GSK-3β overexpression causes reversible alterations on postsynaptic densities and dendritic morphology of hippocampal granule neurons in vivo.
about
Glycogen Synthase Kinase-3 (GSK-3)-Targeted Therapy and ImagingMolecular and biochemical trajectories from diabetes to Alzheimer's disease: A critical appraisalWnt signaling in neuropsychiatric disorders: ties with adult hippocampal neurogenesis and behaviorReelin Regulates the Maturation of Dendritic Spines, Synaptogenesis and Glial Ensheathment of Newborn Granule Cells.Temporal dynamics of hippocampal neurogenesis in chronic neurodegeneration.Deferoxamine attenuates lipopolysaccharide-induced neuroinflammation and memory impairment in mice.Peripherally triggered and GSK-3β-driven brain inflammation differentially skew adult hippocampal neurogenesis, behavioral pattern separation and microglial activation in response to ibuprofen.Inhibition of glycogen synthase kinase-3β enhances cognitive recovery after stroke: the role of TAK1.The Ever-Changing Morphology of Hippocampal Granule Neurons in Physiology and PathologyRole of neuroinflammation in adult neurogenesis and Alzheimer disease: therapeutic approachesMicroglia regulate hippocampal neurogenesis during chronic neurodegenerationThe role of dopamine signaling in epileptogenesis.GSK-3β and MMP-9 Cooperate in the Control of Dendritic Spine MorphologyAcetaminophen attenuates lipopolysaccharide-induced cognitive impairment through antioxidant activity.Forced swim test induces divergent global transcriptomic alterations in the hippocampus of high versus low novelty-seeker rats.The Age of Cortical Neural Networks Affects Their Interactions with Magnetic Nanoparticles.GSK-3β Overexpression Alters the Dendritic Spines of Developmentally Generated Granule Neurons in the Mouse Hippocampal Dentate GyrusNew insights into the role of glycogen synthase kinase-3 in Alzheimer's disease.Glycogen synthase kinase 3 beta (GSK3β) at the tip of neuronal development and regeneration.GSK-3β, a pivotal kinase in Alzheimer disease.Selective alterations of neurons and circuits related to early memory loss in Alzheimer's disease.Absence of CX3CR1 impairs the internalization of Tau by microglia.Novel function of Tau in regulating the effects of external stimuli on adult hippocampal neurogenesis.Retroviral induction of GSK-3β expression blocks the stimulatory action of physical exercise on the maturation of newborn neurons.Direct Evidence of Internalization of Tau by Microglia In Vitro and In Vivo.Versatile use of rtTA-expressing retroviruses in the study of neurodegeneration.Andrographolide Stimulates Neurogenesis in the Adult Hippocampus.Soluble Tau has devastating effects on the structural plasticity of hippocampal granule neurons.Icariin Attenuates Synaptic and Cognitive Deficits in an Aβ1-42-Induced Rat Model of Alzheimer's Disease.Neurons derived from sporadic Alzheimer's disease iPSCs reveal elevated TAU hyperphosphorylation, increased amyloid levels, and GSK3B activation.Our Working Point of View of Tau Protein.Three-dimensional analysis of synapses in the transentorhinal cortex of Alzheimer's disease patients.
P2860
Q26768475-EA028953-373E-4D1C-BA3C-60536AA20802Q26783344-011E9CAA-4477-4F2D-93F5-2D83F9CF1AE1Q27008843-687E7160-E2A0-4595-A0F0-D4DF6E3162D5Q30823270-485D0588-6D57-4AAD-955D-18D06BE6F03CQ33937526-B9B9635E-ED14-48C4-BC26-FDFC2F79344FQ35068436-62B45528-08F1-49CC-B18A-C1CE47F032FCQ35150206-EDFD0301-5895-4125-B075-595F8E743E8BQ35773229-2BC9A03D-3CDF-449D-B767-252997EF048DQ36475393-DCC1E1E5-2E23-4DAC-8C0D-0F4DAEA45041Q36830938-C21392E3-1205-4F97-86EF-10136EFF1F75Q37003405-ED1F807B-9EBA-4243-9432-35B51C46A830Q37176694-DF5A91D2-D2A9-4982-9EA1-6ACF64CBA984Q37569915-C6A196C3-88A6-4359-ADE8-2F8BE81B8B7AQ37597304-589C6290-F787-4814-89E8-09D91222281BQ37615811-21BF97B4-B941-4443-8862-71C881134CF8Q37634348-76247814-1DF3-4890-BB3C-E244AD63D15BQ37692033-5F97EAF7-314E-4313-996C-1ED9DBCA2EB2Q38149943-B46FF843-C096-4D89-97E6-1E8AD73F2C5BQ38155462-0EE99EDB-DCB0-47C2-A0BD-7C415BB6D1A3Q38218085-E5E1935F-EB8F-47E8-A09D-2A3AE9CDF011Q38218089-1C624766-ACDC-4469-8FB5-3BD7CFBB5E07Q38623440-225D69F6-B61E-4242-90FB-EDC6BBC1FCA3Q39752041-658AE152-D6A7-44C4-869D-B86A89E93739Q39891638-7A6ECA5A-E1D0-4776-943B-124FF09CC7BBQ40240888-8CD13844-72A5-4A32-B707-2E3B2DB59D5EQ42315744-4F38F5A0-09C0-44F0-BE20-46788D1CA6D8Q43106553-2711A73A-5BCD-4BFE-AB82-871316E64977Q46245654-ECE535EC-684C-4C4F-B3F8-936A0B1C11A3Q47159915-AF91A76E-5852-4B3F-B990-F6D9DAE684CEQ47345806-C2EC409A-CC59-4BEF-A64F-CCBE17CD7FE2Q47646456-404E4D6A-B254-45A1-84D3-92199F54D584Q50533053-34022731-0976-48C9-B430-BC983E4FA3A0
P2860
GSK-3β overexpression causes reversible alterations on postsynaptic densities and dendritic morphology of hippocampal granule neurons in vivo.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
GSK-3β overexpression causes r ...... ampal granule neurons in vivo.
@en
GSK-3β overexpression causes r ...... ampal granule neurons in vivo.
@nl
type
label
GSK-3β overexpression causes r ...... ampal granule neurons in vivo.
@en
GSK-3β overexpression causes r ...... ampal granule neurons in vivo.
@nl
prefLabel
GSK-3β overexpression causes r ...... ampal granule neurons in vivo.
@en
GSK-3β overexpression causes r ...... ampal granule neurons in vivo.
@nl
P2093
P2860
P50
P356
P1433
P1476
GSK-3β overexpression causes r ...... campal granule neurons in vivo
@en
P2093
A Fuster-Matanzo
J Jurado-Arjona
P2860
P2888
P304
P356
10.1038/MP.2013.4
P407
P577
2013-02-12T00:00:00Z