Brief report: Dclk1 deletion in tuft cells results in impaired epithelial repair after radiation injury. - Wikidata - awgldk - TriplyDB

Brief report: Dclk1 deletion in tuft cells results in impaired epithelial repair after radiation injury.

Brief report: Dclk1 deletion in tuft cells results in impaired epithelial repair after radiation injury.

http://www.wikidata.org/entity/Q36155474

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Reserve stem cells: Differentiated cells reprogram to fuel repair, metaplasia, and neoplasia in the adult gastrointestinal tract Dietary Pectin Increases Intestinal Crypt Stem Cell Survival following Radiation Injury Tuft Cells: New Players in Colitis.Optimized multiplex immunofluorescence single-cell analysis reveals tuft cell heterogeneity.Epithelial cell specific Raptor is required for initiation of type 2 mucosal immunity in small intestine Ablation of Doublecortin-Like Kinase 1 in the Colonic Epithelium Exacerbates Dextran Sulfate Sodium-Induced Colitis.High expression of the putative cancer stem cell marker, DCLK1, in rectal neuroendocrine tumors.Epigenetic changes and alternate promoter usage by human colon cancers for expressing DCLK1-isoforms: Clinical Implications Doublecortin-like kinase 1-positive enterocyte - a new cell type in human intestine.Novel regenerative peptide TP508 mitigates radiation-induced gastrointestinal damage by activating stem cells and preserving crypt integrity Elp3 drives Wnt-dependent tumor initiation and regeneration in the intestine.Dclk1+ small intestinal epithelial tuft cells display the hallmarks of quiescence and self-renewal Interleukin-25 Induces Resistance Against Intestinal Trematodes Survival of Patients with Gastrointestinal Cancers Can Be Predicted by a Surrogate microRNA Signature for Cancer Stem-like Cells Marked by DCLK1 Kinase.Intestinal tuft cells regulate the ATM mediated DNA Damage response via Dclk1 dependent mechanism for crypt restitution following radiation injury.Stem cell and progenitor fate in the mammalian intestine: Notch and lateral inhibition in homeostasis and disease.APSA Awardee Submission: Tumor/cancer stem cell marker doublecortin-like kinase 1 in liver diseases.Small molecule kinase inhibitor LRRK2-IN-1 demonstrates potent activity against colorectal and pancreatic cancer through inhibition of doublecortin-like kinase 1.Unique Cellular Lineage Composition of the First Gland of the Mouse Gastric Corpus.Dclk1-expressing tuft cells: critical modulators of the intestinal niche?The Role of Intestinal Stem Cells in Epithelial Regeneration Following Radiation-Induced Gut Injury.Analysis of the clinical significance of DCLK1 colorectal cancer using novel monoclonal antibodies against DCLK1

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Brief report: Dclk1 deletion in tuft cells results in impaired epithelial repair after radiation injury.

http://www.wikidata.org/entity/Q36155474

description

2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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Brief report: Dclk1 deletion i ...... repair after radiation injury.

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Brief report: Dclk1 deletion i ...... repair after radiation injury.

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Brief report: Dclk1 deletion i ...... repair after radiation injury.

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Brief report: Dclk1 deletion i ...... repair after radiation injury

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Courtney W Houchen

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Dongfeng Qu

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Naushad Ali

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Randal May

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Stanley A Lightfoot

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P2860

Alterations of E-cadherin and beta-catenin in gastric cancer

The intestinal epithelium tuft cells: specification and function

Loss of guanylyl cyclase C (GCC) signaling leads to dysfunctional intestinal barrier

Sox9 expression marks a subset of CD24-expressing small intestine epithelial stem cells that form organoids in vitro

Doublecortin and CaM kinase-like-1 and leucine-rich-repeat-containing G-protein-coupled receptor mark quiescent and cycling intestinal stem cells, respectively.

Distinct ATOH1 and Neurog3 requirements define tuft cells as a new secretory cell type in the intestinal epithelium.

Identification of a novel putative gastrointestinal stem cell and adenoma stem cell marker, doublecortin and CaM kinase-like-1, following radiation injury and in adenomatous polyposis coli/multiple intestinal neoplasia mice.

DCAMKL-1 regulates epithelial-mesenchymal transition in human pancreatic cells through a miR-200a-dependent mechanism.

Distinct SOX9 levels differentially mark stem/progenitor populations and enteroendocrine cells of the small intestine epithelium.

Nanoparticle-based delivery of siDCAMKL-1 increases microRNA-144 and inhibits colorectal cancer tumor growth via a Notch-1 dependent mechanism.

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scholarly article

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10.1002/STEM.1566

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3

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32

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Sripathi Sureban

Parthasarathy Chandrakesan

Nathaniel Weygant

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2014-03-01T00:00:00Z

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257756486

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