Inactivation of the mismatch repair system in Pseudomonas aeruginosa attenuates virulence but favors persistence of oropharyngeal colonization in cystic fibrosis mice.
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Sub-Optimal Treatment of Bacterial BiofilmsRole of small colony variants in persistence of Pseudomonas aeruginosa infections in cystic fibrosis lungsDynamics of mutator and antibiotic-resistant populations in a pharmacokinetic/pharmacodynamic model of Pseudomonas aeruginosa biofilm treatment.Pseudomonas aeruginosa biofilm infections in cystic fibrosis: insights into pathogenic processes and treatment strategies.Epistatic roles for Pseudomonas aeruginosa MutS and DinB (DNA Pol IV) in coping with reactive oxygen species-induced DNA damage.Pseudomonas aeruginosa RsmA plays an important role during murine infection by influencing colonization, virulence, persistence, and pulmonary inflammationAntimicrobial resistance and virulence: a successful or deleterious association in the bacterial world?Analysis of acquisition of Pseudomonas aeruginosa gastrointestinal mucosal colonization and horizontal transmission in a murine model.Hypermutation and stress adaptation in bacteria.Chronic pulmonary pseudomonal infection in patients with cystic fibrosis: A model for early phase symbiotic evolution.Evolution of Pseudomonas aeruginosa Antimicrobial Resistance and Fitness under Low and High Mutation Rates.The thymidine-dependent small-colony-variant phenotype is associated with hypermutability and antibiotic resistance in clinical Staphylococcus aureus isolates.Genetic adaptation of Pseudomonas aeruginosa to the airways of cystic fibrosis patients is catalyzed by hypermutationSublethal ciprofloxacin treatment leads to rapid development of high-level ciprofloxacin resistance during long-term experimental evolution of Pseudomonas aeruginosaIncreased susceptibility to colistin in hypermutable Pseudomonas aeruginosa strains from chronic respiratory infections.Azithromycin in Pseudomonas aeruginosa biofilms: bactericidal activity and selection of nfxB mutants.Playing fast and loose with mutation.Heterogeneous Antimicrobial Susceptibility Characteristics in Pseudomonas aeruginosa Isolates from Cystic Fibrosis Patients.Hypermutation-induced in vivo oxidative stress resistance enhances Vibrio cholerae host adaptation
P2860
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P2860
Inactivation of the mismatch repair system in Pseudomonas aeruginosa attenuates virulence but favors persistence of oropharyngeal colonization in cystic fibrosis mice.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Inactivation of the mismatch r ...... ation in cystic fibrosis mice.
@en
Inactivation of the mismatch r ...... ation in cystic fibrosis mice.
@nl
type
label
Inactivation of the mismatch r ...... ation in cystic fibrosis mice.
@en
Inactivation of the mismatch r ...... ation in cystic fibrosis mice.
@nl
prefLabel
Inactivation of the mismatch r ...... ation in cystic fibrosis mice.
@en
Inactivation of the mismatch r ...... ation in cystic fibrosis mice.
@nl
P2093
P2860
P356
P1476
Inactivation of the mismatch r ...... zation in cystic fibrosis mice
@en
P2093
Antonio Oliver
Bartolomé Moya
José L Pérez
Nuria Borrell
Teresa de Francisco
P2860
P304
P356
10.1128/JB.00120-07
P407
P577
2007-02-16T00:00:00Z