Comparison of inflammatory and acute-phase responses in the brain and peripheral organs of the ME7 model of prion disease - Wikidata - awgldk - TriplyDB

Comparison of inflammatory and acute-phase responses in the brain and peripheral organs of the ME7 model of prion disease

Comparison of inflammatory and acute-phase responses in the brain and peripheral organs of the ME7 model of prion disease

http://www.wikidata.org/entity/Q42709847

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Intracerebral administration of interleukin-12 (IL-12) and IL-18 modifies the course of mouse scrapie Microglia and the urokinase plasminogen activator receptor/uPA system in innate brain inflammation.Presence of an acute phase response in sheep with clinical classical scrapie.Contribution of complement activation pathways to neuropathology differs among mouse models of Alzheimer's disease.Endogenous IL-1 in cognitive function and anxiety: a study in IL-1RI-/- mice Exacerbation of CNS inflammation and neurodegeneration by systemic LPS treatment is independent of circulating IL-1β and IL-6.Prion infection of mouse brain reveals multiple new upregulated genes involved in neuroinflammation or signal transduction.Osteoarthritis accelerates and exacerbates Alzheimer's disease pathology in mice.Interdependent and independent roles of type I interferons and IL-6 in innate immune, neuroinflammatory and sickness behaviour responses to systemic poly I:C.Prions and their lethal journey to the brain.CSF biomarkers in delirium: a systematic review.Sequential activation of microglia and astrocyte cytokine expression precedes increased Iba-1 or GFAP immunoreactivity following systemic immune challenge.A critical analysis of disease-associated DNA polymorphisms in the genes of cattle, goat, sheep, and pig.Defining the Microglia Response during the Time Course of Chronic Neurodegeneration Systemic inflammation induces acute behavioral and cognitive changes and accelerates neurodegenerative disease.Analysis of the hippocampal proteome in ME7 prion disease reveals a predominant astrocytic signature and highlights the brain-restricted production of clusterin in chronic neurodegeneration.The role of microglia in synaptic stripping and synaptic degeneration: a revised perspective.Inflammation and adaptive immunity in Parkinson's disease.Microglia and neurodegeneration: the role of systemic inflammation.Prion disease and the innate immune system.β-Adrenergic receptor antagonism prevents anxiety-like behavior and microglial reactivity induced by repeated social defeat Peripheral Inflammation and Demyelinating Diseases.Parkinson's disease and systemic inflammation.

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Comparison of inflammatory and acute-phase responses in the brain and peripheral organs of the ME7 model of prion disease

http://www.wikidata.org/entity/Q42709847

description

2005 nî lūn-bûn

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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2005年论文

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2005年论文

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name

Comparison of inflammatory and ...... the ME7 model of prion disease

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Comparison of inflammatory and ...... the ME7 model of prion disease

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Comparison of inflammatory and ...... the ME7 model of prion disease

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Comparison of inflammatory and ...... the ME7 model of prion disease

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Comparison of inflammatory and ...... the ME7 model of prion disease

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Comparison of inflammatory and ...... the ME7 model of prion disease

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JVI.79.8.5174-5184.2005

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Journal of Virology

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Comparison of inflammatory and ...... the ME7 model of prion disease

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V Hugh Perry

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P2860

CINC-1 is an acute-phase protein induced by focal brain injury causing leukocyte mobilization and liver injury

Inducible cytokine gene expression in the brain in the ME7/CV mouse model of scrapie is highly restricted, is at a strikingly low level relative to the degree of gliosis and occurs only late in disease.

TGF-beta reduced binding of high-density lipoproteins in murine macrophages and macrophage-derived foam cells.

Cytokines and the hepatic acute-phase response.

Mannose receptor and scavenger receptor: two macrophage pattern recognition receptors with diverse functions in tissue homeostasis and host defense.

Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice.

Quantification of mRNA using real-time reverse transcription PCR (RT-PCR): trends and problems.

Biochemical and functional characterization of the interaction between pentraxin 3 and C1q.

Prion pathogenesis in the absence of Toll-like receptor signalling

Multimer formation and ligand recognition by the long pentraxin PTX3. Similarities and differences with the short pentraxins C-reactive protein and serum amyloid P component.

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5174-5184

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10.1128/JVI.79.8.5174-5184.2005

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David C. Wilcockson

Colm Cunningham

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15795301

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Prion protein family

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1069550

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