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From disease to treatment: from rare skeletal disorders to treatments for osteoporosisFriend or foe: high bone mineral density on routine bone density scanning, a review of causes and managementCirculating sclerostin levels are decreased in patients with endogenous hypercortisolism and increase after treatmentManagement of glucocorticoid-induced osteoporosis.Role and mechanism of action of sclerostin in bone.TGF-β Family Signaling in Connective Tissue and Skeletal Diseases.Decompressive surgery in a patient with hyperostosis corticalis generalisata for relief of cognitive disability and dysaesthesia.Van Buchem disease: clinical, biochemical, and densitometric features of patients and disease carriers.Van Buchem disease: First case report in Taiwan.Protection From Glucocorticoid-Induced Osteoporosis by Anti-Catabolic Signaling in the Absence of Sost/Sclerostin.Sclerostin Blockade-A Dual Mode of Action After All?
P2860
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P2860
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh-hant
name
Glucocorticoids are not always deleterious for bone.
@en
Glucocorticoids are not always deleterious for bone.
@nl
type
label
Glucocorticoids are not always deleterious for bone.
@en
Glucocorticoids are not always deleterious for bone.
@nl
prefLabel
Glucocorticoids are not always deleterious for bone.
@en
Glucocorticoids are not always deleterious for bone.
@nl
P2093
P2860
P356
P1476
Glucocorticoids are not always deleterious for bone.
@en
P2093
Antoon H J M van Lierop
Neveen A T Hamdy
Socrates E Papapoulos
P2860
P304
P356
10.1002/JBMR.151
P577
2010-12-01T00:00:00Z