about
Histocompatibility antigens, aspirin use and cognitive performance in non-demented elderly subjectsGenetic and clinical features of progranulin-associated frontotemporal lobar degenerationPreclinical trials in autosomal dominant AD: implementation of the DIAN-TU trial.Pedigree with frontotemporal lobar degeneration--motor neuron disease and Tar DNA binding protein-43 positive neuropathology: genetic linkage to chromosome 9.Frontotemporal dementia and its subtypes: a genome-wide association study.Association between presenilin-1 Glu318Gly mutation and familial Alzheimer's disease in the Australian population.C9ORF72 repeat expansion in Australian and Spanish frontotemporal dementia patientsFactors associated with the onset and persistence of post-lumbar puncture headache.Neuropathologic assessment of participants in two multi-center longitudinal observational studies: the Alzheimer Disease Neuroimaging Initiative (ADNI) and the Dominantly Inherited Alzheimer Network (DIAN).Frontotemporal dementia-amyotrophic lateral sclerosis syndrome locus on chromosome 16p12.1-q12.2: genetic, clinical and neuropathological analysis.CCNF mutations in amyotrophic lateral sclerosis and frontotemporal dementia.Association of alleles carried at TNFA -850 and BAT1 -22 with Alzheimer's disease.Diffuse Lewy body disease: clinical features in nine cases without coexistent Alzheimer's disease.Variable phenotype of Alzheimer's disease with spastic paraparesis.TMEM106B is a genetic modifier of frontotemporal lobar degeneration with C9orf72 hexanucleotide repeat expansions.Sigma nonopioid intracellular receptor 1 mutations cause frontotemporal lobar degeneration-motor neuron disease.Cell loss in the nucleus basalis is related to regional cortical atrophy in Alzheimer's disease.Progressive supranuclear palsy pathology caused by a novel silent mutation in exon 10 of the tau gene: expansion of the disease phenotype caused by tau gene mutations.High striatal amyloid beta-peptide deposition across different autosomal Alzheimer disease mutation types.Intraneuronal advanced glycation endproducts in presenilin-1 Alzheimer's disease.Presenilin-1 mutation L271V results in altered exon 8 splicing and Alzheimer's disease with non-cored plaques and no neuritic dystrophy.Two novel presenilin-1 mutations (Y256S and Q222H) are associated with early-onset Alzheimer's disease.Pick bodies in a family with presenilin-1 Alzheimer's disease.Very early-onset frontotemporal dementia with no family history predicts underlying fused in sarcoma pathology.Is CHCHD10 Pro34Ser pathogenic for frontotemporal dementia and amyotrophic lateral sclerosis?Plasma amyloid precursor protein is decreased in Alzheimer's disease.Mutations in the tau gene that cause an increase in three repeat tau and frontotemporal dementia.Alzheimer's disease with spastic paraparesis and 'cotton wool' plaques: two pedigrees with PS-1 exon 9 deletions.Mutations in progranulin explain atypical phenotypes with variants in MAPT.Brain stem serotonin-synthesizing neurons in Alzheimer's disease: a clinicopathological correlation.Neuropathology in the S305S tau gene mutation.Further evidence for an association between a mutation in the APP gene and Lewy body formation.No association of presenilin-1 intronic polymorphism and Alzheimer's disease in Australia.Topography of brain atrophy during normal aging and Alzheimer's disease.A model of executive functions in very old community dwellers: evidence from The Sydney Older Persons Study.Phenotypic variability associated with progranulin haploinsufficiency in patients with the common 1477C-->T (Arg493X) mutation: an international initiative.A mutation in codon 717 of the amyloid precursor protein gene in an Australian family with Alzheimer's disease.Two novel (M233T and R278T) presenilin-1 mutations in early-onset Alzheimer's disease pedigrees and preliminary evidence for association of presenilin-1 mutations with a novel phenotype.Two novel presenilin-1 mutations (Ser169Leu and Pro436Gln) associated with very early onset Alzheimer's disease.Neuropathology of three clinical cases prospectively diagnosed as dementia with Lewy bodies.
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description
hulumtues
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onderzoeker
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researcher
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հետազոտող
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name
William S Brooks
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William S Brooks
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William S Brooks
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William S Brooks
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William S Brooks
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type
label
William S Brooks
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William S Brooks
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William S Brooks
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William S Brooks
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William S Brooks
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William S Brooks
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William S Brooks
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William S Brooks
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William S Brooks
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William S Brooks
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P106
P21
P31
P4012
P496
0000-0001-8603-4233