Operative GABAergic inhibition in hippocampal CA1 pyramidal neurons in experimental epilepsy.
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Gamma oscillation underlies hyperthermia-induced epileptiform-like spikes in immature rat hippocampal slicesSensory disturbances, inhibitory deficits, and the P50 wave in schizophreniaKainic acid-induced recurrent mossy fiber innervation of dentate gyrus inhibitory interneurons: possible anatomical substrate of granule cell hyper-inhibition in chronically epileptic ratsTransient impact of spike on theta rhythm in temporal lobe epilepsyChronic epileptogenic cellular alterations in the limbic system after status epilepticus.Vulnerability of hippocampal GABA-ergic interneurons to kainate-induced excitotoxic injury during old ageIncreased pyramidal excitability and NMDA conductance can explain posttraumatic epileptogenesis without disinhibition: a model.What is GABAergic inhibition? How is it modified in epilepsy?Slow state transitions of sustained neural oscillations by activity-dependent modulation of intrinsic excitabilityCell death and synaptic reorganizations produced by seizures.Cellular and network mechanisms of electrographic seizures.Interneuron loss reduces dendritic inhibition and GABA release in hippocampus of aged rats.Selective activation of parvalbumin- or somatostatin-expressing interneurons triggers epileptic seizurelike activity in mouse medial entorhinal cortex.Reorganization of supramammillary-hippocampal pathways in the rat pilocarpine model of temporal lobe epilepsy: evidence for axon terminal sprouting.Molecular biology and ontogeny of glutamate receptors in the mammalian central nervous system.Selective reduction of cholecystokinin-positive basket cell innervation in a model of temporal lobe epilepsyA candidate mechanism underlying the variance of interictal spike propagation.Do structural changes in GABA neurons give rise to the epileptic state?GABAergic synchronization in the limbic system and its role in the generation of epileptiform activity.Hippocampal Expression of Connexin36 and Connexin43 during Epileptogenesis in Pilocarpine Model of Epilepsy.Primary and secondary mechanisms of epileptogenesis in the temporal lobe: there is a before and an after.Homeostasis of brain dynamics in epilepsy: a feedback control systems perspective of seizures.Short- and long-term changes in CA1 network excitability after kainate treatment in rats.In vitro formation of a secondary epileptogenic mirror focus by interhippocampal propagation of seizures.Contribution of intrinsic neuronal factors in the generation of cortically driven electrographic seizures.Changes in mIPSCs and sIPSCs after kainate treatment: evidence for loss of inhibitory input to dentate granule cells and possible compensatory responses.Short-term effects of kainic acid on CA1 hippocampal interneurons differentially vulnerable to excitotoxicity.Correlation between axonal morphologies and synaptic input kinetics of interneurons from mouse visual cortex.Hyperexcitability of the CA1 hippocampal region during epileptogenesis.Inhibitory or excitatory? Optogenetic interrogation of the functional roles of GABAergic interneurons in epileptogenesis.On the relevance of prolonged convulsive status epilepticus in animals to the etiology and neurobiology of human temporal lobe epilepsy.Aging in the rat hippocampus is associated with widespread reductions in the number of glutamate decarboxylase-67 positive interneurons but not interneuron degeneration.Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.Newly formed excitatory pathways provide a substrate for hyperexcitability in experimental temporal lobe epilepsy.Decrease in inhibition in dentate granule cells from patients with medial temporal lobe epilepsy.Cell-specific alterations in synaptic properties of hippocampal CA1 interneurons after kainate treatment.Cell domain-dependent changes in the glutamatergic and GABAergic drives during epileptogenesis in the rat CA1 region.Simultaneous enhancement of excitation and postburst inhibition at the end of focal seizures.Electrophysiological properties and subunit composition of GABAA receptors in patients with gelastic seizures and hypothalamic hamartoma.Transplanted CNS stem cells form functional synapses in vivo.
P2860
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P2860
Operative GABAergic inhibition in hippocampal CA1 pyramidal neurons in experimental epilepsy.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Operative GABAergic inhibition ...... rons in experimental epilepsy.
@ast
Operative GABAergic inhibition ...... rons in experimental epilepsy.
@en
type
label
Operative GABAergic inhibition ...... rons in experimental epilepsy.
@ast
Operative GABAergic inhibition ...... rons in experimental epilepsy.
@en
prefLabel
Operative GABAergic inhibition ...... rons in experimental epilepsy.
@ast
Operative GABAergic inhibition ...... rons in experimental epilepsy.
@en
P2860
P50
P356
P1476
Operative GABAergic inhibition ...... urons in experimental epilepsy
@en
P2093
P2860
P304
12151-12156
P356
10.1073/PNAS.94.22.12151
P407
P577
1997-10-01T00:00:00Z