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Dietary Pectin Increases Intestinal Crypt Stem Cell Survival following Radiation InjuryLoss of p21Waf1/Cip1/Sdi1 enhances intestinal stem cell survival following radiation injuryReg IV regulates normal intestinal and colorectal cancer cell susceptibility to radiation-induced apoptosis.DCLK1 regulates pluripotency and angiogenic factors via microRNA-dependent mechanisms in pancreatic cancer.DCLK1 facilitates intestinal tumor growth via enhancing pluripotency and epithelial mesenchymal transitionDoublecortin and CaM kinase-like-1 and leucine-rich-repeat-containing G-protein-coupled receptor mark quiescent and cycling intestinal stem cells, respectively.Identification of a novel putative gastrointestinal stem cell and adenoma stem cell marker, doublecortin and CaM kinase-like-1, following radiation injury and in adenomatous polyposis coli/multiple intestinal neoplasia mice.DCAMKL-1 regulates epithelial-mesenchymal transition in human pancreatic cells through a miR-200a-dependent mechanism.Fluvastatin interferes with hepatitis C virus replication via microtubule bundling and a doublecortin-like kinase-mediated mechanism.Doublecortin-like kinase 1 is elevated serologically in pancreatic ductal adenocarcinoma and widely expressed on circulating tumor cellsDCLK1 is a broadly dysregulated target against epithelial-mesenchymal transition, focal adhesion, and stemness in clear cell renal carcinomaNanoparticle-based delivery of siDCAMKL-1 increases microRNA-144 and inhibits colorectal cancer tumor growth via a Notch-1 dependent mechanism.Hepatitis C virus-induced cancer stem cell-like signatures in cell culture and murine tumor xenografts.Ablation of Doublecortin-Like Kinase 1 in the Colonic Epithelium Exacerbates Dextran Sulfate Sodium-Induced Colitis.Regulation of miRNAs by agents targeting the tumor stem cell markers DCLK1, MSI1, LGR5, and BMI1.Brief report: Dclk1 deletion in tuft cells results in impaired epithelial repair after radiation injury.Inflammatory and oncogenic roles of a tumor stem cell marker doublecortin-like kinase (DCLK1) in virus-induced chronic liver diseasesDclk1+ small intestinal epithelial tuft cells display the hallmarks of quiescence and self-renewalTranslation regulatory factor RBM3 is a proto-oncogene that prevents mitotic catastrophe.Intestinal tuft cells regulate the ATM mediated DNA Damage response via Dclk1 dependent mechanism for crypt restitution following radiation injury.Isolation of anticancer drug TAXOL from Pestalotiopsis breviseta with apoptosis and B-Cell lymphoma protein docking studies.Dclk1, a tumor stem cell marker, regulates pro-survival signaling and self-renewal of intestinal tumor cells.Inhibition of Notch signaling reduces the number of surviving Dclk1+ reserve crypt epithelial stem cells following radiation injury.Journal of Basic and Clinical Pharmacy signs collaboration agreement with Medknow Publications-Wolters Kluwer Health.(Z)-3,5,4'-Trimethoxystilbene Limits Hepatitis C and Cancer Pathophysiology by Blocking Microtubule Dynamics and Cell-Cycle Progression.Small molecule kinase inhibitor LRRK2-IN-1 demonstrates potent activity against colorectal and pancreatic cancer through inhibition of doublecortin-like kinase 1.XMD8-92 inhibits pancreatic tumor xenograft growth via a DCLK1-dependent mechanism.Wnt inhibitory factor 1 suppresses cancer stemness and induces cellular senescence.Effect of taxol from Pestalotiopsis mangiferae on A549 cells-In vitro studyA word from Journal of Basic and Clinical Pharmacy editor's desk.Identification of the putative intestinal stem cell marker doublecortin and CaM kinase-like-1 in Barrett's esophagus and esophageal adenocarcinoma.Functional antagonism between RNA binding proteins HuR and CUGBP2 determines the fate of COX-2 mRNA translation.Antiviral activity of ancient system of ayurvedic medicinal plant Cissus quadrangularis L. (Vitaceae).DCLK1 is detectable in plasma of patients with Barrett's esophagus and esophageal adenocarcinoma.Knockdown of RNA binding protein musashi-1 leads to tumor regression in vivoEpigenetic variants and biomarkers for colon cancerDCLK1 Monoclonal Antibody-Based CAR-T Cells as a Novel Treatment Strategy against Human Colorectal CancersCorrection: Fluvastatin Interferes with Hepatitis C Virus Replication via Microtubule Bundling and a Doublecortin-like Kinase-Mediated Mechanism
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description
onderzoeker
@nl
researcher
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հետազոտող
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name
Sripathi Sureban
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Sripathi Sureban
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Sripathi Sureban
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Sripathi Sureban
@nl
Sripathi Sureban
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Sripathi Sureban
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Sripathi Sureban
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Sripathi Sureban
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Sripathi Sureban
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Sripathi Sureban
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Sripathi Sureban
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Sripathi Sureban
@en
Sripathi Sureban
@es
Sripathi Sureban
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Sripathi Sureban
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P106
P1153
16048704800
P31
P496
0000-0001-5476-9593