Nonrandom loss of chromosome 15 in Syrian hamster tumours induced by v-Ha-ras plus v-myc oncogenes.
about
Partial reversion of the transformed phenotype in HRAS-transfected tumorigenic cells by transfer of a human gene.Glucocorticoids facilitate the stable transformation of embryonal rat fibroblasts by a polyomavirus large tumor antigen-deficient mutant.Regulation of microfilament organization and anchorage-independent growth by tropomyosin 1.Detection of genes with a potential for suppressing the transformed phenotype associated with activated ras genesRole of a tumor-suppressor gene in the negative control of anchorage-independent growth of Syrian hamster cells.Transfection with plasmid pSV2gptEJ induces chromosome rearrangements in CHEF cellsCellular and molecular mechanisms of multistep carcinogenesis: relevance to carcinogen risk assessmentTransforming gene from human stomach cancers and a noncancerous portion of stomach mucosaInhibition of human and simian immunodeficiency virus protease function by targeting Vpx-protease-mutant fusion protein into viral particlesIn vitro mRNA degradation system to study the virion host shutoff function of herpes simplex virus.Regulation of c-myc mRNA stability in vitro by a labile destabilizer with an essential nucleic acid component.Resistance to oncogenic transformation in revertant R1 of human ras-transformed NIH 3T3 cellsViral protein R of human immunodeficiency virus types 1 and 2 is dispensable for replication and cytopathogenicity in lymphoid cells.Cooperative transforming activities of ras, myc, and src viral oncogenes in nonestablished rat adrenocortical cells.Synergism of v-myc and v-Ha-ras in the in vitro neoplastic progression of murine lymphoid cellsCoinfection with viruses carrying the v-Ha-ras and v-myc oncogenes leads to growth factor independence by an indirect mechanism.Cellular and molecular mechanisms of chemically induced renal carcinogenesis.Loss of tumor-suppressive function during chemically induced neoplastic progression of Syrian hamster embryo cells.Overlapping loss of heterozygosity by mitotic recombination on mouse chromosome 7F1-ter in skin carcinogenesis.Multiple changes in oncogenes and tumor suppressor genes in human retinoblastoma.A specific chromosome change and distinctive transforming genes are necessary for malignant progression of spontaneous transformation in cultured Chinese hamster embryo cells.Assignment of the mu-class glutathione S-transferase gene (hGSTYBX) to Syrian hamster chromosome 15 by FISH.The Ha-ras-induced transformed phenotype of rat-1 cells can be suppressed in hybrids with rat embryonic fibroblasts.
P2860
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P2860
Nonrandom loss of chromosome 15 in Syrian hamster tumours induced by v-Ha-ras plus v-myc oncogenes.
description
1985 nî lūn-bûn
@nan
1985年の論文
@ja
1985年学术文章
@wuu
1985年学术文章
@zh
1985年学术文章
@zh-cn
1985年学术文章
@zh-hans
1985年学术文章
@zh-my
1985年学术文章
@zh-sg
1985年學術文章
@yue
1985年學術文章
@zh-hant
name
Nonrandom loss of chromosome 1 ...... v-Ha-ras plus v-myc oncogenes.
@en
Nonrandom loss of chromosome 1 ...... v-Ha-ras plus v-myc oncogenes.
@nl
type
label
Nonrandom loss of chromosome 1 ...... v-Ha-ras plus v-myc oncogenes.
@en
Nonrandom loss of chromosome 1 ...... v-Ha-ras plus v-myc oncogenes.
@nl
prefLabel
Nonrandom loss of chromosome 1 ...... v-Ha-ras plus v-myc oncogenes.
@en
Nonrandom loss of chromosome 1 ...... v-Ha-ras plus v-myc oncogenes.
@nl
P2093
P356
P1433
P1476
Nonrandom loss of chromosome 1 ...... v-Ha-ras plus v-myc oncogenes.
@en
P2093
P2888
P304
P356
10.1038/316636A0
P407
P577
1985-08-01T00:00:00Z
P6179
1024682421