Effects of charybdotoxin, a blocker of Ca2+-activated K+ channels, on motor nerve terminals
about
Altered properties of quantal neurotransmitter release at endplates of mice lacking P/Q-type Ca2+ channelsThe mechanism for prejunctional enhancement of neuromuscular transmission by ethanol in the mouse.Estimation of presynaptic calcium currents and endogenous calcium buffers at the frog neuromuscular junction with two different calcium fluorescent dyes.Hexamethonium- and methyllycaconitine-induced changes in acetylcholine release from rat motor nerve terminalsReversal by cysteine of the cadmium-induced block of skeletal neuromuscular transmission in vitro.Effects of brevetoxin-B on motor nerve terminals of mouse skeletal muscle.Transmitter-mediated local contracture of the endplate region of the focally innervated mouse diaphragm treated with anticholinesterase.Selective disruption of the mammalian secretory apparatus enhances or eliminates calcium current modulation in nerve endingsPotassium channel toxins and transmitter release.Neuropharmacology of potassium ion channels.Effects of Ca2+ channel blocker neurotoxins on transmitter release and presynaptic currents at the mouse neuromuscular junction.Apparent block of K+ currents in mouse motor nerve terminals by tetrodotoxin, mu-conotoxin and reduced external sodium.Effects of tacrine, velnacrine (HP029), suronacrine (HP128), and 3,4-diaminopyridine on skeletal neuromuscular transmission in vitro.On the blockade of acetylcholine release at mouse motor nerve terminals by beta-bungarotoxin and crotoxin.Decrease in calcium currents induced by aminoglycoside antibiotics in frog motor nerve endings.Modulation of acetylcholine release at mouse neuromuscular junctions by interaction of three homologous scorpion toxins with K+ channelsPresynaptic BK channels control transmitter release: physiological relevance and potential therapeutic implications.Antagonism of calcium currents and neurotransmitter release by barium ions at frog motor nerve endings.Modulation of presynaptic Ca(2+) currents in frog motor nerve terminals by high pressure.Red blood cell K+ could be a marker of K+ changes in other cells involved in blood pressure regulation.Adenosine decreases both presynaptic calcium currents and neurotransmitter release at the mouse neuromuscular junction.Modulation of calcium currents is eliminated after cleavage of a strategic component of the mammalian secretory apparatus.Single-channel properties of BK-type calcium-activated potassium channels at a cholinergic presynaptic nerve terminal.Effects of Ca2+ channel blockers on transmitter release and presynaptic currents at the frog neuromuscular junction
P2860
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P2860
Effects of charybdotoxin, a blocker of Ca2+-activated K+ channels, on motor nerve terminals
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
1988年论文
@zh
1988年论文
@zh-cn
name
Effects of charybdotoxin, a bl ...... nels, on motor nerve terminals
@en
Effects of charybdotoxin, a bl ...... nels, on motor nerve terminals
@nl
type
label
Effects of charybdotoxin, a bl ...... nels, on motor nerve terminals
@en
Effects of charybdotoxin, a bl ...... nels, on motor nerve terminals
@nl
prefLabel
Effects of charybdotoxin, a bl ...... nels, on motor nerve terminals
@en
Effects of charybdotoxin, a bl ...... nels, on motor nerve terminals
@nl
P2093
P2860
P1476
Effects of charybdotoxin, a bl ...... nels, on motor nerve terminals
@en
P2093
A J Anderson
A L Harvey
P N Strong
P2860
P304
P356
10.1111/J.1476-5381.1988.TB11772.X
P407
P577
1988-12-01T00:00:00Z