In vitro degradation of endothelial catenins by a neutrophil protease.
about
fMLP-stimulated neutrophils increase endothelial [Ca2+](i) and microvessel permeability in the absence of adhesion: role of reactive oxygen speciesLeucocyte/endothelium interactions and microvessel permeability: coupled or uncoupled?Cortactin deficiency is associated with reduced neutrophil recruitment but increased vascular permeability in vivoNeutrophil recruitment under shear flow: it's all about endothelial cell rings and gaps.Relocalization of endothelial cell beta-catenin after coculture with activated neutrophils from patients undergoing cardiac surgery with cardiopulmonary bypass.Blockade of neutrophil elastase attenuates severe liver injury in hepatitis B transgenic miceMyeloid-specific deletion of tumor suppressor PTEN augments neutrophil transendothelial migration during inflammation.Monocytes induce reversible focal changes in vascular endothelial cadherin complex during transendothelial migration under flow.Mechanisms of neutrophil transendothelial migrationThe inhibition of neutrophil elastase ameliorates mouse liver damage due to ischemia and reperfusion.Neutrophil and monocyte recruitment by PECAM, CD99, and other molecules via the LBRCRegulation of RhoA activity by adhesion molecules and mechanotransduction.VE-PTP maintains the endothelial barrier via plakoglobin and becomes dissociated from VE-cadherin by leukocytes and by VEGF.PMN transendothelial migration decreases nuclear NFkappaB in IL-1beta-activated endothelial cells: role of PECAM-1.Locking endothelial junctions blocks leukocyte extravasation, but not in all tissues.Tumor necrosis factor-alpha-induced leukocyte adhesion and microvessel permeability.Protection against acute lung injury by intravenous or intratracheal pretreatment with EPI-HNE-4, a new potent neutrophil elastase inhibitor.Src-dependent, neutrophil-mediated vascular hyperpermeability and beta-catenin modification.Proteolysis of enteric cell villin by Entamoeba histolytica cysteine proteinases.fMLP-stimulated release of reactive oxygen species from adherent leukocytes increases microvessel permeability.Decreased expression of VE-cadherin and claudin-5 and increased phosphorylation of VE-cadherin in vascular endothelium in nasal polyps.Proteolytic cleavage of beta-catenin by caspases: an in vitro analysis.Endothelial paxillin and focal adhesion kinase (FAK) play a critical role in neutrophil transmigration.Transference of recombinant VE-cadherin cytoplasmic domain alters endothelial junctional integrity and porcine microvascular permeability.FAK and PAX-illin get involved in leukocyte diapedesis.Decreased distribution of lung epithelial junction proteins after intratracheal antigen or lipopolysaccharide challenge: correlation with neutrophil influx and levels of BALF sE-cadherin.
P2860
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P2860
In vitro degradation of endothelial catenins by a neutrophil protease.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
In vitro degradation of endothelial catenins by a neutrophil protease.
@en
In vitro degradation of endothelial catenins by a neutrophil protease.
@nl
type
label
In vitro degradation of endothelial catenins by a neutrophil protease.
@en
In vitro degradation of endothelial catenins by a neutrophil protease.
@nl
prefLabel
In vitro degradation of endothelial catenins by a neutrophil protease.
@en
In vitro degradation of endothelial catenins by a neutrophil protease.
@nl
P2860
P356
P1476
In vitro degradation of endothelial catenins by a neutrophil protease.
@en
P2093
P2860
P304
P356
10.1083/JCB.140.2.403
P407
P577
1998-01-01T00:00:00Z