about
Tissue-resident macrophagesInnate and adaptive type 2 immune cell responses in genetically controlled resistance to intestinal helminth infectionDendritic cells activated with products released by schistosome larvae drive Th2-type immune responses, which can be inhibited by manipulation of CD40 costimulation.Alternatively activated macrophages derived from monocytes and tissue macrophages are phenotypically and functionally distinct.Similarity and diversity in macrophage activation by nematodes, trematodes, and cestodes.Unconventional maturation of dendritic cells induced by particles from the laminated layer of larval Echinococcus granulosusLocal macrophage proliferation, rather than recruitment from the blood, is a signature of TH2 inflammation.The adult murine heart has a sparse, phagocytically active macrophage population that expands through monocyte recruitment and adopts an 'M2' phenotype in response to Th2 immunologic challenge.Schistosome larvae stimulate macrophage cytokine production through TLR4-dependent and -independent pathways.Alternatively activated dendritic cells regulate CD4+ T-cell polarization in vitro and in vivoMacrophage origin limits functional plasticity in helminth-bacterial co-infectionCSF1 Restores Innate Immunity After Liver Injury in Mice and Serum Levels Indicate Outcomes of Patients With Acute Liver Failure.Dendritic cell activation and function in response to Schistosoma mansoni.IL-4 directly signals tissue-resident macrophages to proliferate beyond homeostatic levels controlled by CSF-1Distinct bone marrow-derived and tissue-resident macrophage lineages proliferate at key stages during inflammation.Homeostasis in the mononuclear phagocyte system.Induction of IL-4Rα-dependent microRNAs identifies PI3K/Akt signaling as essential for IL-4-driven murine macrophage proliferation in vivo.Distinct sources and targets of IL-10 during dendritic cell-driven Th1 and Th2 responses in vivo.A pivotal role for CD40-mediated IL-6 production by dendritic cells during IL-17 induction in vivo.Full development of Th2 immunity requires both innate and adaptive sources of CD154.Csf1r-mApple Transgene Expression and Ligand Binding In Vivo Reveal Dynamics of CSF1R Expression within the Mononuclear Phagocyte System.The Mannose Receptor (CD206) is an important pattern recognition receptor (PRR) in the detection of the infective stage of the helminth Schistosoma mansoni and modulates IFNγ production.Interleukin-4 activated macrophages mediate immunity to filarial helminth infection by sustaining CCR3-dependent eosinophilia.Local proliferation of monocytesDeletion of a Csf1r enhancer selectively impacts CSF1R expression and development of tissue macrophage populationsRate of replenishment and microenvironment contribute to the sexually dimorphic phenotype and function of peritoneal macrophages
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description
hulumtues
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researcher
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wetenschapper
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հետազոտող
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name
Stephen J Jenkins
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Stephen J Jenkins
@en
Stephen J Jenkins
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Stephen J Jenkins
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Stephen J Jenkins
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type
label
Stephen J Jenkins
@ast
Stephen J Jenkins
@en
Stephen J Jenkins
@es
Stephen J Jenkins
@nl
Stephen J Jenkins
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prefLabel
Stephen J Jenkins
@ast
Stephen J Jenkins
@en
Stephen J Jenkins
@es
Stephen J Jenkins
@nl
Stephen J Jenkins
@sl
P106
P21
P31
P496
0000-0002-0233-5424