Abnormal cardiac function associated with sympathetic nervous system hyperactivity in mice.
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Exercise training prevents oxidative stress and ubiquitin-proteasome system overactivity and reverse skeletal muscle atrophy in heart failureCardiac Dysregulation and Myocardial Injury in a 6-Hydroxydopamine-Induced Rat Model of Sympathetic DenervationDifferential targeting and function of alpha2A and alpha2C adrenergic receptor subtypes in cultured sympathetic neuronsReduction of sympathetic activity via adrenal-targeted GRK2 gene deletion attenuates heart failure progression and improves cardiac function after myocardial infarctionTachycardia, reduced vagal capacity, and age-dependent ventricular dysfunction arising from diminished expression of the presynaptic choline transporter.Usefulness of running wheel for detection of congestive heart failure in dilated cardiomyopathy mouse model.Sympathetic hyperactivity differentially affects skeletal muscle mass in developing heart failure: role of exercise training.Myocardial oxidative stress contributes to transgenic β₂-adrenoceptor activation-induced cardiomyopathy and heart failure.Integrative effect of carvedilol and aerobic exercise training therapies on improving cardiac contractility and remodeling in heart failure mice.Impact of leucine supplementation on exercise training induced anti-cardiac remodeling effect in heart failure mice.Collateral damage: cardiovascular consequences of chronic sympathetic activation with human aging.S-Nitrosoglutathione Reductase Deficiency Enhances the Proliferative Expansion of Adult Heart Progenitors and Myocytes Post Myocardial InfarctionKnockout of beta(1)- and beta(2)-adrenoceptors attenuates pressure overload-induced cardiac hypertrophy and fibrosis.Neurohumoral activation in heart failure: the role of adrenergic receptors.Whole-exome sequencing identifies ADRA2A mutation in atypical familial partial lipodystrophyAdrenergic nervous system in heart failure: pathophysiology and therapy.Aerobic exercise training in heart failure: impact on sympathetic hyperactivity and cardiac and skeletal muscle function.Physiology and pharmacology of the cardiovascular adrenergic system.Skeletal myopathy in heart failure: effects of aerobic exercise training.Adrenal adrenoceptors in heart failure.Cirrhotic cardiomyopathy: review of pathophysiology and treatment.Aerobic exercise training improves skeletal muscle function and Ca2+ handling-related protein expression in sympathetic hyperactivity-induced heart failure.Exercise training inhibits inflammatory cytokines and more than prevents myocardial dysfunction in rats with sustained beta-adrenergic hyperactivity.Exercise training as a treatment for heart failure: potential mechanisms and clinical implications.Exercise training reduces cardiac angiotensin II levels and prevents cardiac dysfunction in a genetic model of sympathetic hyperactivity-induced heart failure in mice.Lack of β2-AR improves exercise capacity and skeletal muscle oxidative phenotype in mice.Cardiac anti-remodelling effect of aerobic training is associated with a reduction in the calcineurin/NFAT signalling pathway in heart failure mice.Differential cardioprotective/cardiotoxic effects mediated by beta-adrenergic receptor subtypes.Role of beta1- and alpha2c-adrenergic receptor polymorphisms and their combination in heart failure: a case-control study.The role of local and systemic renin angiotensin system activation in a genetic model of sympathetic hyperactivity-induced heart failure in mice.Exercise training delays cardiac dysfunction and prevents calcium handling abnormalities in sympathetic hyperactivity-induced heart failure mice.Echocardiographic assessment of β-adrenoceptor stimulation-induced heart failure with reduced heart rate in mice.Correlation between the high-frequency content of the QRS on murine surface electrocardiogram and the sympathetic nerves density in left ventricle after myocardial infarction: Experimental study.Double disruption of α2A- and α2C-adrenoceptors results in sympathetic hyperactivity and high-bone-mass phenotype.Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling.Thyroid hormone interacts with the sympathetic nervous system to modulate bone mass and structure in young adult mice.The α2C-Del322–325 adrenoceptor polymorphism and the occurrence of left ventricular hypertrophy in hypertensives
P2860
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P2860
Abnormal cardiac function associated with sympathetic nervous system hyperactivity in mice.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Abnormal cardiac function asso ...... system hyperactivity in mice.
@en
Abnormal cardiac function asso ...... system hyperactivity in mice.
@nl
type
label
Abnormal cardiac function asso ...... system hyperactivity in mice.
@en
Abnormal cardiac function asso ...... system hyperactivity in mice.
@nl
prefLabel
Abnormal cardiac function asso ...... system hyperactivity in mice.
@en
Abnormal cardiac function asso ...... system hyperactivity in mice.
@nl
P2093
P2860
P1476
Abnormal cardiac function asso ...... system hyperactivity in mice.
@en
P2093
Andrew Patterson
Daniel Bernstein
Patricia C Brum
P2860
P304
P356
10.1152/AJPHEART.01063.2001
P577
2002-11-01T00:00:00Z