Potassium channel openers protect cardiac mitochondria by attenuating oxidant stress at reoxygenation.
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Cardiomyocyte Remodeling in Atrial Fibrillation and Hibernating Myocardium: Shared Pathophysiologic Traits Identify Novel Treatment Strategies?Diazoxide triggers cardioprotection against apoptosis induced by oxidative stress.Hypoxia-induced preconditioning in adult stimulated cardiomyocytes is mediated by the opening and trafficking of sarcolemmal KATP channelsCardioprotective effects of PKG activation by soluble GC activator, BAY 60-2770, in ischemia-reperfusion-injured rat hearts.Diazoxide, a K(ATP) channel opener, prevents ischemia-reperfusion injury in rodent pancreatic islets.Protection of cardiac mitochondria by diazoxide and protein kinase C: implications for ischemic preconditioning.Mitochondrial targets for volatile anesthetics against cardiac ischemia-reperfusion injuryCardiac mitochondria and arrhythmias.KATP channel openers have opposite effects on mitochondrial respiration under different energetic conditionsSuccinate metabolism: a new therapeutic target for myocardial reperfusion injury.The role of mitochondria in protection of the heart by preconditioning.GSK3beta inhibition and K(ATP) channel opening mediate acute opioid-induced cardioprotection at reperfusion.Cellular remodeling in heart failure disrupts K(ATP) channel-dependent stress toleranceRespiratory uncoupling by increased H(+) or K(+) flux is beneficial for heart mitochondrial turnover of reactive oxygen species but not for permeability transitionNicorandil in patients with acute myocardial infarction undergoing primary percutaneous coronary intervention: a systematic review and meta-analysis.Accelerated recovery of mitochondrial membrane potential by GSK-3β inactivation affords cardiomyocytes protection from oxidant-induced necrosis.Nicorandil prevents endothelial dysfunction due to antioxidative effects via normalisation of NADPH oxidase and nitric oxide synthase in streptozotocin diabetic rats.Evidence for mitochondrial K+ channels and their role in cardioprotection.Nicorandil protects mesenchymal stem cells against hypoxia and serum deprivation-induced apoptosis.Uncoupling protein-2 expression and effects on mitochondrial membrane potential and oxidant stress in heart tissue.Altered expression of mitochondrial electron transport chain proteins and improved myocardial energetic state during late ischemic preconditioning.Mitochondrial K(ATP) channels in cell survival and death.Biochemical dysfunction in heart mitochondria exposed to ischaemia and reperfusion.Cytoprotective channels in mitochondria.The endogenous mitochondrial complex II inhibitor malonate regulates mitochondrial ATP-sensitive potassium channels: implications for ischemic preconditioning.Role of uncoupling protein 3 in ischemia-reperfusion injury, arrhythmias, and preconditioning.Complex I and ATP synthase mediate membrane depolarization and matrix acidification by isoflurane in mitochondriaMechanism of Mitochondrial Connexin43's Protection of the Neurovascular Unit under Acute Cerebral Ischemia-Reperfusion Injury.Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation.Role of mitochondrial dysfunction in insulin resistance.Myocardial Hsp70 phosphorylation and PKC-mediated cardioprotection following exercise.The mitochondrial permeability transition pore as a target for preconditioning and postconditioning.Mitochondrial involvement in cardiac apoptosis during ischemia and reperfusion: can we close the box?Metabolic fingerprint of ischaemic cardioprotection: importance of the malate-aspartate shuttle.Inhibitors of succinate: quinone reductase/Complex II regulate production of mitochondrial reactive oxygen species and protect normal cells from ischemic damage but induce specific cancer cell death.Diazoxide-induced respiratory inhibition - a putative mitochondrial K(ATP) channel independent mechanism of pharmacological preconditioning.Mitochondrial ATP-sensitive K+ channels prevent oxidative stress, permeability transition and cell death.The effect of the mitochondrial permeability transition pore on apoptosis in Eimeria tenella host cells.Mitochondrial K(ATP) channel openers activate the ERK kinase by an oxidant-dependent mechanism.Diazoxide reverses the enhanced expression of KATP subunits in cholinergic neurons caused by exposure to Aβ₁₋₄₂.
P2860
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P2860
Potassium channel openers protect cardiac mitochondria by attenuating oxidant stress at reoxygenation.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Potassium channel openers prot ...... idant stress at reoxygenation.
@en
Potassium channel openers prot ...... idant stress at reoxygenation.
@nl
type
label
Potassium channel openers prot ...... idant stress at reoxygenation.
@en
Potassium channel openers prot ...... idant stress at reoxygenation.
@nl
prefLabel
Potassium channel openers prot ...... idant stress at reoxygenation.
@en
Potassium channel openers prot ...... idant stress at reoxygenation.
@nl
P2093
P2860
P1476
Potassium channel openers prot ...... idant stress at reoxygenation.
@en
P2093
Andre Terzic
Cevher Ozcan
Martin Bienengraeber
Petras P Dzeja
P2860
P304
P356
10.1152/AJPHEART.00552.2001
P577
2002-02-01T00:00:00Z