Recruitment of antigen-specific CD8+ T cells in response to infection is markedly efficient.
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The Timing of T Cell Priming and CyclingThe role of naive T cell precursor frequency and recruitment in dictating immune response magnitudeT cell responses to antigen: hasty proposals resolved through long engagementsEcological analysis of antigen-specific CTL repertoires defines the relationship between naive and immune T-cell populationsImmune evasion proteins of murine cytomegalovirus preferentially affect cell surface display of recently generated peptide presentation complexes.Primary CTL response magnitude in mice is determined by the extent of naive T cell recruitment and subsequent clonal expansionOne naive T cell, multiple fates in CD8+ T cell differentiation.Random migration and signal integration promote rapid and robust T cell recruitmentDirect presentation regulates the magnitude of the CD8+ T cell response to cell-associated antigen through prolonged T cell proliferation.Rapid expansion of CD8+ T cells in wild-type and type I interferon receptor-deficient mice correlates with protection after low-dose emergency immunization with modified vaccinia virus Ankara.Lack of original antigenic sin in recall CD8(+) T cell responses.Polyfunctional type-1, -2, and -17 CD8⁺ T cell responses to apoptotic self-antigens correlate with the chronic evolution of hepatitis C virus infection.Convergent recombination shapes the clonotypic landscape of the naive T-cell repertoire.Modulating numbers and phenotype of CD8+ T cells in secondary immune responses.T cell-tumor interaction directs the development of immunotherapies in head and neck cancer.The TCR's sensitivity to self peptide-MHC dictates the ability of naive CD8(+) T cells to respond to foreign antigens.Plasmodium suppresses expansion of T cell responses to heterologous infections.CD4+ T cells support cytotoxic T lymphocyte priming by controlling lymph node input.Persistence of EBV antigen-specific CD8 T cell clonotypes during homeostatic immune reconstitution in cancer patients.Migration of cytotoxic lymphocytes in cell cycle permits local MHC I-dependent control of division at sites of viral infectionParasite fate and involvement of infected cells in the induction of CD4+ and CD8+ T cell responses to Toxoplasma gondii.Tracking single hematopoietic stem cells in vivo using high-throughput sequencing in conjunction with viral genetic barcodingVirotherapy induces massive infiltration of neutrophils in a subset of tumors defined by a strong endogenous interferon response activity.The mechanism of HLA-DM induced peptide exchange in the MHC class II antigen presentation pathwayImmunization route dictates cross-priming efficiency and impacts the optimal timing of adjuvant deliveryReproducibility of Illumina platform deep sequencing errors allows accurate determination of DNA barcodes in cells.Rapid Recovery of CD3+CD8+ T Cells on Day 90 Predicts Superior Survival after Unmanipulated Haploidentical Blood and Marrow Transplantation.From optical bench to cageside: intravital microscopy on the long road to rational vaccine design.The race for the prize: T-cell trafficking strategies for optimal surveillanceEffective Respiratory CD8 T-Cell Immunity to Influenza Virus Induced by Intranasal Carbomer-Lecithin-Adjuvanted Non-replicating Vaccines.Alpha-alumina nanoparticles induce efficient autophagy-dependent cross-presentation and potent antitumour responseT cell vaccinology: exploring the known unknowns.Persistent antigen at vaccination sites induces tumor-specific CD8⁺ T cell sequestration, dysfunction and deletion.Quantifying T lymphocyte turnover.Diversification of the antigen-specific T cell receptor repertoire after varicella zoster vaccination.High viral burden restricts short-lived effector cell number at late times postinfection through increased natural regulatory T cell expansion.Interrogating the relationship between naïve and immune antiviral T cell repertoiresCD38 controls the innate immune response against Listeria monocytogenes.pMHC affinity controls duration of CD8+ T cell-DC interactions and imprints timing of effector differentiation versus expansion.T cells translate individual, quantal activation into collective, analog cytokine responses via time-integrated feedbacks.
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Recruitment of antigen-specific CD8+ T cells in response to infection is markedly efficient.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Recruitment of antigen-specifi ...... fection is markedly efficient.
@en
Recruitment of antigen-specifi ...... fection is markedly efficient.
@nl
type
label
Recruitment of antigen-specifi ...... fection is markedly efficient.
@en
Recruitment of antigen-specifi ...... fection is markedly efficient.
@nl
prefLabel
Recruitment of antigen-specifi ...... fection is markedly efficient.
@en
Recruitment of antigen-specifi ...... fection is markedly efficient.
@nl
P2093
P2860
P50
P356
P1433
P1476
Recruitment of antigen-specifi ...... fection is markedly efficient.
@en
P2093
Cláudio Nunes-Alves
Erwin Swart
Jeroen W J van Heijst
Ramon Arens
Ron M Kerkhoven
Ton N M Schumacher
P2860
P304
P356
10.1126/SCIENCE.1175455
P407
P577
2009-09-01T00:00:00Z