about
A novel nuclear interactor of ARF and MDM2 (NIAM) that maintains chromosomal stabilityp53 Acetylation: Regulation and ConsequencesARF directly binds DP1: interaction with DP1 coincides with the G1 arrest function of ARF.Development and translational imaging of a TP53 porcine tumorigenesis modelLarge-scale molecular comparison of human schwann cells to malignant peripheral nerve sheath tumor cell lines and tissues.NIAM-deficient mice are predisposed to the development of proliferative lesions including B-cell lymphomasRGS6 suppresses Ras-induced cellular transformation by facilitating Tip60-mediated Dnmt1 degradation and promoting apoptosis.RABL6A promotes G1-S phase progression and pancreatic neuroendocrine tumor cell proliferation in an Rb1-dependent mannerRABL6A, a novel RAB-like protein, controls centrosome amplification and chromosome instability in primary fibroblasts.The ARF tumor suppressor inhibits tumor cell colonization independent of p53 in a novel mouse model of pancreatic ductal adenocarcinoma metastasis.Generation and characterization of monoclonal antibodies to NIAM: a nuclear interactor of ARF and Mdm2.Residues in the alternative reading frame tumor suppressor that influence its stability and p53-independent activitiesRABL6A Promotes Oxaliplatin Resistance in Tumor Cells and Is a New Marker of Survival for Resected Pancreatic Ductal Adenocarcinoma Patients.Myst2/Kat7 histone acetyltransferase interaction proteomics reveals tumour-suppressor Niam as a novel binding partner in embryonic stem cellsImmunohistochemical Markers for Prospective Studies in Neurofibromatosis-1 Porcine Models.NEK2 induces osteoclast differentiation and bone destruction via heparanase in multiple myeloma.Porcine cancer models for translational oncology.Overexpression of mouse D-type cyclins accelerates G1 phase in rodent fibroblasts.Respiratory syncytial virus decreases p53 protein to prolong survival of airway epithelial cells.The t(8;21) fusion protein, AML1 ETO, specifically represses the transcription of the p14(ARF) tumor suppressor in acute myeloid leukemia.Expression of the p16INK4a tumor suppressor versus other INK4 family members during mouse development and aging.Deletion and altered regulation of p16INK4a and p15INK4b in undifferentiated mouse skin tumors.Combination of Proteasome and Histone Deacetylase Inhibitors Overcomes the Impact of Gain-of-Function p53 MutationsA porcine model of neurofibromatosis type 1 that mimics the human diseaseRadiomic biomarkers informative of cancerous transformation in neurofibromatosis-1 plexiform tumorsRABL6A is an essential driver of MPNSTs that negatively regulates the RB1 pathway and sensitizes tumor cells to CDK4/6 inhibitorsGene Expression Signatures Identify Novel Therapeutics for Metastatic Pancreatic Neuroendocrine TumorsAssessment of nociception and related quality-of-life measures in a porcine model of neurofibromatosis type 1
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description
hulumtuese
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researcher
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wetenschapper
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հետազոտող
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name
Dawn Quelle
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Dawn Quelle
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Dawn Quelle
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Dawn Quelle
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Dawn Quelle
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type
label
Dawn Quelle
@ast
Dawn Quelle
@en
Dawn Quelle
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Dawn Quelle
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Dawn Quelle
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prefLabel
Dawn Quelle
@ast
Dawn Quelle
@en
Dawn Quelle
@es
Dawn Quelle
@nl
Dawn Quelle
@sl
P106
P21
P31
P496
0000-0001-8776-0122
P569
2000-01-01T00:00:00Z