All the 1p19q codeleted gliomas are mutated on IDH1 or IDH2. - Wikidata - awgldk - TriplyDB

All the 1p19q codeleted gliomas are mutated on IDH1 or IDH2.

All the 1p19q codeleted gliomas are mutated on IDH1 or IDH2.

http://www.wikidata.org/entity/Q43081541

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IDH1 and IDH2 mutations in gliomas Genetics and Epigenetics of Glioblastoma: Applications and Overall Incidence of IDH1 Mutation In vivo models of brain tumors: roles of genetically engineered mouse models in understanding tumor biology and use in preclinical studies Modern brain tumor imaging Metabolic consequences of oncogenic IDH mutations The epidemiology of glioma in adults: a "state of the science" review The global DNA methylation surrogate LINE-1 methylation is correlated with MGMT promoter methylation and is a better prognostic factor for glioma Recursive partitioning analysis of prognostic variables in newly diagnosed anaplastic oligodendroglial tumors International retrospective study of over 1000 adults with anaplastic oligodendroglial tumors Assessing CpG island methylator phenotype, 1p/19q codeletion, and MGMT promoter methylation from epigenome-wide data in the biomarker cohort of the NOA-04 trial.IDH mutations: genotype-phenotype correlation and prognostic impact.Low rate of R132H IDH1 mutation in infratentorial and spinal cord grade II and III diffuse gliomas.1p/19q testing has no significance in the workup of glioblastomas.Imaging growth and isocitrate dehydrogenase 1 mutation are independent predictors for diffuse low-grade gliomas.Mitotic index, microvascular proliferation, and necrosis define 3 groups of 1p/19q codeleted anaplastic oligodendrogliomas associated with different genomic alterations.SNP array analysis reveals novel genomic abnormalities including copy neutral loss of heterozygosity in anaplastic oligodendrogliomas.Better prognosis of patients with glioma expressing FGF2-dependent PDGFRA irrespective of morphological diagnosis.Glial progenitors as targets for transformation in glioma.Novel CIC point mutations and an exon-spanning, homozygous deletion identified in oligodendroglial tumors by a comprehensive genomic approach including transcriptome sequencing.Acetate supplementation induces growth arrest of NG2/PDGFRα-positive oligodendroglioma-derived tumor-initiating cells IDH mutation status and role of WHO grade and mitotic index in overall survival in grade II-III diffuse gliomas.The importance of 10q status in an outcomes-based comparison between 1p/19q fluorescence in situ hybridization and polymerase chain reaction-based microsatellite loss of heterozygosity analysis of oligodendrogliomas.How molecular testing can help (and hurt) in the workup of gliomas.TCF12 is mutated in anaplastic oligodendroglioma Indications for Treatment: Is Observation or Chemotherapy Alone a Reasonable Approach in the Management of Low-Grade Gliomas?Intramedullary pilomyxoid astrocytoma with intracerebral metastasis exhibiting oligoden-droglioma-like features Isocitrate dehydrogenase mutations in gliomas Adult brainstem gliomas: Correlation of clinical and molecular features.What do we know about IDH1/2 mutations so far, and how do we use it?Association between glioma susceptibility loci and tumour pathology defines specific molecular etiologies Inherited variant on chromosome 11q23 increases susceptibility to IDH-mutated but not IDH-normal gliomas regardless of grade or histology.Genetic risk variants in the CDKN2A/B, RTEL1 and EGFR genes are associated with somatic biomarkers in glioma Isocitrate dehydrogenase 1 analysis differentiates gangliogliomas from infiltrative gliomas N-acetylaspartate (NAA) and N-acetylaspartylglutamate (NAAG) promote growth and inhibit differentiation of glioma stem-like cells Mitochondrial protein ATPase family, AAA domain containing 3A correlates with radioresistance in glioblastoma.Deferred radiotherapy and upfront procarbazine-ACNU-vincristine administration for 1p19q codeleted oligodendroglial tumors are associated with favorable outcome without compromising patient performance, regardless of WHO grade.Clinical value of chromosome arms 19q and 11p losses in low-grade gliomas.Benefit from procarbazine, lomustine, and vincristine in oligodendroglial tumors is associated with mutation of IDH.A glioma classification scheme based on coexpression modules of EGFR and PDGFRA.Not all 1p/19q non-codeleted oligodendroglial tumors are astrocytic.

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All the 1p19q codeleted gliomas are mutated on IDH1 or IDH2.

http://www.wikidata.org/entity/Q43081541

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2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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All the 1p19q codeleted gliomas are mutated on IDH1 or IDH2.

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All the 1p19q codeleted gliomas are mutated on IDH1 or IDH2.

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type

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All the 1p19q codeleted gliomas are mutated on IDH1 or IDH2.

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All the 1p19q codeleted gliomas are mutated on IDH1 or IDH2.

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prefLabel

All the 1p19q codeleted gliomas are mutated on IDH1 or IDH2.

@en

All the 1p19q codeleted gliomas are mutated on IDH1 or IDH2.

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