Structural consequences of Kcna1 gene deletion and transfer in the mouse hippocampus.
about
Homeostasis or channelopathy? Acquired cell type-specific ion channel changes in temporal lobe epilepsy and their antiepileptic potentialDeletion of the Kv2.1 delayed rectifier potassium channel leads to neuronal and behavioral hyperexcitability.Reduction of seizures by transplantation of cortical GABAergic interneuron precursors into Kv1.1 mutant mice.Kv1.1 potassium channel deficiency reveals brain-driven cardiac dysfunction as a candidate mechanism for sudden unexplained death in epilepsy.Basolateral amygdala regulation of adult hippocampal neurogenesis and fear-related activation of newborn neurons.The promise of an interneuron-based cell therapy for epilepsyUbiquitin-positive intranuclear inclusions in neuronal and glial cells in a mouse model of the fragile X premutation.Ketogenic diets: evidence for short- and long-term efficacy.Ketone bodies mediate antiseizure effects through mitochondrial permeability transition.Orexin Receptor Antagonism Improves Sleep and Reduces Seizures in Kcna1-null Mice.Antibody-based validation of CNS ion channel drug targets.Loss of the Kv1.1 potassium channel promotes pathologic sharp waves and high frequency oscillations in in vitro hippocampal slices.Kv1 potassium channel complexes in vivo require Kvbeta2 subunits in dorsal spinal neurons.Issues in Clinical Epileptology: A View from the Bench. A Festschrift in Honor of Philip A. Schwartzkroin, PhD.Targeting deficiencies in mitochondrial respiratory complex I and functional uncoupling exerts anti-seizure effects in a genetic model of temporal lobe epilepsy and in a model of acute temporal lobe seizures.Kv1.1 and Kv1.2: similar channels, different seizure models.Presynaptic mechanisms of neuronal plasticity and their role in epilepsy.Breathe Easy: Modifying Mitochondrial Respiration to Treat Seizures.Potassium Channels in Epilepsy.Ketogenic diet treatment increases longevity in Kcna1-null mice, a model of sudden unexpected death in epilepsyIn vitro and intrathecal siRNA mediated K(V)1.1 knock-down in primary sensory neurons.Regulation of action potential delays via voltage-gated potassium Kv1.1 channels in dentate granule cells during hippocampal epilepsyThe importance of immunohistochemical analyses in evaluating the phenotype of Kv channel knockout mice.Spontaneous seizures in Kcna1-null mice lacking voltage-gated Kv1.1 channels activate Fos expression in select limbic circuits.Ion Channels in Genetic Epilepsy: From Genes and Mechanisms to Disease-Targeted Therapies.Accumulation of rest deficiency precedes sudden death of epileptic Kv1.1 knockout mice, a model of sudden unexpected death in epilepsy.Respiratory dysfunction progresses with age in Kcna1-null mice, a model of sudden unexpected death in epilepsy.Somatodendritic ion channel expression in substantia nigra pars compacta dopaminergic neurons across postnatal development.Human limbic encephalitis serum enhances hippocampal mossy fiber-CA3 pyramidal cell synaptic transmission.
P2860
Q28080655-955291AD-0427-487C-94D6-81E7BAC9B31BQ28510330-500618CE-7EBC-4F38-A254-7BEAF84121AFQ30490096-E5E84BA6-F4E9-469F-93BA-7E96B5D553A4Q30494617-A28DF596-ABCD-4B99-B94C-7C6264D0CD65Q30619232-539BE102-53F3-4AF8-9979-0CDD9659F7CDQ34685588-47E2E133-D1C5-484A-B3F8-13FDD3A0EC03Q34915612-5E0DDF0B-E728-446E-BF75-8A49BA23CF2DQ34971377-1A52095C-D033-4574-A4EF-07E6912310ABQ35779437-B332AA44-ABA7-4200-A3BA-BC44BE62900AQ36461014-7281CEAE-5D10-41E1-8DF6-104EF86DD529Q36593655-4D622651-2D1C-4D1E-89CD-B573D96F4D22Q36829101-85C06769-079C-4E13-9CC4-85413F26FC09Q36957003-73B54E86-ABAA-47F0-B1C5-6F2CED965052Q37372251-7024106C-3363-4A0D-9FE0-7945B206857FQ37708764-8ACD90D6-D8F3-4F15-B6B0-91840B70530EQ38011837-362FD71D-6715-4447-A21D-009DEFF6B23DQ38225535-39983FC8-B402-4F48-BADE-4C3AED9E8810Q38350356-743DE46B-0517-4DA8-BE00-D34D9F22BAABQ38824328-6CF5D09C-9984-4D79-A6AD-EEF23F769A1FQ38848243-0F7717F8-0C54-4E4D-BF97-E4D423394433Q39865756-9A0DD66B-1A7A-48DC-9E24-2513EE8B3334Q42181854-2BB78BE3-335E-4F00-8D6B-6FB0050D0497Q42186187-78B4C985-27FC-4C44-9B78-C0266747A526Q42478890-0AA1C866-329F-4CFE-8E28-B639F818E6D4Q47269323-1B04C992-9C6F-457D-92CE-CCE7353E3E1FQ47387278-B740FE8E-C470-40E1-84EB-9469E50C1E2DQ47658597-1A3543B0-B0EF-4E7D-A2CE-99E76D80DB99Q48114005-B360E637-3BEC-4C0D-AE31-943BF25DE240Q51031349-B1970FD0-24C9-4E3B-82A1-C8CC17203F4B
P2860
Structural consequences of Kcna1 gene deletion and transfer in the mouse hippocampus.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Structural consequences of Kcna1 gene deletion and transfer in the mouse hippocampus.
@en
Structural consequences of Kcna1 gene deletion and transfer in the mouse hippocampus.
@nl
type
label
Structural consequences of Kcna1 gene deletion and transfer in the mouse hippocampus.
@en
Structural consequences of Kcna1 gene deletion and transfer in the mouse hippocampus.
@nl
prefLabel
Structural consequences of Kcna1 gene deletion and transfer in the mouse hippocampus.
@en
Structural consequences of Kcna1 gene deletion and transfer in the mouse hippocampus.
@nl
P2093
P2860
P1433
P1476
Structural consequences of Kcna1 gene deletion and transfer in the mouse hippocampus.
@en
P2093
Angela L Lee
Bruce L Tempel
Eliana Clark
H Jürgen Wenzel
Helene Vacher
James S Trimmer
Philip A Schwartzkroin
Robert M Sapolsky
P2860
P304
P356
10.1111/J.1528-1167.2007.01189.X
P577
2007-07-25T00:00:00Z