolecular interactions between the connexin 32 carboxyl-terminal domain and the protein partners synapse-associated protein 97 and calmodulin.CMTX Disorder and CamKinase.Neuronal activity in the hub of extrasynaptic Schwann cell-axon interactions.P2X7 receptor inhibition increases CNTF in the subventricular zone, but not neurogenesis or neuroprotection after stroke in adult mice.Murine therapeutic models for Charcot-Marie-Tooth (CMT) disease.Diadenosine homodinucleotide products of ADP-ribosyl cyclases behave as modulators of the purinergic receptor P2X7.Alternative Splicing in the Human PMP22 Gene: Implications in CMT1A Neuropathy.Sphingomyelin as a myelin biomarker in CSF of acquired demyelinating neuropathiesCaveats in the Established Understanding of CMT1A.The diadenosine homodinucleotide P18 improves in vitro myelination in experimental Charcot-Marie-Tooth type 1A.Role of connexin 32 hemichannels in the release of ATP from peripheral nerves.A dual role for Integrin α6β4 in modulating Hereditary Neuropathy with liability to Pressure Palsies.Purinergic signaling mediated by P2X7 receptors controls myelination in sciatic nerves.Myelin abnormality in Charcot-Marie-Tooth type 4J recapitulates features of acquired demyelination.Re-evaluation of neuronal P2X7 expression using novel mouse models and a P2X7-specific nanobody
P2860
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P2860
P2X7-mediated increased intracellular calcium causes functional derangement in Schwann cells from rats with CMT1A neuropathy.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
P2X7-mediated increased intrac ...... om rats with CMT1A neuropathy.
@en
P2X7-mediated increased intrac ...... om rats with CMT1A neuropathy.
@nl
type
label
P2X7-mediated increased intrac ...... om rats with CMT1A neuropathy.
@en
P2X7-mediated increased intrac ...... om rats with CMT1A neuropathy.
@nl
prefLabel
P2X7-mediated increased intrac ...... om rats with CMT1A neuropathy.
@en
P2X7-mediated increased intrac ...... om rats with CMT1A neuropathy.
@nl
P2093
P2860
P50
P356
P1476
P2X7-mediated increased intrac ...... om rats with CMT1A neuropathy.
@en
P2093
Angelo Schenone
Antonio De Flora
Federica Benvenuto
Fulvia Fiorese
Giovanna Basile
Iliana Moreschi
Lucilla Nobbio
P2860
P304
23146-23158
P356
10.1074/JBC.M109.027128
P407
P577
2009-06-22T00:00:00Z