Treatment-recalcitrant chronic rhinosinusitis with polyps is associated with altered epithelial cell expression of interleukin-33. - Wikidata - awgldk - TriplyDB

Treatment-recalcitrant chronic rhinosinusitis with polyps is associated with altered epithelial cell expression of interleukin-33.

Treatment-recalcitrant chronic rhinosinusitis with polyps is associated with altered epithelial cell expression of interleukin-33.

http://www.wikidata.org/entity/Q42733194

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Role of innate immunity in the pathogenesis of chronic rhinosinusitis: progress and new avenues Interleukin-33 in asthma: how big of a role does it play?Chronic rhinosinusitis: therapeutic efficacy of anti-inflammatory and antibiotic approaches.IL-33-responsive innate lymphoid cells are an important source of IL-13 in chronic rhinosinusitis with nasal polyps ICON: chronic rhinosinusitis.Chronic rhinosinusitis as a multifactorial inflammatory disorder.Pathogenesis of nasal polyposis Expression of the receptor for advanced glycation end products, a target for high mobility group box 1 protein, and its role in chronic recalcitrant rhinosinusitis with nasal polyps.The Induction of IL-33 in the Sinus Epithelium and Its Influence on T-Helper Cell Responses.Barrier function of the nasal mucosa in health and type-2 biased airway diseases.Aspirin-Exacerbated Respiratory Disease Involves a Cysteinyl Leukotriene-Driven IL-33-Mediated Mast Cell Activation Pathway IL-25/IL-33-responsive TH2 cells characterize nasal polyps with a default TH17 signature in nasal mucosa.Genetics of chronic rhinosinusitis: state of the field and directions forward Immunopathology features of chronic rhinosinusitis in high-altitude dwelling Tibetans.Damage-associated molecular patterns stimulate interleukin-33 expression in nasal polyp epithelial cells.Type 2 innate immune responses and the natural helper cell.Interleukin-33 biology with potential insights into human diseases.Gene expression signatures: a new approach to understanding the pathophysiology of chronic rhinosinusitis.The role of interleukin-33 in rhinitis.Clinical and biological markers of difficult-to-treat severe chronic rhinosinusitis.Biomarkers in the evaluation and management of chronic rhinosinusitis with nasal polyposis.International Consensus Statement on Allergy and Rhinology: Rhinosinusitis.Is there a future for biologics in the management of chronic rhinosinusitis?General antibiotic exposure is associated with increased risk of developing chronic rhinosinusitis.Sinonasal epithelial cell response to Staphylococcus aureus burden in chronic rhinosinusitis.IL-33: biological properties, functions, and roles in airway disease.Clinical severity and epithelial endotypes in chronic rhinosinusitis.The effect of nasal irrigation formulation on the antimicrobial activity of nasal secretions.Dynamic role of epithelium-derived cytokines in asthma The mouse interleukin (Il)33 gene is expressed in a cell type- and stimulus-dependent manner from two alternative promoters.P-glycoprotein functions as an immunomodulator in healthy human primary nasal epithelial cells.Eosinophilic rhinosinusitis is not a disease of ostiomeatal occlusion.IL-33 Expression in Chronic Rhinosinusitis with Nasal Polyps and Its Relationship with Clinical Severity.Type 2 Cysteinyl Leukotriene Receptors Drive IL-33-Dependent Type 2 Immunopathology and Aspirin Sensitivity.Alternatively activated macrophages and impaired phagocytosis of S. aureus in chronic rhinosinusitis.Subtyping of polyposis nasi: phenotypes, endotypes and comorbidities.Tumor necrosis factor-α regulates interleukin-33 expression through extracellular signal-regulated kinase, p38, and nuclear factor-κB pathways in airway epithelial cells.Activation of group 2 innate lymphoid cells exacerbates and confers corticosteroid resistance to mouse nasal type 2 inflammation.IL-1β, IL-4 and IL-12 control the fate of group 2 innate lymphoid cells in human airway inflammation in the lungs.Expression of IL-33 and its receptor ST2 in chronic rhinosinusitis with nasal polyps.

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P2860

Treatment-recalcitrant chronic rhinosinusitis with polyps is associated with altered epithelial cell expression of interleukin-33.

http://www.wikidata.org/entity/Q42733194

description

2010 nî lūn-bûn

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name

Treatment-recalcitrant chronic ...... expression of interleukin-33.

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Treatment-recalcitrant chronic ...... expression of interleukin-33.

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Treatment-recalcitrant chronic ...... expression of interleukin-33.

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Treatment-recalcitrant chronic ...... expression of interleukin-33.

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Douglas D Reh

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Murugappan Ramanathan

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Yadong Wang

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P2860

IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines

A novel IL-1 family cytokine, IL-33, potently activates human eosinophils

Up-regulation of Toll-like receptors 2, 3 and 4 in allergic rhinitis.

Bacterial biofilms: do they play a role in chronic sinusitis?

Biofilm formation by Staphylococcus aureus and Pseudomonas aeruginosa is associated with an unfavorable evolution after surgery for chronic sinusitis and nasal polyposis.

Adult chronic rhinosinusitis: definitions, diagnosis, epidemiology, and pathophysiology.

Superantigens and nasal polyps.

Recurrence rates after endoscopic sinus surgery for massive sinus polyposis.

The role of ubiquitous airborne fungi in chronic rhinosinusitis.

Innate and acquired immunity and epithelial cell function in chronic rhinosinusitis.

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