Prostaglandin E2 synthesis and suppression of fibroblast proliferation by alveolar epithelial cells is cyclooxygenase-2-dependent.
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Feedback amplification of fibrosis through matrix stiffening and COX-2 suppressionMulti-walled carbon nanotubes induce COX-2 and iNOS expression via MAP kinase-dependent and -independent mechanisms in mouse RAW264.7 macrophagesPathogenetic mechanisms in usual interstitial pneumonia/idiopathic pulmonary fibrosis.Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosisFollowing the path of CCL2 from prostaglandins to periostin in lung fibrosisAsthmatic airway epithelial cells differentially regulate fibroblast expression of extracellular matrix components.Reduced miR-146a increases prostaglandin E₂in chronic obstructive pulmonary disease fibroblastsModulation of lung inflammation by the Epstein-Barr virus protein Zta.Novel therapeutic approaches for pulmonary fibrosis.Fibroblast-myofibroblast transition is differentially regulated by bronchial epithelial cells from asthmatic children.Severity of lung injury in cyclooxygenase-2-deficient mice is dependent on reduced prostaglandin E(2) production.Involvement of cyclooxygenase-2 and prostaglandins in the molecular pathogenesis of inflammatory lung diseases.Prostaglandin E2 and the pathogenesis of pulmonary fibrosis.Epithelial interactions and local engraftment of lung-resident mesenchymal stem cellsNormal Human Lung Epithelial Cells Inhibit Transforming Growth Factor-β Induced Myofibroblast Differentiation via Prostaglandin E2Systemic EP4 Inhibition Increases Adhesion Formation in a Murine Model of Flexor Tendon Repair.Idiopathic pulmonary fibrosis: emerging concepts on pharmacotherapy.Prostaglandin E(2) inhibits fibroblast migration by E-prostanoid 2 receptor-mediated increase in PTEN activityAntimitogenic effects of HDL and APOE mediated by Cox-2-dependent IP activation.Emerging drugs for idiopathic pulmonary fibrosis.The role of cyclooxygenase-2 in mechanical ventilation-induced lung injury.Epithelial-mesenchymal interactions in pulmonary fibrosis.Idiopathic pulmonary fibrosis : new concepts in pathogenesis and implications for drug therapyCholera toxin induces a shift from inactive to active cyclooxygenase 2 in alveolar macrophages activated by Mycobacterium bovis BCG.Hepatocyte growth factor regulates cyclooxygenase-2 expression via beta-catenin, Akt, and p42/p44 MAPK in human bronchial epithelial cells.Matrices of physiologic stiffness potently inactivate idiopathic pulmonary fibrosis fibroblasts.Lung fibroblast repair functions in patients with chronic obstructive pulmonary disease are altered by multiple mechanisms.Alveolar Epithelial Cell-Derived Prostaglandin E2 Serves as a Request Signal for Macrophage Secretion of Suppressor of Cytokine Signaling 3 during Innate Inflammation.Current and emerging drugs for idiopathic pulmonary fibrosis.Computational Modeling Predicts Simultaneous Targeting of Fibroblasts and Epithelial Cells Is Necessary for Treatment of Pulmonary Fibrosis.Evaluation of the Effects of Caffeic Acid Phenethyl Ester on Prostaglandin E2 and Two Key Cytokines Involved in Bleomycin-induced Pulmonary FibrosisA cross-talk between NFAT and NF-κB pathways is crucial for nickel-induced COX-2 expression in Beas-2B cellsPro-inflammatory phenotype of COPD fibroblasts not compatible with repair in COPD lung.Protease-activated receptors and prostaglandins in inflammatory lung disease.Prostaglandin E2 mediates IL-1beta-related fibroblast mitogenic effects in acute lung injury through differential utilization of prostanoid receptors.Opposing roles of leukotrienes and prostaglandins in fibrotic lung disease.Cellular and molecular mechanisms of chronic inflammation-associated organ fibrosis.The pathogenesis of bleomycin-induced lung injury in animals and its applicability to human idiopathic pulmonary fibrosis.The Role of PGE2 in Alveolar Epithelial and Lung Microvascular Endothelial Crosstalk.Lung Pericytes and Resident Fibroblasts: Busy Multitaskers.
P2860
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P2860
Prostaglandin E2 synthesis and suppression of fibroblast proliferation by alveolar epithelial cells is cyclooxygenase-2-dependent.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Prostaglandin E2 synthesis and ...... is cyclooxygenase-2-dependent.
@en
Prostaglandin E2 synthesis and ...... is cyclooxygenase-2-dependent.
@nl
type
label
Prostaglandin E2 synthesis and ...... is cyclooxygenase-2-dependent.
@en
Prostaglandin E2 synthesis and ...... is cyclooxygenase-2-dependent.
@nl
prefLabel
Prostaglandin E2 synthesis and ...... is cyclooxygenase-2-dependent.
@en
Prostaglandin E2 synthesis and ...... is cyclooxygenase-2-dependent.
@nl
P2093
P2860
P356
P1476
Prostaglandin E2 synthesis and ...... is cyclooxygenase-2-dependent
@en
P2093
Galen B Toews
Paul Christensen
Vibha Lama
P2860
P304
P356
10.1165/RCMB.4857
P577
2002-12-01T00:00:00Z