Vulnerability and plasticity of the GABA system in the pilocarpine model of spontaneous recurrent seizures.
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GABAergic transmission in temporal lobe epilepsy: the role of neurosteroids.Increased expression of GABA(A) receptor beta-subunits in the hippocampus of patients with temporal lobe epilepsyElectrophysiological evidence of monosynaptic excitatory transmission between granule cells after seizure-induced mossy fiber sproutingLoss of GABAergic neurons in the subiculum and its functional implications in temporal lobe epilepsyPlasticity of rat central inhibitory synapses through GABA metabolismLoss of NADPH diaphorase-positive neurons in the hippocampal formation of chronic pilocarpine-epileptic ratsCellular expression and localization of DGKζ-interacting NAP1-like proteins in the brain and functional implications under hypoxic stressMidkine, heparin-binding growth factor, blocks kainic acid-induced seizure and neuronal cell death in mouse hippocampusSelective loss of dentate hilar interneurons contributes to reduced synaptic inhibition of granule cells in an electrical stimulation-based animal model of temporal lobe epilepsyMassive and specific dysregulation of direct cortical input to the hippocampus in temporal lobe epilepsy.Surviving hilar somatostatin interneurons enlarge, sprout axons, and form new synapses with granule cells in a mouse model of temporal lobe epilepsy.Spatially clustered neuronal assemblies comprise the microstructure of synchrony in chronically epileptic networks.Excitatory input onto hilar somatostatin interneurons is increased in a chronic model of epilepsyStructural plasticity of dentate granule cell mossy fibers during the development of limbic epilepsyChronic epileptogenic cellular alterations in the limbic system after status epilepticus.Vulnerability of hippocampal GABA-ergic interneurons to kainate-induced excitotoxic injury during old ageWhat is GABAergic inhibition? How is it modified in epilepsy?Enhanced astrocytic Ca2+ signals contribute to neuronal excitotoxicity after status epilepticusMossy fiber plasticity and enhanced hippocampal excitability, without hippocampal cell loss or altered neurogenesis, in an animal model of prolonged febrile seizuresWater maze experience and prenatal choline supplementation differentially promote long-term hippocampal recovery from seizures in adulthood.Heterogeneity of the supramammillary-hippocampal pathways: evidence for a unique GABAergic neurotransmitter phenotype and regional differences.GABAA receptor modulation by neurosteroids in models of temporal lobe epilepsies.Aberrant neuronal synaptic connectivity in CA1 area of the hippocampus from pilocarpine-induced epileptic rats observed by fluorogold.Neuroanatomical clues to altered neuronal activity in epilepsy: from ultrastructure to signaling pathways of dentate granule cells.Initial loss but later excess of GABAergic synapses with dentate granule cells in a rat model of temporal lobe epilepsy.Interneuron loss reduces dendritic inhibition and GABA release in hippocampus of aged rats.Cortical GABAergic neurons are more severely impaired by alkalosis than acidosis.Promise of resveratrol for easing status epilepticus and epilepsy.Prenatal choline deficiency does not enhance hippocampal vulnerability after kainic acid-induced seizures in adulthood.Homeostatic increase in excitability in area CA1 after Schaffer collateral transection in vivo.Rapamycin suppresses axon sprouting by somatostatin interneurons in a mouse model of temporal lobe epilepsy.Treadmill exercise prevents GABAergic neuronal loss with suppression of neuronal activation in the pilocarpine-induced epileptic rats.Reorganization of supramammillary-hippocampal pathways in the rat pilocarpine model of temporal lobe epilepsy: evidence for axon terminal sprouting.Traumatic Brain Injury Increases Cortical Glutamate Network Activity by Compromising GABAergic ControlFunctional implications of seizure-induced neurogenesis.Selective reduction of cholecystokinin-positive basket cell innervation in a model of temporal lobe epilepsyDevelopment of pharmacoresistance to benzodiazepines but not cannabinoids in the hippocampal neuronal culture model of status epilepticus.Do structural changes in GABA neurons give rise to the epileptic state?Hilar mossy cell degeneration causes transient dentate granule cell hyperexcitability and impaired pattern separation.Operative GABAergic inhibition in hippocampal CA1 pyramidal neurons in experimental epilepsy.
P2860
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P2860
Vulnerability and plasticity of the GABA system in the pilocarpine model of spontaneous recurrent seizures.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Vulnerability and plasticity o ...... pontaneous recurrent seizures.
@en
Vulnerability and plasticity o ...... pontaneous recurrent seizures.
@nl
type
label
Vulnerability and plasticity o ...... pontaneous recurrent seizures.
@en
Vulnerability and plasticity o ...... pontaneous recurrent seizures.
@nl
prefLabel
Vulnerability and plasticity o ...... pontaneous recurrent seizures.
@en
Vulnerability and plasticity o ...... pontaneous recurrent seizures.
@nl
P1433
P1476
Vulnerability and plasticity o ...... spontaneous recurrent seizures
@en
P2093
C R Houser
P304
P356
10.1016/S0920-1211(96)00054-X
P577
1996-12-01T00:00:00Z