The M2 gene product of murine gammaherpesvirus 68 is required for efficient colonization of splenic follicles but is not necessary for expansion of latently infected germinal centre B cells.
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Defining immune engagement thresholds for in vivo control of virus-driven lymphoproliferationRole of Src homology domain binding in signaling complexes assembled by the murid γ-herpesvirus M2 protein.Gammaherpesvirus-driven plasma cell differentiation regulates virus reactivation from latently infected B lymphocytes.Inhibition of NF-kappaB activation in vivo impairs establishment of gammaherpesvirus latency.The Gammaherpesvirus m2 protein manipulates the Fyn/Vav pathway through a multidocking mechanism of assemblyThe MHV68 M2 protein drives IL-10 dependent B cell proliferation and differentiationIdentification of closely spaced but distinct transcription initiation sites for the murine gammaherpesvirus 68 latency-associated M2 gene.A single CD8+ T cell epitope sets the long-term latent load of a murid herpesvirusVirus-encoded microRNAs facilitate gammaherpesvirus latency and pathogenesis in vivoCharacterization of a novel wood mouse virus related to murid herpesvirus 4.A gammaherpesvirus cooperates with interferon-alpha/beta-induced IRF2 to halt viral replication, control reactivation, and minimize host lethality.Activation of Vav by the gammaherpesvirus M2 protein contributes to the establishment of viral latency in B lymphocytesChemokine binding protein M3 of murine gammaherpesvirus 68 modulates the host response to infection in a natural hostEstablishment of murine gammaherpesvirus latency in B cells is not a stochastic event.Type I Interferons Direct Gammaherpesvirus Host ColonizationNF-kappaB p50 plays distinct roles in the establishment and control of murine gammaherpesvirus 68 latency.Murid Gammaherpesvirus Latency-Associated Protein M2 Promotes the Formation of Conjugates between Transformed B Lymphoma Cells and T Helper Cells.Use of a virus-encoded enzymatic marker reveals that a stable fraction of memory B cells expresses latency-associated nuclear antigen throughout chronic gammaherpesvirus infection.Deregulation of DNA damage signal transduction by herpesvirus latency-associated M2.Role for MyD88 signaling in murine gammaherpesvirus 68 latency.Gamma-herpesvirus colonization of the spleen requires lytic replication in B cells.
P2860
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P2860
The M2 gene product of murine gammaherpesvirus 68 is required for efficient colonization of splenic follicles but is not necessary for expansion of latently infected germinal centre B cells.
description
2004 nî lūn-bûn
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2004年の論文
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2004年論文
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name
The M2 gene product of murine ...... ected germinal centre B cells.
@en
The M2 gene product of murine ...... ected germinal centre B cells.
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type
label
The M2 gene product of murine ...... ected germinal centre B cells.
@en
The M2 gene product of murine ...... ected germinal centre B cells.
@nl
prefLabel
The M2 gene product of murine ...... ected germinal centre B cells.
@en
The M2 gene product of murine ...... ected germinal centre B cells.
@nl
P50
P356
P1476
The M2 gene product of murine ...... ected germinal centre B cells.
@en
P2093
Heiko Adler
Stacey Efstathiou
P304
P356
10.1099/VIR.0.80138-0
P407
P577
2004-10-01T00:00:00Z