In vivo key role of reactive oxygen species and NHE-1 activation in determining excessive cardiac hypertrophy.
about
The autocrine/paracrine loop after myocardial stretch: mineralocorticoid receptor activationRoles of Na⁺/H⁺ exchanger type 1 and intracellular pH in angiotensin II-induced reactive oxygen species generation and podocyte apoptosis.Transient receptor potential channels contribute to pathological structural and functional remodeling after myocardial infarctionThe metabolic syndrome, oxidative stress, environment, and cardiovascular disease: the great exploration.Exercise Attenuates Intermittent Hypoxia-Induced Cardiac Fibrosis Associated with Sodium-Hydrogen Exchanger-1 in RatsPressure-volume relation analysis of mouse ventricular function.Cardiovascular function in male and female JCR:LA-cp rats: effect of high-fat/high-sucrose diet.Uric Acid and New Onset Left Ventricular Hypertrophy: Findings From the PAMELA Population.Inhibition of carbonic anhydrase prevents the Na(+)/H(+) exchanger 1-dependent slow force response to rat myocardial stretch.
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In vivo key role of reactive oxygen species and NHE-1 activation in determining excessive cardiac hypertrophy.
description
2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年學術文章
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2011年學術文章
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name
In vivo key role of reactive o ...... excessive cardiac hypertrophy.
@en
In vivo key role of reactive o ...... excessive cardiac hypertrophy.
@nl
type
label
In vivo key role of reactive o ...... excessive cardiac hypertrophy.
@en
In vivo key role of reactive o ...... excessive cardiac hypertrophy.
@nl
prefLabel
In vivo key role of reactive o ...... excessive cardiac hypertrophy.
@en
In vivo key role of reactive o ...... excessive cardiac hypertrophy.
@nl
P2093
P2860
P50
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P1476
In vivo key role of reactive o ...... excessive cardiac hypertrophy.
@en
P2093
Eduardo M Escudero
Gloria Cónsole
María C Alvarez
Néstor G Pérez
Oscar H Cingolani
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P2888
P304
P356
10.1007/S00424-011-1020-8
P577
2011-08-26T00:00:00Z