Involvement of mitochondrial permeability transition in acetaminophen-induced liver injury in mice.
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Towards a unifying, systems biology understanding of large-scale cellular death and destruction caused by poorly liganded iron: Parkinson's, Huntington's, Alzheimer's, prions, bactericides, chemical toxicology and others as examplesMechanisms of acetaminophen-induced liver necrosisExperimental models of hepatotoxicity related to acute liver failureAcetaminophen-induced Liver Injury: from Animal Models to HumansNovel in vitro and mathematical models for the prediction of chemical toxicityEvidence-based selection of training compounds for use in the mechanism-based integrated prediction of drug-induced liver injury in manAcetaminophen induces apoptosis in rat cortical neuronsEthanol and Acetaminophen Synergistically Induce Hepatic Aggregation and TCH346-Insensitive Nuclear Translocation of GAPDHLong-lived ames dwarf mice are resistant to chemical stressorsDrug-Induced Liver Injury: Cascade of Events Leading to Cell Death, Apoptosis or NecrosisMechanisms of acetaminophen hepatotoxicity and their translation to the human pathophysiology.Acute and chronic effects of IL-22 on acetaminophen-induced liver injuryStress resistance and aging: influence of genes and nutrition.Acetaminophen induces human neuroblastoma cell death through NFKB activation.Ozagrel hydrochloride, a selective thromboxane A₂ synthase inhibitor, alleviates liver injury induced by acetaminophen overdose in mice.Protective effects from Houttuynia cordata aqueous extract against acetaminophen-induced liver injury.Role of the mitochondrion in programmed necrosisCurrent issues with acetaminophen hepatotoxicity--a clinically relevant model to test the efficacy of natural products.Proteomic analysis of acetaminophen-induced changes in mitochondrial protein expression using spectral countingPleurotus ostreatus opposes mitochondrial dysfunction and oxidative stress in acetaminophen-induced hepato-renal injury.Acetaminophen hepatotoxicity and HIF-1α induction in acetaminophen toxicity in mice occurs without hypoxia.Hepatoprotective role of liver fatty acid binding protein in acetaminophen induced toxicity.Hepatic proteome analysis of Atlantic salmon (Salmo salar) after exposure to environmental concentrations of human pharmaceuticals.Lower susceptibility of female mice to acetaminophen hepatotoxicity: Role of mitochondrial glutathione, oxidant stress and c-jun N-terminal kinase.Role of TRAIL and the pro-apoptotic Bcl-2 homolog Bim in acetaminophen-induced liver damage.Evaluation of the contribution of multiple DAMPs and DAMP receptors in cell death-induced sterile inflammatory responses.Hepatitis C virus structural proteins can exacerbate or ameliorate acetaminophen-induced liver injury in mice.Bee venom phospholipase A2 protects against acetaminophen-induced acute liver injury by modulating regulatory T cells and IL-10 in mice.Resveratrol prevents protein nitration and release of endonucleases from mitochondria during acetaminophen hepatotoxicity.Changes in mouse liver protein glutathionylation after acetaminophen exposure.The mitochondrial death pathway: a promising therapeutic target in diseasesThe mechanism underlying acetaminophen-induced hepatotoxicity in humans and mice involves mitochondrial damage and nuclear DNA fragmentation.Cell death and cell death responses in liver disease: mechanisms and clinical relevanceNovel protective mechanisms for S-adenosyl-L-methionine against acetaminophen hepatotoxicity: improvement of key antioxidant enzymatic function.Strategies, models and biomarkers in experimental non-alcoholic fatty liver disease researchThe Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology.Prediction of liver injury induced by chemicals in human with a multiparametric assay on isolated mouse liver mitochondria.Intracellular signaling mechanisms of acetaminophen-induced liver cell death.Lysosomal instability and cathepsin B release during acetaminophen hepatotoxicity.Complement 1q-binding protein inhibits the mitochondrial permeability transition pore and protects against oxidative stress-induced death.
P2860
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P2860
Involvement of mitochondrial permeability transition in acetaminophen-induced liver injury in mice.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Involvement of mitochondrial p ...... -induced liver injury in mice.
@en
Involvement of mitochondrial p ...... -induced liver injury in mice.
@nl
type
label
Involvement of mitochondrial p ...... -induced liver injury in mice.
@en
Involvement of mitochondrial p ...... -induced liver injury in mice.
@nl
prefLabel
Involvement of mitochondrial p ...... -induced liver injury in mice.
@en
Involvement of mitochondrial p ...... -induced liver injury in mice.
@nl
P2093
P1476
Involvement of mitochondrial p ...... -induced liver injury in mice.
@en
P2093
Chieko Suda
Toshiharu Horie
Yasuhiro Masubuchi
P304
P356
10.1016/J.JHEP.2004.09.015
P577
2005-01-01T00:00:00Z