Selective blockade of endothelial Ca2+-activated small- and intermediate-conductance K+-channels suppresses EDHF-mediated vasodilation. - Wikidata - awgldk - TriplyDB

Selective blockade of endothelial Ca2+-activated small- and intermediate-conductance K+-channels suppresses EDHF-mediated vasodilation.

Selective blockade of endothelial Ca2+-activated small- and intermediate-conductance K+-channels suppresses EDHF-mediated vasodilation.

http://www.wikidata.org/entity/Q42588835

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Selective positive modulation of the SK3 and SK2 subtypes of small conductance Ca2+-activated K+ channels Endothelium-dependent smooth muscle hyperpolarization: do gap junctions provide a unifying hypothesis?The Gatekeepers in the Mouse Ophthalmic Artery: Endothelium-Dependent Mechanisms of Cholinergic Vasodilation.Role of arachidonic acid lipoxygenase metabolites in the regulation of vascular tone Definitive role for natriuretic peptide receptor-C in mediating the vasorelaxant activity of C-type natriuretic peptide and endothelium-derived hyperpolarising factor Connexins: gaps in our knowledge of vascular function.Gap junctions in the control of vascular function.Interactions between NO, CO and an endothelium-derived hyperpolarizing factor (EDHF) in maintaining patency of the ductus arteriosus in the mouse.Novel role of endothelial BKCa channels in altered vasoreactivity following hypoxia Arterial response to shear stress critically depends on endothelial TRPV4 expression.Naphtho[1,2-d]thiazol-2-ylamine (SKA-31), a new activator of KCa2 and KCa3.1 potassium channels, potentiates the endothelium-derived hyperpolarizing factor response and lowers blood pressure Cerebrovascular responses to insulin in rats.Prenatal testosterone induces sex-specific dysfunction in endothelium-dependent relaxation pathways in adult male and female rats Recycling of the Ca2+-activated K+ channel, KCa2.3, is dependent upon RME-1, Rab35/EPI64C, and an N-terminal domain Ovariectomy-induced reductions in endothelial SK3 channel activity and endothelium-dependent vasorelaxation in murine mesenteric arteries.SK3-1C, a dominant-negative suppressor of SKCa and IKCa channels.ESCRT-dependent targeting of plasma membrane localized KCa3.1 to the lysosomes A key role for Na+/K+-ATPase in the endothelium-dependent oscillatory activity of mouse small mesenteric arteries.Potassium channels in the peripheral microcirculation.Role of in vivo vascular redox in resistance arteries Emerging role of the calcium-activated, small conductance, SK3 K+ channel in distal tubule function: regulation by TRPV4 Endothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouse P2Y₂ receptor activation decreases blood pressure via intermediate conductance potassium channels and connexin 37 Endothelium-dependent nitric oxide and hyperpolarization-mediated venous relaxation pathways in rat inferior vena cava.Adaptive response of vascular endothelial cells to an acute increase in shear stress magnitude Factors, fiction and endothelium-derived hyperpolarizing factor.Endothelial potassium channels, endothelium-dependent hyperpolarization and the regulation of vascular tone in health and disease.Therapeutic potential of KCa3.1 blockers: recent advances and promising trends.Expression of a Diverse Array of Ca2+-Activated K+ Channels (SK1/3, IK1, BK) that Functionally Couple to the Mechanosensitive TRPV4 Channel in the Collecting Duct System of Kidney.Vascular Tone Regulation Induced by C-Type Natriuretic Peptide: Differences in Endothelium-Dependent and -Independent Mechanisms Involved in Normotensive and Spontaneously Hypertensive Rats Enalapril Normalizes Endothelium-Derived Hyperpolarizing Factor-Mediated Relaxation in Mesenteric Artery of Adult Hypertensive Rats Prenatally Exposed to Testosterone.Ca2+-activated K+ channels in murine endothelial cells: block by intracellular calcium and magnesium.SKA-31, a novel activator of SK(Ca) and IK(Ca) channels, increases coronary flow in male and female rat hearts KCa 3.1 channels maintain endothelium-dependent vasodilatation in isolated perfused kidneys of spontaneously hypertensive rats after chronic inhibition of NOS.Modeling Ca2+ signaling in the microcirculation: intercellular communication and vasoreactivity.K+ channel modulators for the treatment of neurological disorders and autoimmune diseases Endothelial Ca+-activated K+ channels in normal and impaired EDHF-dilator responses--relevance to cardiovascular pathologies and drug discovery TRPV4-dependent dilation of peripheral resistance arteries influences arterial pressure.Upregulation of intermediate calcium-activated potassium channels counterbalance the impaired endothelium-dependent vasodilation in stroke-prone spontaneously hypertensive rats.Endothelial calcium-activated potassium channels as therapeutic targets to enhance availability of nitric oxide.

P2860

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P2860

Selective blockade of endothelial Ca2+-activated small- and intermediate-conductance K+-channels suppresses EDHF-mediated vasodilation.

http://www.wikidata.org/entity/Q42588835

description

2003 nî lūn-bûn

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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2003年论文

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2003年论文

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name

Selective blockade of endothel ...... es EDHF-mediated vasodilation.

@en

Selective blockade of endothel ...... es EDHF-mediated vasodilation.

@nl

type

label

Selective blockade of endothel ...... es EDHF-mediated vasodilation.

@en

Selective blockade of endothel ...... es EDHF-mediated vasodilation.

@nl

prefLabel

Selective blockade of endothel ...... es EDHF-mediated vasodilation.

@en

Selective blockade of endothel ...... es EDHF-mediated vasodilation.

@nl

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British Journal of Pharmacology

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Selective blockade of endothel ...... es EDHF-mediated vasodilation.

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P2093

Andrea Knorr

http://www.w3.org/2001/XMLSchema#string

Axel R Pries

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Ines Eichler

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Ivica Grgic

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Joachim Hoyer

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Judith Wibawa

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Ralf Köhler

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Susanne Brakemeier

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P2860

A novel gene, hKCa4, encodes the calcium-activated potassium channel in human T lymphocytes

Design of a potent and selective inhibitor of the intermediate-conductance Ca2+-activated K+ channel, IKCa1: a potential immunosuppressant

K+ currents underlying the action of endothelium-derived hyperpolarizing factor in guinea-pig, rat and human blood vessels

Cytochrome P450 2C is an EDHF synthase in coronary arteries

Activation of vascular smooth muscle K+ channels by endothelium-derived relaxing factors.

Endothelium-dependent hyperpolarization of vascular smooth muscle cells.

Ion channels and their functional role in vascular endothelium.

Potassium ions and endothelium-derived hyperpolarizing factor in guinea-pig carotid and porcine coronary arteries

Apamin-sensitive, non-nitric oxide (NO) endothelium-dependent relaxations to bradykinin in the bovine isolated coronary artery: no role for cytochrome P450 and K+.

Effects of inhibitors of small- and intermediate-conductance calcium-activated potassium channels, inwardly-rectifying potassium channels and Na(+)/K(+) ATPase on EDHF relaxations in the rat hepatic artery.

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594-601

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10.1038/SJ.BJP.0705075

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4

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138

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2003-02-01T00:00:00Z

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10890771

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12598413

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1573692

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