Insights from knock-out models concerning postischemic release of TNFalpha from isolated mouse hearts.
about
Adenosine A2A receptor activation reduces infarct size in the isolated, perfused mouse heart by inhibiting resident cardiac mast cell degranulation.Inhibition of matrix metalloproteinase activity prevents increases in myocardial tumor necrosis factor-alpha.Tumor necrosis factor-alpha produced in cardiomyocytes mediates a predominant myocardial inflammatory response to stretch in early volume overload.Protection from adverse myocardial remodeling secondary to chronic volume overload in mast cell deficient ratsAprotinin exacerbates left ventricular dysfunction after ischemia/reperfusion in mice lacking tumor necrosis factor receptor I.ETA selective receptor antagonism prevents ventricular remodeling in volume-overloaded rats.SPECT imaging of inflammatory response in ischemic-reperfused rat hearts using a 99mTc-labeled dual-domain cytokine ligand.TNF-alpha inhibition attenuates adverse myocardial remodeling in a rat model of volume overload.Conditional deletion of cardiomyocyte peroxisome proliferator-activated receptor γ enhances myocardial ischemia-reperfusion injury in miceRegulation of cellular gas exchange, oxygen sensing, and metabolic control.Tumor necrosis factor-alpha--converting enzyme roles in hypertension-induced hypertrophy: look both ways when crossing the street.Cardiac Rac1 overexpression in mice creates a substrate for atrial arrhythmias characterized by structural remodelling.An important role of increase in tetrahydrobiopterin via H2O2-JAK2 signalling pathway in late phase of ischaemic preconditioning.Ischaemic postconditioning protects against reperfusion injury via the SAFE pathway.Targeted disruption of the meprin metalloproteinase beta gene protects against renal ischemia-reperfusion injury in mice.TNF-α, myocardial perfusion and function in patients with ST-segment elevation myocardial infarction and primary percutaneous coronary intervention.Comparison of cardioprotective and anti-inflammatory effects of ischemia pre- and postconditioning in rats with myocardial ischemia-reperfusion injury.Aldosterone promotes atrial fibrillationEugenol protects the transplanted heart against ischemia/reperfusion injury in rats by inhibiting the inflammatory response and apoptosis
P2860
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P2860
Insights from knock-out models concerning postischemic release of TNFalpha from isolated mouse hearts.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
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2006年學術文章
@zh-hant
name
Insights from knock-out models ...... ha from isolated mouse hearts.
@en
Insights from knock-out models ...... ha from isolated mouse hearts.
@nl
type
label
Insights from knock-out models ...... ha from isolated mouse hearts.
@en
Insights from knock-out models ...... ha from isolated mouse hearts.
@nl
prefLabel
Insights from knock-out models ...... ha from isolated mouse hearts.
@en
Insights from knock-out models ...... ha from isolated mouse hearts.
@nl
P2093
P1476
Insights from knock-out models ...... ha from isolated mouse hearts.
@en
P2093
Matrisian LM
P304
P356
10.1016/J.YJMCC.2006.09.020
P577
2006-11-13T00:00:00Z