Progressive glomerulosclerosis in type 2 diabetes is associated with renal histone H3K9 and H3K23 acetylation, H3K4 dimethylation and phosphorylation at serine 10. - Wikidata - awgldk - TriplyDB

Progressive glomerulosclerosis in type 2 diabetes is associated with renal histone H3K9 and H3K23 acetylation, H3K4 dimethylation and phosphorylation at serine 10.

Progressive glomerulosclerosis in type 2 diabetes is associated with renal histone H3K9 and H3K23 acetylation, H3K4 dimethylation and phosphorylation at serine 10.

http://www.wikidata.org/entity/Q43196259

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The role of epigenetics in the pathology of diabetic complications Histone Acetylation and Its Modifiers in the Pathogenesis of Diabetic Nephropathy Human genetics of diabetic vascular complications Epigenetics of Renal Development and Disease Environmental chemical exposures and human epigenetics Acetylation of retinal histones in diabetes increases inflammatory proteins: effects of minocycline and manipulation of histone acetyltransferase (HAT) and histone deacetylase (HDAC)Genetic and epigenetic events in diabetic wound healing.Microplate-based platform for combined chromatin and DNA methylation immunoprecipitation assays Fetal environment, epigenetics, and pediatric renal disease.Epigenetics: deciphering its role in diabetes and its chronic complications.Valproic acid attenuates proteinuria and kidney injury.Histone acetyltransferase PCAF regulates inflammatory molecules in the development of renal injury Epigenetic modifications and diabetic nephropathy.Histone deacetylase inhibitors attenuate P-aIgA1-induced cell proliferation and extracellular matrix synthesis in human renal mesangial cells in vitro.Involvement of p300/CBP and epigenetic histone acetylation in TGF-β1-mediated gene transcription in mesangial cells High glucose-induced cytoplasmic translocation of Dnmt3a contributes to CTGF hypo-methylation in mesangial cells.New insights into molecular mechanisms of diabetic kidney disease.Glycemic memories and the epigenetic component of diabetic nephropathy.Danger control programs cause tissue injury and remodeling.Human genetics of diabetic retinopathy.Histone lysine methylation in diabetic nephropathy.Novel effective drugs for diabetic kidney disease? or not?Epigenetics and Cardiovascular Disease in Diabetes.Links between coagulation, inflammation, regeneration, and fibrosis in kidney pathology.Chronic kidney disease in children and the role of epigenetics: Future therapeutic trajectories Epigenetics of kidney disease.Involvement of Histone Lysine Methylation in p21 Gene Expression in Rat Kidney In Vivo and Rat Mesangial Cells In Vitro under Diabetic Conditions.Four danger response programs determine glomerular and tubulointerstitial kidney pathology: clotting, inflammation, epithelial and mesenchymal healing.Epigenetic bystander-like effects of stroke in somatic organs Chromatin-modifying agents reactivate embryonic renal stem/progenitor genes in human adult kidney epithelial cells but abrogate dedifferentiation and stemness.Dual blockade of the homeostatic chemokine CXCL12 and the proinflammatory chemokine CCL2 has additive protective effects on diabetic kidney disease.The diabetes pandemic suggests unmet needs for 'CKD with diabetes' in addition to 'diabetic nephropathy'-implications for pre-clinical research and drug testing.Epigenetic changes and alteration of Fbn1 and Col3A1 gene expression under hyperglycaemic and hyperinsulinaemic conditions.Apelin inhibits the development of diabetic nephropathy by regulating histone acetylation in Akita mouse.Epigenetic changes in renal genes dysregulated in mouse and rat models of type 1 diabetes.Podocyte loss involves MDM2-driven mitotic catastrophe.IL-22 sustains epithelial integrity in progressive kidney remodeling and fibrosis VEGF genetic polymorphisms may contribute to the risk of diabetic nephropathy in patients with diabetes mellitus: a meta-analysis

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P2860

Progressive glomerulosclerosis in type 2 diabetes is associated with renal histone H3K9 and H3K23 acetylation, H3K4 dimethylation and phosphorylation at serine 10.

http://www.wikidata.org/entity/Q43196259

description

2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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2010年學術文章

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name

Progressive glomerulosclerosis ...... phosphorylation at serine 10.

@en

Progressive glomerulosclerosis ...... phosphorylation at serine 10.

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type

label

Progressive glomerulosclerosis ...... phosphorylation at serine 10.

@en

Progressive glomerulosclerosis ...... phosphorylation at serine 10.

@nl

prefLabel

Progressive glomerulosclerosis ...... phosphorylation at serine 10.

@en

Progressive glomerulosclerosis ...... phosphorylation at serine 10.

@nl

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P1433

Nephrology Dialysis Transplantation

P1476

Progressive glomerulosclerosis ...... phosphorylation at serine 10.

@en

P2093

Anil Bhanudas Gaikwad

http://www.w3.org/2001/XMLSchema#string

Dirk Eulberg

http://www.w3.org/2001/XMLSchema#string

Hans-Joachim Anders

http://www.w3.org/2001/XMLSchema#string

Julia Lichtnekert

http://www.w3.org/2001/XMLSchema#string

Kulbhushan Tikoo

http://www.w3.org/2001/XMLSchema#string

Onkar Kulkarni

http://www.w3.org/2001/XMLSchema#string

Sufyan G Sayyed

http://www.w3.org/2001/XMLSchema#string

Sven Klussmann

http://www.w3.org/2001/XMLSchema#string

P2860

Chromatin modifications and their function

The complex language of chromatin regulation during transcription

Advanced glycation end products cause epithelial-myofibroblast transdifferentiation via the receptor for advanced glycation end products (RAGE)

Global and societal implications of the diabetes epidemic

Transcriptional control at regulatory checkpoints by histone deacetylases: molecular connections between cancer and chromatin.

In vivo chromatin remodeling events leading to inflammatory gene transcription under diabetic conditions.

From fibrosis to sclerosis: mechanisms of glomerulosclerosis in diabetic nephropathy.

Chemokines and chemokine receptors are involved in the resolution or progression of renal disease.

Phosphorylation of serine 10 in histone H3, what for?

Change in post-translational modifications of histone H3, heat-shock protein-27 and MAP kinase p38 expression by curcumin in streptozotocin-induced type I diabetic nephropathy.

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1811-1817

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10.1093/NDT/GFP730

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6

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25

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2010-01-12T00:00:00Z

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20067909

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phosphorylation

type-2 diabetes