Distinct tumor protein p53 mutants in breast cancer subgroups. - Wikidata - awgldk - TriplyDB

Distinct tumor protein p53 mutants in breast cancer subgroups.

Distinct tumor protein p53 mutants in breast cancer subgroups.

http://www.wikidata.org/entity/Q42511112

about

Current Management Strategies in Breast Cancer by Targeting Key Altered Molecular Players Emerging Non-Canonical Functions and Regulation by p53: p53 and Stemness PLA2G7 associates with hormone receptor negativity in clinical breast cancer samples and regulates epithelial-mesenchymal transition in cultured breast cancer cells Pre-clinical validation of a selective anti-cancer stem cell therapy for Numb-deficient human breast cancers.Identifying a highly-aggressive DCIS subgroup by studying intra-individual DCIS heterogeneity among invasive breast cancer patients Adiposity is associated with p53 gene mutations in breast cancer Obesity, independent of p53 gene dosage, promotes mammary tumor progression and upregulates the p53 regulator microRNA-504.Overexpression of miR-146a in basal-like breast cancer cells confers enhanced tumorigenic potential in association with altered p53 status Human breast tumor cells are more resistant to cardiac glycoside toxicity than non-tumorigenic breast cells.Predictive Significance of p53, Ki-67, and Bcl-2 Expression for Pathologic Complete Response after Neoadjuvant Chemotherapy for Triple-Negative Breast Cancer.p53 and ΔNp63α Coregulate the Transcriptional and Cellular Response to TGFβ and BMP Signals Identification of a new class of natural product MDM2 inhibitor: In vitro and in vivo anti-breast cancer activities and target validation.p53 Family Members Regulate Phenotypic Response to Aurora Kinase A Inhibition in Triple-Negative Breast Cancer Distinguishing Low-Risk Luminal A Breast Cancer Subtypes with Ki-67 and p53 Is More Predictive of Long-Term Survival P53 and Ki-67 as prognostic markers in triple-negative breast cancer patients.Targeting Mdmx to treat breast cancers with wild-type p53.High Expression of Three-Gene Signature Improves Prediction of Relapse-Free Survival in Estrogen Receptor-Positive and Node-Positive Breast Tumors.p53 deficiency linked to B cell translocation gene 2 (BTG2) loss enhances metastatic potential by promoting tumor growth in primary and metastatic sites in patient-derived xenograft (PDX) models of triple-negative breast cancer.Molecular markers for breast cancer: prediction on tumor behavior.Mutant p53 in cancer: new functions and therapeutic opportunities.TP53 mutations in human cancer: database reassessment and prospects for the next decade.The Role of the p53 Protein in Stem-Cell Biology and Epigenetic Regulation.Determination of HER2 and p53 Mutations by Sequence Analysis Method and EGFR/Chromosome 7 Gene Status by Fluorescence in Situ Hybridization for the Predilection of Targeted Therapy Modalities in Immunohistochemically Triple Negative Breast Carcinoma p53 deficiency induces cancer stem cell pool expansion in a mouse model of triple-negative breast tumors.A Numb-Mdm2 fuzzy complex reveals an isoform-specific involvement of Numb in breast cancer.SRC increases MYC mRNA expression in ER+ breast cancer via mRNA stabilization and inhibition of p53 function.Phosphorylation of NTRK1 at Y674/Y675 induced by TP53-dependent repression of PTPN6 expression: a potential novel prognostic marker for breast cancer.HDAC2 and HDAC5 Up-Regulations Modulate Survivin and miR-125a-5p Expressions and Promote Hormone Therapy Resistance in Estrogen Receptor Positive Breast Cancer Cells.Frequent overexpression of AMAP1, an Arf6 effector in cell invasion, is characteristic of the MMTV-PyMT rather than the MMTV-Neu human breast cancer model.18FDG-PET/CT and molecular markers to predict response to neoadjuvant chemotherapy and outcome in HER2-negative advanced luminal breast cancers patients.Coexistent Loss of the Expressions of BRCA1 and p53 Predicts Poor Prognosis in Triple-Negative Breast Cancer.TP53 protein levels, RNA-based pathway assessment, and race among invasive breast cancer cases.Complexes formed by mutant p53 and their roles in breast cancer.ERβ alters the chemosensitivity of luminal breast cancer cells by regulating p53 function.Salivary Duct Carcinoma: An Update on Morphologic Mimics and Diagnostic Use of Androgen Receptor Immunohistochemistry.Prevalence of MDM2 amplification and coalterations in 523 advanced cancer patients in the MD Anderson phase 1 clinic

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P2860

Distinct tumor protein p53 mutants in breast cancer subgroups.

http://www.wikidata.org/entity/Q42511112

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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name

Distinct tumor protein p53 mutants in breast cancer subgroups.

@en

Distinct tumor protein p53 mutants in breast cancer subgroups.

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type

label

Distinct tumor protein p53 mutants in breast cancer subgroups.

@en

Distinct tumor protein p53 mutants in breast cancer subgroups.

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prefLabel

Distinct tumor protein p53 mutants in breast cancer subgroups.

@en

Distinct tumor protein p53 mutants in breast cancer subgroups.

@nl

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Distinct tumor protein p53 mutants in breast cancer subgroups.

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P2093

Anne Dumay

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Christos Sotiriou

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Evelyne Wittmer

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Fabrice André

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Jacqueline Lehmann-Che

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Jean-Paul Feugeas

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Lajos Pusztai

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Louis-François Plassa

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Marc Espié

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Michel Marty

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P2860

Exquisite sensitivity of TP53 mutant and basal breast cancers to a dose-dense epirubicin-cyclophosphamide regimen

Gene expression patterns of breast carcinomas distinguish tumor subclasses with clinical implications

Repeated observation of breast tumor subtypes in independent gene expression data sets

Live or let die: the cell's response to p53

An embryonic stem cell-like gene expression signature in poorly differentiated aggressive human tumors

Supervised risk predictor of breast cancer based on intrinsic subtypes

Mutant p53 gain-of-function in cancer.

Quality control of mRNA function.

A simple p53 functional assay for screening cell lines, blood, and tumors.

Mechanisms of spontaneous human cancers

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