Senescence is a Spi1-induced anti-proliferative mechanism in primary hematopoietic cells.

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Senescence is a Spi1-induced anti-proliferative mechanism in primary hematopoietic cells.

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2017 nî lūn-bûn

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Senescence is a Spi1-induced anti-proliferative mechanism in primary hematopoietic cells.

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Senescence is a Spi1-induced anti-proliferative mechanism in primary hematopoietic cells.

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Senescence is a Spi1-induced anti-proliferative mechanism in primary hematopoietic cells.

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Senescence is a Spi1-induced anti-proliferative mechanism in primary hematopoietic cells.

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Senescence is a Spi1-induced anti-proliferative mechanism in primary hematopoietic cells.

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Senescence is a Spi1-induced anti-proliferative mechanism in primary hematopoietic cells.

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P1476

Senescence is a Spi1-induced anti-proliferative mechanism in primary hematopoietic cells

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P2093

Christel Guillouf

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Cyril Quiveron

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Elena Mylonas

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Hengxiang Cui

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Laure Delestré

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Michela Esposito

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Oliver Bischof

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P2860

Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions

Cellular senescence: when bad things happen to good cells

Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpoints

Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication

BRAFE600-associated senescence-like cell cycle arrest of human naevi

Reduced PU.1 expression causes myeloid progenitor expansion and increased leukemia penetrance in mice expressing PML-RARalpha.

A large gene network in immature erythroid cells is controlled by the myeloid and B cell transcriptional regulator PU.1.

The Runx-PU.1 pathway preserves normal and AML/ETO9a leukemic stem cells.

Inactivation of PU.1 in adult mice leads to the development of myeloid leukemia

Epigenetic silencing of Bim transcription by Spi-1/PU.1 promotes apoptosis resistance in leukaemia

P304

1850-1860

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P31

scholarly article

P356

10.3324/HAEMATOL.2016.157636

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P433

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P478

102

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P50

Virginie Penard-Lacronique

P577

2017-09-14T00:00:00Z

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P698

28912174

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P932

5664389

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