about
The murine G+C-rich promoter binding protein mGPBP is required for promoter-specific transcriptionPITSLRE p110 protein kinases associate with transcription complexes and affect their activitySpecificity in Toll-like receptor signalling through distinct effector functions of TRAF3 and TRAF6A NOD2-NALP1 complex mediates caspase-1-dependent IL-1beta secretion in response to Bacillus anthracis infection and muramyl dipeptideActivation of liver X receptors and retinoid X receptors prevents bacterial-induced macrophage apoptosis.A five-amino-acid motif in the undefined region of the TLR8 ectodomain is required for species-specific ligand recognition.The ubiquitin ligase ZNRF1 promotes caveolin-1 ubiquitination and degradation to modulate inflammation.Development of CpG-oligodeoxynucleotides for effective activation of rabbit TLR9 mediated immune responses.The pore-forming toxin β hemolysin/cytolysin triggers p38 MAPK-dependent IL-10 production in macrophages and inhibits innate immunity.TNFalpha induces ABCA1 through NF-kappaB in macrophages and in phagocytes ingesting apoptotic cells.Functional interaction of heat shock protein 90 and Beclin 1 modulates Toll-like receptor-mediated autophagy.NF-kappaB is a negative regulator of IL-1beta secretion as revealed by genetic and pharmacological inhibition of IKKbeta.I{kappa}B kinase (IKK){beta}, but not IKK{alpha}, is a critical mediator of osteoclast survival and is required for inflammation-induced bone loss.IL-1β-driven neutrophilia preserves antibacterial defense in the absence of the kinase IKKβStreptolysin O promotes group A Streptococcus immune evasion by accelerated macrophage apoptosis.TLR-induced PAI-2 expression suppresses IL-1β processing via increasing autophagy and NLRP3 degradationAssociation of STAT6 genetic variants with childhood atopic dermatitis in Taiwanese population.The protein kinase PKR is required for macrophage apoptosis after activation of Toll-like receptor 4.Nod2 mutation in Crohn's disease potentiates NF-kappaB activity and IL-1beta processing.Natural Modulators of Endosomal Toll-Like Receptor-Mediated Psoriatic Skin Inflammation.Gene expression of P-glycoprotein and cytochrome P450 3A4 in peripheral blood mononuclear cells and correlation with expression in liver.Involvement of M1 Macrophage Polarization in Endosomal Toll-Like Receptors Activated Psoriatic Inflammation
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Li-chung Hsu
@ast
Li-chung Hsu
@en
Li-chung Hsu
@es
Li-chung Hsu
@nl
Li-chung Hsu
@sl
type
label
Li-chung Hsu
@ast
Li-chung Hsu
@en
Li-chung Hsu
@es
Li-chung Hsu
@nl
Li-chung Hsu
@sl
altLabel
徐立中
@en
prefLabel
Li-chung Hsu
@ast
Li-chung Hsu
@en
Li-chung Hsu
@es
Li-chung Hsu
@nl
Li-chung Hsu
@sl
P106
P1153
7401644689
P31
P496
0000-0001-7206-5231