Are increased frequency of macrophage-like and natural killer (NK) cells, together with high levels of NKT and CD4+CD25high T cells balancing activated CD8+ T cells, the key to control Chagas' disease morbidity?
about
Modulation of paraoxonases during infectious diseases and its potential impact on atherosclerosisModulation of immune response in experimental Chagas diseaseRegulatory T cells phenotype in different clinical forms of Chagas' diseaseIncreased CD4+/CD8+ double-positive T cells in chronic Chagasic patients.Trypanosoma cruzi-induced activation of functionally distinct αβ and γδ CD4- CD8- T cells in individuals with polar forms of Chagas' disease.Deficient regulatory T cell activity and low frequency of IL-17-producing T cells correlate with the extent of cardiomyopathy in human Chagas' disease.T lymphocytes from chagasic patients are activated but lack proliferative capacity and down-regulate CD28 and CD3ζ.Innate immune responses and antioxidant/oxidant imbalance are major determinants of human Chagas disease.Polymorphic sites at the immunoregulatory CTLA-4 gene are associated with chronic chagas disease and its clinical manifestations.Polyfunctional T cell responses in children in early stages of chronic Trypanosoma cruzi infection contrast with monofunctional responses of long-term infected adults.Cytokine profiling in Chagas disease: towards understanding the association with infecting Trypanosoma cruzi discrete typing units (a BENEFIT TRIAL sub-study).Killer Cell Immunoglobulin-like Receptors and Their HLA Ligands are Related with the Immunopathology of Chagas Disease.Phenotypic Features of Circulating Leukocytes from Non-human Primates Naturally Infected with Trypanosoma cruzi Resemble the Major Immunological Findings Observed in Human Chagas Disease.Association of the Functional MICA-129 Polymorphism With the Severity of Chronic Chagas Heart Disease.Lymphocyte subpopulations in myocardial infarction: a comparison between peripheral and intracoronary blood.Current concepts in immunoregulation and pathology of human Chagas disease.Innate immunity and regulatory T-cells in human Chagas disease: what must be understood?Blood Gene Signatures of Chagas Cardiomyopathy With or Without Ventricular Dysfunction.Cellular and genetic mechanisms involved in the generation of protective and pathogenic immune responses in human Chagas disease.Immunoregulatory actions of melatonin and zinc during chronic Trypanosoma cruzi infection.Depletion of regulatory T cells decreases cardiac parasitosis and inflammation in experimental Chagas disease.Increase in the expression of CD4 + CD25+ lymphocytic T cells in the indeterminate clinical form of human Chagas disease after stimulation with recombinant antigens of Trypanosoma cruzi.Identification of phenotypic markers of B cells from patients with Chagas disease.Benznidazole affects expression of Th1, Th17 and Treg cytokines during acute experimental Trypanosoma cruzi infection.Evaluation of CD4+CD25+ T lymphocyte response time kinetics in patients with chronic Chagas disease after in vitro stimulation with recombinant Trypanosoma cruzi antigens.Genetic susceptibility to Chagas disease: an overview about the infection and about the association between disease and the immune response genes.Distinct Treatment Outcomes of Antiparasitic Therapy in -Infected Children Is Associated With Early Changes in Cytokines, Chemokines, and T-Cell PhenotypesThe Unsolved Jigsaw Puzzle of the Immune Response in Chagas DiseaseDifferent parasite inocula determine the modulation of the immune response and outcome of experimental Trypanosoma cruzi infection
P2860
Q27004131-F04920C9-9777-4DA1-A7F0-24EAA5352339Q27022885-1F0B29EE-9F75-467D-A393-1169A7A85EAEQ33927456-30DC5450-B0EE-47F0-9413-78A00CFF6112Q34009094-725113B3-9C91-4AC8-A2B7-FD711A1120D0Q34177376-15EF3E1A-E1B0-4FE7-9AD7-B9AF4E5D0EC6Q34251395-49B8C099-1CB1-4E7F-A504-2697FB92E7C2Q34577261-D9E51511-D52A-469A-8AF1-D3363302CAFAQ34952422-3EE19CC7-53B3-45E7-96DD-47019D1A8DF1Q35036373-932B76BE-F059-4D1E-A5C0-F6AF41E1068CQ35071204-5615633E-E230-40A9-9DAA-010E3B9C7A24Q35114499-4C2EBBE0-479A-4CB0-A91A-14B177F96804Q35610533-D747F9A6-85FE-43FF-B927-B1AEECD1E800Q35903600-9BADF074-6B37-4201-8AD0-D8384907A2BEQ36094257-0866D719-04BA-46EB-806D-E46B582BE48FQ36339037-3AD4D838-EF31-4CE4-AAFE-B4FE307012CCQ37151780-8DD40847-5CD4-4CFD-870A-B53A1623EA3FQ37597066-C3712D41-ECF8-4FD7-95FF-5922FE1D1F33Q39003348-1F8E6529-B0AB-405C-94B5-E4163D508991Q41485954-D7D7F339-BB0D-40A8-A55D-991DE5566B28Q41528104-28F0AE5A-1CA0-42A8-BE23-BB6F2B55E1A3Q41552272-C356D0C6-2618-48EE-BC76-07EAA651670FQ41745518-65EDF4DE-9E0B-49F8-A869-8DD2E7D852C0Q45341357-733F9BA6-F78B-4C0D-BD5D-C4E53D78FF8CQ47093622-87D11EE9-3E03-4684-A673-736C98662F85Q50987616-FE66970C-DABA-4F60-9E3C-C51AB48B3CBFQ53792154-28611CFE-0CCC-4EB0-A996-DB91E77336C5Q57110653-574D1C18-863B-4C05-AF81-B2820EA62BD5Q58705710-E1EAD4BC-09C7-4D71-8F51-E4DBEBACC383Q58843342-04386C15-6410-4053-AC18-4AE742D1FAC7
P2860
Are increased frequency of macrophage-like and natural killer (NK) cells, together with high levels of NKT and CD4+CD25high T cells balancing activated CD8+ T cells, the key to control Chagas' disease morbidity?
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
Are increased frequency of mac ...... rol Chagas' disease morbidity?
@en
Are increased frequency of mac ...... rol Chagas' disease morbidity?
@nl
type
label
Are increased frequency of mac ...... rol Chagas' disease morbidity?
@en
Are increased frequency of mac ...... rol Chagas' disease morbidity?
@nl
prefLabel
Are increased frequency of mac ...... rol Chagas' disease morbidity?
@en
Are increased frequency of mac ...... rol Chagas' disease morbidity?
@nl
P2093
P2860
P1476
Are increased frequency of mac ...... rol Chagas' disease morbidity?
@en
P2093
A Teixeira-Carvalho
D M Vitelli-Avelar
J C P Dias
J D Borges
O A Martins-Filho
R Sathler-Avelar
S M Elói-Santos
P2860
P356
10.1111/J.1365-2249.2006.03123.X
P577
2006-07-01T00:00:00Z