Aerobic exercise training improves skeletal muscle function and Ca2+ handling-related protein expression in sympathetic hyperactivity-induced heart failure.
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Aerobic Exercise and Pharmacological Therapies for Skeletal Myopathy in Heart Failure: Similarities and DifferencesExercise training prevents oxidative stress and ubiquitin-proteasome system overactivity and reverse skeletal muscle atrophy in heart failureAldehyde dehydrogenase 2 activation in heart failure restores mitochondrial function and improves ventricular function and remodelling.Differential regulation of the fiber type-specific gene expression of the sarcoplasmic reticulum calcium-ATPase isoforms induced by exercise training.Action potential-evoked calcium release is impaired in single skeletal muscle fibers from heart failure patients.Combined effect of AMPK/PPAR agonists and exercise training in mdx mice functional performance.Effects of exercise training on neurovascular control and skeletal myopathy in systolic heart failure.Aldehydic load and aldehyde dehydrogenase 2 profile during the progression of post-myocardial infarction cardiomyopathy: benefits of Alda-1Abnormalities of calcium handling proteins in skeletal muscle mirror those of the heart in humans with heart failure: a shared mechanism?Cardiac Cachexia: Perspectives for Prevention and Treatment.Aerobic exercise training in heart failure: impact on sympathetic hyperactivity and cardiac and skeletal muscle function.Intrinsic skeletal muscle alterations in chronic heart failure patients: a disease-specific myopathy or a result of deconditioning?Skeletal myopathy in heart failure: effects of aerobic exercise training.Suppressed sympathetic outflow to skeletal muscle, muscle thermogenesis, and activity energy expenditure with calorie restriction.Exercise training decreases NADPH oxidase activity and restores skeletal muscle mass in heart failure rats.Exercise training prevents ventricular tachycardia in CPVT1 due to reduced CaMKII-dependent arrhythmogenic Ca2+ release.Autophagy signaling in skeletal muscle of infarcted rats.Levels of Hepatic Activating Transcription Factor 6 and Caspase-3 Are Downregulated in Mice after Excessive Training.Exercise Training Restores Cardiac MicroRNA-1 and MicroRNA-29c to Nonpathological Levels in Obese Rats.Exercise training improves neurovascular control and calcium cycling gene expression in patients with heart failure with cardiac resynchronization therapy.The influence of aetiology on the benefits of exercise training in patients with heart failure.Examining the relationship between exercise tolerance and isoproterenol-based cardiac reserve in murine models of heart failure.High- versus moderate-intensity aerobic exercise training effects on skeletal muscle of infarcted rats.Case 2/2018 - 73-Year-Old Male with Ischemic Cardiomyopathy, Cachexia and Shock.Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy
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P2860
Aerobic exercise training improves skeletal muscle function and Ca2+ handling-related protein expression in sympathetic hyperactivity-induced heart failure.
description
2010 nî lūn-bûn
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年學術文章
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name
Aerobic exercise training impr ...... ctivity-induced heart failure.
@en
Aerobic exercise training impr ...... ctivity-induced heart failure.
@nl
type
label
Aerobic exercise training impr ...... ctivity-induced heart failure.
@en
Aerobic exercise training impr ...... ctivity-induced heart failure.
@nl
prefLabel
Aerobic exercise training impr ...... ctivity-induced heart failure.
@en
Aerobic exercise training impr ...... ctivity-induced heart failure.
@nl
P2093
P2860
P1476
Aerobic exercise training impr ...... activity-induced heart failure
@en
P2093
A V N Bacurau
M G Pereira
P2860
P304
P356
10.1152/JAPPLPHYSIOL.00281.2010
P407
P577
2010-07-01T00:00:00Z