Foxo transcription factors blunt cardiac hypertrophy by inhibiting calcineurin signaling - Wikidata - awgldk - TriplyDB

Foxo transcription factors blunt cardiac hypertrophy by inhibiting calcineurin signaling

Foxo transcription factors blunt cardiac hypertrophy by inhibiting calcineurin signaling

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Angiotensin II requires zinc and downregulation of the zinc transporters ZnT3 and ZnT10 to induce senescence of vascular smooth muscle cells Short sequence motifs, overrepresented in mammalian conserved non-coding sequences New Molecular Insights of Insulin in Diabetic Cardiomyopathy Redox regulation of FoxO transcription factors MAPK/JNK signalling: a potential autophagy regulation pathway Therapeutic targeting of autophagy: potential and concerns in treating cardiovascular disease The ubiquitin-proteasome system and cardiovascular disease Autophagy plays an essential role in mediating regression of hypertrophy during unloading of the heart Forkhead box transcription factor 1: role in the pathogenesis of diabetic cardiomyopathy FoxO1: a novel insight into its molecular mechanisms in the regulation of skeletal muscle differentiation and fiber type specification The miR-19a/b family positively regulates cardiomyocyte hypertrophy by targeting atrogin-1 and MuRF-1 Myosin regulatory light chain phosphorylation attenuates cardiac hypertrophy HER2 phosphorylation is maintained by a PKB negative feedback loop in response to anti-HER2 herceptin in breast cancer RNA binding protein QKI inhibits the ischemia/reperfusion-induced apoptosis in neonatal cardiomyocytes Transcription factor Foxo3a prevents apoptosis by regulating calcium through the apoptosis repressor with caspase recruitment domain Alpha B-crystallin suppresses pressure overload cardiac hypertrophy TGF-β1, released by myofibroblasts, differentially regulates transcription and function of sodium and potassium channels in adult rat ventricular myocytes Foxo3a inhibits cardiomyocyte hypertrophy through transactivating catalase Opposing roles of FoxP1 and Nfat3 in transcriptional control of cardiomyocyte hypertrophy PGC-1 alpha serine 570 phosphorylation and GCN5-mediated acetylation by angiotensin II drive catalase down-regulation and vascular hypertrophy PARP-inhibitor treatment prevents hypertension induced cardiac remodeling by favorable modulation of heat shock proteins, Akt-1/GSK-3β and several PKC isoforms.Elevated nuclear Foxo1 suppresses excitability of skeletal muscle fibers Activation of AMPK inhibits cardiomyocyte hypertrophy by modulating of the FOXO1/MuRF1 signaling pathway in vitro FOXO transcription factors: their clinical significance and regulation.Dynamic switch of negative feedback regulation in Drosophila Akt-TOR signaling.Cardiovascular actions of neurotrophins Cyclic GMP/PKG-dependent inhibition of TRPC6 channel activity and expression negatively regulates cardiomyocyte NFAT activation Novel mechanism of cardiac stress modulation by PDE5 inhibition Differential alterations in gene expression profiles contribute to time-dependent effects of nandrolone to prevent denervation atrophy.Metabolic and Biochemical Stressors in Diabetic Cardiomyopathy.TEAD1-dependent expression of the FoxO3a gene in mouse skeletal muscle.Molecular Signals Elicited by GPCR Agonists in Hypertension, Cardiovascular Remodeling: Are MMPs and ADAMs Elusive Therapeutic Targets?Whole genome siRNA cell-based screen links mitochondria to Akt signaling network through uncoupling of electron transport chain The CCAAT/enhancer binding protein beta (C/EBPbeta) cooperates with NFAT to control expression of the calcineurin regulatory protein RCAN1-4.TRPC1 channels are critical for hypertrophic signaling in the heart.Calcineurin and its regulator, RCAN1, confer time-of-day changes in susceptibility of the heart to ischemia/reperfusion Influence of gestational overfeeding on cardiac morphometry and hypertrophic protein markers in fetal sheep The FoxO family in cardiac function and dysfunction.Deletion of FoxO1 leads to shortening of QRS by increasing Na(+) channel activity through enhanced expression of both cardiac NaV1.5 and β3 subunit Reactive oxygen species suppress cardiac NaV1.5 expression through Foxo1.Cardiac-specific mindin overexpression attenuates cardiac hypertrophy via blocking AKT/GSK3β and TGF-β1-Smad signalling.

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Foxo transcription factors blunt cardiac hypertrophy by inhibiting calcineurin signaling

http://www.wikidata.org/entity/Q42500888

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2006 nî lūn-bûn

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2006年の論文

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年學術文章

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2006年學術文章

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2006年學術文章

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name

Foxo transcription factors blunt cardiac hypertrophy by inhibiting calcineurin signaling

@en

Foxo transcription factors blunt cardiac hypertrophy by inhibiting calcineurin signaling

@nl

type

label

Foxo transcription factors blunt cardiac hypertrophy by inhibiting calcineurin signaling

@en

Foxo transcription factors blunt cardiac hypertrophy by inhibiting calcineurin signaling

@nl

prefLabel

Foxo transcription factors blunt cardiac hypertrophy by inhibiting calcineurin signaling

@en

Foxo transcription factors blunt cardiac hypertrophy by inhibiting calcineurin signaling

@nl

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Foxo transcription factors blunt cardiac hypertrophy by inhibiting calcineurin signaling

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Asim Dey

http://www.w3.org/2001/XMLSchema#string

Beverly A Rothermel

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David J Morris

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Diego H Castrillon

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Guangrong Lu

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Joseph A Hill

http://www.w3.org/2001/XMLSchema#string

Jun Cheng

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Kambeez Berenji

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Misook Oh

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Na Wang

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P2860

Protein kinase cascades in the regulation of cardiac hypertrophy

Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy

Muscle-specific RING finger 1 is a bona fide ubiquitin ligase that degrades cardiac troponin I

Insulin-induced phosphorylation of FKHR (Foxo1) targets to proteasomal degradation

A calcineurin-dependent transcriptional pathway for cardiac hypertrophy

Multiple roles of the DSCR1 (Adapt78 or RCAN1) gene and its protein product calcipressin 1 (or RCAN1) in disease

Targeted inhibition of calcineurin prevents agonist-induced cardiomyocyte hypertrophy

Calcineurin is necessary for the maintenance but not embryonic development of slow muscle fibers.

Disruption of forkhead transcription factor (FOXO) family members in mice reveals their functional diversification

A small molecular activator of cardiac hypertrophy uncovered in a chemical screen for modifiers of the calcineurin signaling pathway

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