Loss of Rhb1, a Rheb-related GTPase in fission yeast, causes growth arrest with a terminal phenotype similar to that caused by nitrogen starvation.
about
Signaling by target of rapamycin proteins in cell growth controlBiochemical and functional characterizations of small GTPase Rheb and TSC2 GAP activityRegulation of TOR by small GTPasesRheb GTPase is a direct target of TSC2 GAP activity and regulates mTOR signalingNutrient-sensing mechanisms across evolutionA role in vacuolar arginine transport for yeast Btn1p and for human CLN3, the protein defective in Batten disease.Early development of Moniliophthora perniciosa basidiomata and developmentally regulated genesFission yeast TORC1 regulates phosphorylation of ribosomal S6 proteins in response to nutrients and its activity is inhibited by rapamycin.Two ras pathways in fission yeast are differentially regulated by two ras guanine nucleotide exchange factorsA defect in protein farnesylation suppresses a loss of Schizosaccharomyces pombe tsc2+, a homolog of the human gene predisposing to tuberous sclerosis complex.Deletion of the Aspergillus fumigatus gene encoding the Ras-related protein RhbA reduces virulence in a model of Invasive pulmonary aspergillosis.Point mutations in TOR confer Rheb-independent growth in fission yeast and nutrient-independent mammalian TOR signaling in mammalian cellsRhb1 regulates the expression of secreted aspartic protease 2 through the TOR signaling pathway in Candida albicans.Constitutively active Rheb induces oncogenic transformation.Distinctive responses to nitrogen starvation in the dominant active mutants of the fission yeast Rheb GTPase.Loss of the TOR kinase Tor2 mimics nitrogen starvation and activates the sexual development pathway in fission yeast.Schizosaccharomyces pombe essential genes: a pilot study.Opposite effects of tor1 and tor2 on nitrogen starvation responses in fission yeastNitrogen regulates AMPK to control TORC1 signaling.Conservation of the Tsc/Rheb/TORC1/S6K/S6 Signaling in Fission Yeast.Ragulator and GATOR1 complexes promote fission yeast growth by attenuating TOR complex 1 through Rag GTPases.Amino acid and small GTPase regulation of mTORC1.TORC1 and TORC2 converge to regulate the SAGA co-activator in response to nutrient availability.The Target of Rapamycin and Mechanisms of Cell Growth.
P2860
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P2860
Loss of Rhb1, a Rheb-related GTPase in fission yeast, causes growth arrest with a terminal phenotype similar to that caused by nitrogen starvation.
description
2000 nî lūn-bûn
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2000年の論文
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2000年論文
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2000年論文
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2000年論文
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2000年論文
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2000年论文
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name
Loss of Rhb1, a Rheb-related G ...... caused by nitrogen starvation.
@en
Loss of Rhb1, a Rheb-related G ...... caused by nitrogen starvation.
@nl
type
label
Loss of Rhb1, a Rheb-related G ...... caused by nitrogen starvation.
@en
Loss of Rhb1, a Rheb-related G ...... caused by nitrogen starvation.
@nl
prefLabel
Loss of Rhb1, a Rheb-related G ...... caused by nitrogen starvation.
@en
Loss of Rhb1, a Rheb-related G ...... caused by nitrogen starvation.
@nl
P2093
P2860
P1433
P1476
Loss of Rhb1, a Rheb-related G ...... caused by nitrogen starvation
@en
P2093
P2860
P304
P407
P577
2000-06-01T00:00:00Z