Role of NF-κβ and oxidative pathways in atherosclerosis: cross-talk between dyslipidemia and candesartan.

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Role of NF-κβ and oxidative pathways in atherosclerosis: cross-talk between dyslipidemia and candesartan.

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2013 nî lūn-bûn

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2013年の論文

@ja

2013年学术文章

@wuu

2013年学术文章

@zh

2013年学术文章

@zh-cn

2013年学术文章

@zh-hans

2013年学术文章

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2013年学术文章

@zh-sg

2013年學術文章

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2013年學術文章

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name

Role of NF-κβ and oxidative pa ...... dyslipidemia and candesartan.

@en

Role of NF-κβ and oxidative pa ...... dyslipidemia and candesartan.

@nl

type

Item

label

Role of NF-κβ and oxidative pa ...... dyslipidemia and candesartan.

@en

Role of NF-κβ and oxidative pa ...... dyslipidemia and candesartan.

@nl

prefLabel

Role of NF-κβ and oxidative pa ...... dyslipidemia and candesartan.

@en

Role of NF-κβ and oxidative pa ...... dyslipidemia and candesartan.

@nl

P1476

Role of NF-κβ and oxidative pa ...... dyslipidemia and candesartan.

@en

P2093

Bassim I Mohammad

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Fadhil G Al-Amran

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Maitham G Yousif

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Mohammed S Abdulzahra

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Murooge L Majeed

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Najah R Hadi

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Nasser Ghaly Yousif

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P2860

Risk factors in coronary atherosclerosis athero-inflammation: the meeting point

Early endothelial dysfunction in cholesterol-fed rabbits: a non-invasive in vivo ultrasound study

Novel Toll-like receptor-4 deficiency attenuates trastuzumab (Herceptin) induced cardiac injury in mice.

Effect of candesartan monotherapy on lipid metabolism in patients with hypertension: a retrospective longitudinal survey using data from electronic medical records.

Angiotensin receptor blockade with candesartan attenuates atherosclerosis, plaque disruption, and macrophage accumulation within the plaque in a rabbit model

Effects of candesartan on mortality and morbidity in patients with chronic heart failure: the CHARM-Overall programme.

Vitamin E and telmisartan attenuates doxorubicin induced cardiac injury in rat through down regulation of inflammatory response.

Feasibility of treating prehypertension with an angiotensin-receptor blocker.

The NF-kappa B signal transduction pathway in aortic endothelial cells is primed for activation in regions predisposed to atherosclerotic lesion formation.

Nuclear factor kappaB signaling in atherogenesis.

P304

381-387

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P31

scholarly article

P356

10.1111/1755-5922.12033

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P433

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P478

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P577

2013-12-01T00:00:00Z

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P698

23566285

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P921

dyslipidemias