The PI3K/Akt signaling pathway mediates the high glucose-induced expression of extracellular matrix molecules in human retinal pigment epithelial cells.
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The CLC-2 Chloride Channel Modulates ECM Synthesis, Differentiation, and Migration of Human Conjunctival Fibroblasts via the PI3K/Akt Signaling Pathway.NPPB modulates apoptosis, proliferation, migration and extracellular matrix synthesis of conjunctival fibroblasts by inhibiting PI3K/AKT signaling.Identification of the functional pathways altered by placental cell exposure to high glucose: lessons from the transcript and metabolite interactome.Effects of High Glucose on the Expression of LAMA1 and Biological Behavior of Choroid Retinal Endothelial Cells.LRP1 is required for novobiocin-mediated fibronectin turnover
P2860
The PI3K/Akt signaling pathway mediates the high glucose-induced expression of extracellular matrix molecules in human retinal pigment epithelial cells.
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name
The PI3K/Akt signaling pathway ...... inal pigment epithelial cells.
@en
The PI3K/Akt signaling pathway ...... inal pigment epithelial cells.
@nl
type
label
The PI3K/Akt signaling pathway ...... inal pigment epithelial cells.
@en
The PI3K/Akt signaling pathway ...... inal pigment epithelial cells.
@nl
prefLabel
The PI3K/Akt signaling pathway ...... inal pigment epithelial cells.
@en
The PI3K/Akt signaling pathway ...... inal pigment epithelial cells.
@nl
P2093
P2860
P356
P1476
The PI3K/Akt signaling pathway ...... inal pigment epithelial cells.
@en
P2093
P2860
P304
P356
10.1155/2015/920280
P577
2015-01-28T00:00:00Z