about
Proper protein glycosylation promotes mitogen-activated protein kinase signal fidelityGlutathione biosynthesis is a metabolic vulnerability in PI(3)K/Akt-driven breast cancer.Oncogenic AKT1(E17K) mutation induces mammary hyperplasia but prevents HER2-driven tumorigenesis.Selenoprotein H is an essential regulator of redox homeostasis that cooperates with p53 in development and tumorigenesisPhosphoinositide 3-Kinase Regulates Glycolysis through Mobilization of Aldolase from the Actin Cytoskeleton.MSC-regulated microRNAs converge on the transcription factor FOXP2 and promote breast cancer metastasis.Metabolic Reprogramming by the PI3K-Akt-mTOR Pathway in Cancer.Yap reprograms glutamine metabolism to increase nucleotide biosynthesis and enable liver growthInhibition of Rb Phosphorylation Leads to mTORC2-Mediated Activation of Akt.Oncogenic PI3K promotes methionine dependency in breast cancer cells through the cystine-glutamate antiporter xCT.
P50
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P50
description
hulumtues
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researcher
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wetenschapper
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հետազոտող
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name
Evan C Lien
@ast
Evan C Lien
@en
Evan C Lien
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Evan C Lien
@nl
Evan C Lien
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type
label
Evan C Lien
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Evan C Lien
@en
Evan C Lien
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Evan C Lien
@nl
Evan C Lien
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prefLabel
Evan C Lien
@ast
Evan C Lien
@en
Evan C Lien
@es
Evan C Lien
@nl
Evan C Lien
@sl
P106
P21
P31
P496
0000-0001-7866-4761