Ca2+ dysregulation induces mitochondrial depolarization and apoptosis: role of Na+/Ca2+ exchanger and AKT. - Wikidata - awgldk - TriplyDB

Ca2+ dysregulation induces mitochondrial depolarization and apoptosis: role of Na+/Ca2+ exchanger and AKT.

Ca2+ dysregulation induces mitochondrial depolarization and apoptosis: role of Na+/Ca2+ exchanger and AKT.

http://www.wikidata.org/entity/Q43415387

about

Group B streptococcal beta-hemolysin/cytolysin directly impairs cardiomyocyte viability and function Human cardiac tissue in a microperfusion chamber simulating extracorporeal circulation--ischemia and apoptosis studies.Excitation-contraction coupling in ventricular myocytes is enhanced by paracrine signaling from mesenchymal stem cells.Two close, too close: sarcoplasmic reticulum-mitochondrial crosstalk and cardiomyocyte fate Control of mitochondrial activity by miRNAs.A novel initiation mechanism of death in Streptococcus pneumoniae induced by the human milk protein-lipid complex HAMLET and activated during physiological death.Transient receptor potential melastatin 4 and cell death.Endoplasmic reticulum-mitochondria crosstalk in NIX-mediated murine cell death Nuclear and mitochondrial signalling Akts in cardiomyocytes Apoptotic and non-apoptotic programmed cardiomyocyte death in ventricular remodelling.Akt mediated mitochondrial protection in the heart: metabolic and survival pathways to the rescue.The Mitochondrial Voltage-Dependent Anion Channel 1, Ca2+ Transport, Apoptosis, and Their Regulation.Gq protein-induced apoptosis is mediated by AKT kinase inhibition that leads to protein kinase C-induced c-Jun N-terminal kinase activation.Nematode Sodium Calcium Exchangers: A Surprising Lack of Transport.Nix-mediated apoptosis links myocardial fibrosis, cardiac remodeling, and hypertrophy decompensation.Highly pathogenic H5N1 avian influenza virus induces extracellular Ca2+ influx, leading to apoptosis in avian cells.The challenge to verify ceramide's role of apoptosis induction in human cardiomyocytes--a pilot study.RhoA/Rho kinase up-regulate Bax to activate a mitochondrial death pathway and induce cardiomyocyte apoptosis.Oxygen reperfusion is limited in the postischemic hypertrophic myocardium.Akt mediates mitochondrial protection in cardiomyocytes through phosphorylation of mitochondrial hexokinase-II.Simple kinetic model of mitochondrial swelling in cardiac cells.Pretreatment with PTD-calbindin D 28k alleviates rat brain injury induced by ischemia and reperfusion.Animal Toxins as Therapeutic Tools to Treat Neurodegenerative Diseases.Deletion of the aquaporin-4 gene alters expression and phosphorylation of protective kinases in the mouse heart.

P2860

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P2860

Ca2+ dysregulation induces mitochondrial depolarization and apoptosis: role of Na+/Ca2+ exchanger and AKT.

http://www.wikidata.org/entity/Q43415387

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

@zh-my

2005年学术文章

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2005年學術文章

@yue

2005年學術文章

@zh-hant

name

Ca2+ dysregulation induces mit ...... of Na+/Ca2+ exchanger and AKT.

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Ca2+ dysregulation induces mit ...... of Na+/Ca2+ exchanger and AKT.

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type

label

Ca2+ dysregulation induces mit ...... of Na+/Ca2+ exchanger and AKT.

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Ca2+ dysregulation induces mit ...... of Na+/Ca2+ exchanger and AKT.

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prefLabel

Ca2+ dysregulation induces mit ...... of Na+/Ca2+ exchanger and AKT.

@en

Ca2+ dysregulation induces mit ...... of Na+/Ca2+ exchanger and AKT.

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P1433

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P356

P1433

Journal of Biological Chemistry

P1476

Ca2+ dysregulation induces mit ...... of Na+/Ca2+ exchanger and AKT.

@en

P2093

Amy L Howes

http://www.w3.org/2001/XMLSchema#string

Gerald W Dorn

http://www.w3.org/2001/XMLSchema#string

Joan Heller Brown

http://www.w3.org/2001/XMLSchema#string

John W Adams

http://www.w3.org/2001/XMLSchema#string

Shigeki Miyamoto

http://www.w3.org/2001/XMLSchema#string

P2860

Transient cardiac expression of constitutively active Galphaq leads to hypertrophy and dilated cardiomyopathy by calcineurin-dependent and independent pathways

Loss of a gp130 cardiac muscle cell survival pathway is a critical event in the onset of heart failure during biomechanical stress

Cardiac excitation-contraction coupling

Propagation of the apoptotic signal by mitochondrial waves

Functional adult myocardium in the absence of Na+-Ca2+ exchange: cardiac-specific knockout of NCX1

Akt-mediated cardiomyocyte survival pathways are compromised by G alpha q-induced phosphoinositide 4,5-bisphosphate depletion

Calcium, ATP, and ROS: a mitochondrial love-hate triangle

BAX and BAK regulation of endoplasmic reticulum Ca2+: a control point for apoptosis

Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

The mitochondrial permeability transition, release of cytochrome c and cell death. Correlation with the duration of pore openings in situ.

P304

38505-38512

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scholarly article

P356

10.1074/JBC.M505223200

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46

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P478

280

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2005-08-01T00:00:00Z

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P5875

7686997

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P698

16061478

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