Activation of HuR downstream of p38 MAPK promotes cardiomyocyte hypertrophy.

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Activation of HuR downstream of p38 MAPK promotes cardiomyocyte hypertrophy.

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2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

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Activation of HuR downstream of p38 MAPK promotes cardiomyocyte hypertrophy.

@en

Activation of HuR downstream of p38 MAPK promotes cardiomyocyte hypertrophy.

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Item

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Activation of HuR downstream of p38 MAPK promotes cardiomyocyte hypertrophy.

@en

Activation of HuR downstream of p38 MAPK promotes cardiomyocyte hypertrophy.

@nl

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Activation of HuR downstream of p38 MAPK promotes cardiomyocyte hypertrophy.

@en

Activation of HuR downstream of p38 MAPK promotes cardiomyocyte hypertrophy.

@nl

P1476

Activation of HuR downstream of p38 MAPK promotes cardiomyocyte hypertrophy.

@en

P2093

Jeffrey Aube

http://www.w3.org/2001/XMLSchema#string

Joshua B Benoit

http://www.w3.org/2001/XMLSchema#string

Liang Xu

http://www.w3.org/2001/XMLSchema#string

Sarah R Anthony

http://www.w3.org/2001/XMLSchema#string

Xiaoqing Wu

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P2860

Protein kinase cascades in the regulation of cardiac hypertrophy

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Posttranslational modification of the AU-rich element binding protein HuR by protein kinase Cdelta elicits angiotensin II-induced stabilization and nuclear export of cyclooxygenase 2 mRNA

Protein kinase C alpha-dependent phosphorylation of the mRNA-stabilizing factor HuR: implications for posttranscriptional regulation of cyclooxygenase-2

Relief of microRNA-mediated translational repression in human cells subjected to stress

Posttranscriptional regulation of cancer traits by HuR

A novel high throughput biochemical assay to evaluate the HuR protein-RNA complex formation

Myocardial knockdown of mRNA-stabilizing protein HuR attenuates post-MI inflammatory response and left ventricular dysfunction in IL-10-null mice.

Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy.

G-proteins in growth and apoptosis: lessons from the heart.

P304

1735-1741

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P31

scholarly article

P356

10.1016/J.CELLSIG.2016.08.005

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Samuel Slone

Michael Tranter

P577

2016-08-10T00:00:00Z

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P698

27521603

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P932

5032653

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