Secondary elevation of extracellular neurotransmitter amino acids in the reperfusion phase following focal cerebral ischemia.
about
Increases in GABA concentrations during cerebral ischaemia: a microdialysis study of extracellular amino acidsBench to bedside: brain edema and cerebral resuscitation: the present and future.Early Changes in Glutamate Metabolism and Perfusion in Basal Ganglia following Hypoxia-Ischemia in Neonatal Piglets: A Multi-Sequence 3.0T MR StudyFunctional modifications of acid-sensing ion channels by ligand-gated chloride channels.High-density expression of Ca2+-permeable ASIC1a channels in NG2 glia of rat hippocampus.Non-invasive diagnostic biomarkers for estimating the onset time of permanent cerebral ischemia.Perspectives on reperfusion-induced damage in rodent models of experimental focal ischemia and role of gamma-protein kinase C.Structure-activity relationship study of vitamin k derivatives yields highly potent neuroprotective agentsReperfusion injury: demonstration of brain damage produced by reperfusion after transient focal ischemia in rats.Activation of glycine and extrasynaptic GABA(A) receptors by taurine on the substantia gelatinosa neurons of the trigeminal subnucleus caudalis.Neuroprotective Mechanisms of Taurine against Ischemic Stroke.Novel mechanism of inhibition of rat kidney-type glutaminase by bis-2-(5-phenylacetamido-1,2,4-thiadiazol-2-yl)ethyl sulfide (BPTES)DCPIB, a specific inhibitor of volume regulated anion channels (VRACs), reduces infarct size in MCAo and the release of glutamate in the ischemic cortical penumbra.Different dynamic patterns of extracellular glutamate release in rat hippocampus after permanent or 30-min transient cerebral ischemia and histological correlation.Transient lack of glucose but not O2 is involved in ischemic postconditioning-induced neuroprotection.Glutamate release by an Na+ load and oxidative stress in nerve terminals: relevance to ischemia/reperfusion.Relationship between cerebral interstitial levels of amino acids and phosphorylation potential during secondary energy failure in hypoxic-ischemic newborn piglets.L-theanine administration results in neuroprotection and prevents glutamate receptor agonist-mediated injury in the rat model of cerebral ischemia-reperfusion.Excitatory GABAergic activation of cortical dividing glial cells.Increased production of extracellular glutamate by the mitochondrial glutaminase following neuronal death.Spatial stability of extracellular potassium ion and blood flow distribution in rat cerebral cortex after permanent middle cerebral artery occlusion.Changes in intracellular Ca2+ induced by GABAA receptor activation and reduction in Cl- gradient in neonatal rat neocortex.NMDA receptor-mediated differential laminar susceptibility to the intracellular Ca2+ accumulation induced by oxygen-glucose deprivation in rat neocortical slices.
P2860
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P2860
Secondary elevation of extracellular neurotransmitter amino acids in the reperfusion phase following focal cerebral ischemia.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
Secondary elevation of extrace ...... owing focal cerebral ischemia.
@en
Secondary elevation of extrace ...... owing focal cerebral ischemia.
@nl
type
label
Secondary elevation of extrace ...... owing focal cerebral ischemia.
@en
Secondary elevation of extrace ...... owing focal cerebral ischemia.
@nl
prefLabel
Secondary elevation of extrace ...... owing focal cerebral ischemia.
@en
Secondary elevation of extrace ...... owing focal cerebral ischemia.
@nl
P2093
P2860
P1476
Secondary elevation of extrace ...... owing focal cerebral ischemia.
@en
P2093
P2860
P304
P356
10.1097/00004647-199601000-00014
P577
1996-01-01T00:00:00Z
P5875
P6179
1053565288