Chemical preconditioning with 3-nitropropionic acid in hearts: role of mitochondrial K(ATP) channel.
about
Multiprotein complex containing succinate dehydrogenase confers mitochondrial ATP-sensitive K+ channel activityPhysiological consequences of complex II inhibition for aging, disease, and the mKATP channelMitochondrial toxin 3-nitropropionic acid induces cardiac and neurotoxicity differentially in miceCardioprotective mechanism of diazoxide involves the inhibition of succinate dehydrogenaseThe mitochondrial complex II and ATP-sensitive potassium channel interaction: quantitation of the channel in heart mitochondria.KATP channel openers have opposite effects on mitochondrial respiration under different energetic conditionsThe role of mitochondria in protection of the heart by preconditioning.Evidence for mitochondrial K+ channels and their role in cardioprotection.Neonatal exendin-4 leads to protection from reperfusion injury and reduced rates of oxidative phosphorylation in the adult rat heart.Mitochondrial K(ATP) channels in cell survival and death.Cardioprotection and mitochondrial S-nitrosation: effects of S-nitroso-2-mercaptopropionyl glycine (SNO-MPG) in cardiac ischemia-reperfusion injuryFatty acid nitroalkenes induce resistance to ischemic cardiac injury by modulating mitochondrial respiration at complex IIThe mitochondrial K(ATP) channel--fact or fiction?The endogenous mitochondrial complex II inhibitor malonate regulates mitochondrial ATP-sensitive potassium channels: implications for ischemic preconditioning.Inhibition of Succinate Dehydrogenase by Diazoxide Is Independent of the ATP-Sensitive Potassium Channel Subunit Sulfonylurea Type 1 Receptor.In vivo cardioprotection by S-nitroso-2-mercaptopropionyl glycineThe complex II inhibitor atpenin A5 protects against cardiac ischemia-reperfusion injury via activation of mitochondrial KATP channelsThe Slo(w) path to identifying the mitochondrial channels responsible for ischemic protection.The responses of HT22 cells to the blockade of mitochondrial complexes and potential protective effect of selenium supplementation.Activation of hypoxia-inducible factor-1 via prolyl-4 hydoxylase-2 gene silencing attenuates acute inflammatory responses in postischemic myocardium.Potassium channel openers protect cardiac mitochondria by attenuating oxidant stress at reoxygenation.Ketamine abolishes ischemic preconditioning through inhibition of K(ATP) channels in rabbit hearts.K(ATP) channel-independent targets of diazoxide and 5-hydroxydecanoate in the heart.The effects of ischaemic preconditioning, diazoxide and 5-hydroxydecanoate on rat heart mitochondrial volume and respiration.Targeting nucleotide-requiring enzymes: implications for diazoxide-induced cardioprotection.Cardioprotection afforded by chronic exercise is mediated by the sarcolemmal, and not the mitochondrial, isoform of the KATP channel in the rat.
P2860
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P2860
Chemical preconditioning with 3-nitropropionic acid in hearts: role of mitochondrial K(ATP) channel.
description
2001 nî lūn-bûn
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2001年の論文
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2001年学术文章
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2001年学术文章
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name
Chemical preconditioning with 3-nitropropionic acid in hearts: role of mitochondrial K
@nl
Chemical preconditioning with ...... mitochondrial K(ATP) channel.
@en
type
label
Chemical preconditioning with 3-nitropropionic acid in hearts: role of mitochondrial K
@nl
Chemical preconditioning with ...... mitochondrial K(ATP) channel.
@en
prefLabel
Chemical preconditioning with 3-nitropropionic acid in hearts: role of mitochondrial K
@nl
Chemical preconditioning with ...... mitochondrial K(ATP) channel.
@en
P2093
P2860
P1476
Chemical preconditioning with ...... mitochondrial K(ATP) channel.
@en
P2093
Bhargava P
Kukreja RC
Ockaili RA
P2860
P304
P356
10.1152/AJPHEART.2001.280.5.H2406
P577
2001-05-01T00:00:00Z