about
Frataxin interacts functionally with mitochondrial electron transport chain proteinsPhospholipase C-epsilon regulates epidermal morphogenesis in Caenorhabditis elegansThe Wnt receptor Ryk reduces neuronal and cell survival capacity by repressing FOXO activity during the early phases of mutant huntingtin pathogenicityInositol 1,4,5-trisphosphate signalling regulates the avoidance response to nose touch in Caenorhabditis elegansIntegration of β-catenin, sirtuin, and FOXO signaling protects from mutant huntingtin toxicityTRPM channels are required for rhythmicity in the ultradian defecation rhythm of C. elegansImproved gene targeting in C. elegans using counter-selection and Flp-mediated marker excision.Metformin intake associates with better cognitive function in patients with Huntington's disease.Large-scale functional RNAi screen in C. elegans identifies genes that regulate the dysfunction of mutant polyglutamine neurons.Targeted next generation sequencing for molecular diagnosis of Usher syndrome.Absence of effects of Sir2 overexpression on lifespan in C. elegans and DrosophilaDisruption of the ATP-binding cassette B7 (ABTM-1/ABCB7) induces oxidative stress and premature cell death in Caenorhabditis elegansIP3 signalling regulates exogenous RNAi in Caenorhabditis elegans.Delaying aging and the aging-associated decline in protein homeostasis by inhibition of tryptophan degradation.AMPK activation protects from neuronal dysfunction and vulnerability across nematode, cellular and mouse models of Huntington's disease.Genetic analysis of IP3 and calcium signalling pathways in C. elegans.USH2A Gene Editing Using the CRISPR SystemThe frataxin-encoding operon of Caenorhabditis elegans shows complex structure and regulation.Reduction of Caenorhabditis elegans frataxin increases sensitivity to oxidative stress, reduces lifespan, and causes lethality in a mitochondrial complex II mutant.Differential expression pattern of coq-8 gene during development in Caenorhabditis elegans.Reverse genetic strategies in Caenorhabditis elegans: towards controlled manipulation of the genome.Metformin treatment reduces motor and neuropsychiatric phenotypes in the zQ175 mouse model of Huntington disease.
P50
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P50
description
hulumtues
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հետազոտող
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Rafael P Vázquez-Manrique
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Rafael P Vázquez-Manrique
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Rafael P Vázquez-Manrique
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Rafael P Vázquez-Manrique
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Rafael P Vázquez-Manrique
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Rafael P Vázquez-Manrique
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Rafael P Vázquez-Manrique
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Rafael P Vázquez-Manrique
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Rafael P Vázquez-Manrique
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Rafael P Vázquez-Manrique
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Rafael P Vázquez-Manrique
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P106
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P496
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