In-depth analysis of spatial cognition in Williams syndrome: A critical assessment of the role of the LIMK1 gene.
about
Induced chromosome deletions cause hypersociability and other features of Williams-Beuren syndrome in mice.Challenging the use of adult neuropsychological models for explaining neurodevelopmental disorders: developed versus developing brainsAuditory attraction: activation of visual cortex by music and sound in Williams syndrome.Bridging the gene-behavior divide through neuroimaging deletion syndromes: Velocardiofacial (22q11.2 Deletion) and Williams (7q11.23 Deletion) syndromes.Visual phenotype in Williams-Beuren syndrome challenges magnocellular theories explaining human neurodevelopmental visual cortical disordersDefining the social phenotype in Williams syndrome: a model for linking gene, the brain, and behavior.Genetic contributions to visuospatial cognition in Williams syndrome: insights from two contrasting partial deletion patientsMapping genetically controlled neural circuits of social behavior and visuo-motor integration by a preliminary examination of atypical deletions with Williams syndromeAutistic disorder in patients with Williams-Beuren syndrome: a reconsideration of the Williams-Beuren syndrome phenotype.An atypical 7q11.23 deletion in a normal IQ Williams-Beuren syndrome patient.BDNF repairs podocyte damage by microRNA-mediated increase of actin polymerization.LIMK1 regulates long-term memory and synaptic plasticity via the transcriptional factor CREB.Genomic sister-disorders of neurodevelopment: an evolutionary approachSocial cognition in williams syndrome: genotype/phenotype insights from partial deletion patientsActin Out: Regulation of the Synaptic Cytoskeleton.Microdomains in forebrain spines: an ultrastructural perspective.Language abilities in Williams syndrome: a critical review.Inhibition of versican expression by siRNA facilitates tropoelastin synthesis and elastic fiber formation by human SK-LMS-1 leiomyosarcoma smooth muscle cells in vitro and in vivo.Insights into brain development from neurogenetic syndromes: evidence from fragile X syndrome, Williams syndrome, Turner syndrome and velocardiofacial syndrome.Roles of LIM kinases in central nervous system function and dysfunction.A conserved region in the 3' untranslated region of the human LIMK1 gene is critical for proper expression of LIMK1 at the post-transcriptional level.N-cofilin is associated with neuronal migration disorders and cell cycle control in the cerebral cortex.A battery for the assessment of visuo-spatial abilities involved in drawing tasks.The development of route learning in Down syndrome, Williams syndrome and typical development: investigations with virtual environments.Inefficient search of large-scale space in Williams syndrome: further insights on the role of LIMK1 deletion in deficits of spatial cognition.
P2860
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P2860
In-depth analysis of spatial cognition in Williams syndrome: A critical assessment of the role of the LIMK1 gene.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
In-depth analysis of spatial c ...... of the role of the LIMK1 gene.
@en
In-depth analysis of spatial c ...... of the role of the LIMK1 gene.
@nl
type
label
In-depth analysis of spatial c ...... of the role of the LIMK1 gene.
@en
In-depth analysis of spatial c ...... of the role of the LIMK1 gene.
@nl
prefLabel
In-depth analysis of spatial c ...... of the role of the LIMK1 gene.
@en
In-depth analysis of spatial c ...... of the role of the LIMK1 gene.
@nl
P2093
P1433
P1476
In-depth analysis of spatial c ...... of the role of the LIMK1 gene.
@en
P2093
Elaine Funnell
May Tassabehji
Victoria Gray
P304
P356
10.1016/J.NEUROPSYCHOLOGIA.2005.08.007
P577
2005-10-10T00:00:00Z