IL-4 directly signals tissue-resident macrophages to proliferate beyond homeostatic levels controlled by CSF-1
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How Mouse Macrophages Sense What Is Going On.The development and maintenance of resident macrophagesMacrophages in vascular inflammation--From atherosclerosis to vasculitisMacrophage heterogeneity in tissues: phenotypic diversity and functionsMechanisms driving macrophage diversity and specialization in distinct tumor microenvironments and parallelisms with other tissuesMacrophage proliferation, provenance, and plasticity in macroparasite infectionProximal tubule-derived colony stimulating factor-1 mediates polarization of renal macrophages and dendritic cells, and recovery in acute kidney injuryImmune Modulation of Cardiac Repair and Regeneration: The Art of Mending Broken HeartsImmunity to gastrointestinal nematodes: mechanisms and mythsInnate and adaptive type 2 immune cell responses in genetically controlled resistance to intestinal helminth infectionM-CSF Mediates Host Defense during Bacterial Pneumonia by Promoting the Survival of Lung and Liver Mononuclear Phagocytes.Antibody-mediated trapping of helminth larvae requires CD11b and Fcγ receptor I.Alternatively activated macrophages derived from monocytes and tissue macrophages are phenotypically and functionally distinct.Mycobacterium tuberculosis arrests host cycle at the G1/S transition to establish long term infectionThe journey from stem cell to macrophage.Gata6 regulates aspartoacylase expression in resident peritoneal macrophages and controls their survival.Lineage-specific enhancers activate self-renewal genes in macrophages and embryonic stem cellsThe MacBlue binary transgene (csf1r-gal4VP16/UAS-ECFP) provides a novel marker for visualisation of subsets of monocytes, macrophages and dendritic cells and responsiveness to CSF1 administration.Cell-intrinsic lysosomal lipolysis is essential for alternative activation of macrophagesLy6Chi monocyte recruitment is responsible for Th2 associated host-protective macrophage accumulation in liver inflammation due to schistosomiasis.A question of persistence: Langerhans cells and graft-versus-host diseaseIncomplete deletion of IL-4Rα by LysM(Cre) reveals distinct subsets of M2 macrophages controlling inflammation and fibrosis in chronic schistosomiasis.From Monocytes to M1/M2 Macrophages: Phenotypical vs. Functional Differentiation.Dynamic changes in macrophage activation and proliferation during the development and resolution of intestinal inflammation.Macrophages in Tissue Repair, Regeneration, and Fibrosis.Regulation of macrophage development and function in peripheral tissuesOntogeny and polarization of macrophages in inflammation: blood monocytes versus tissue macrophagesThe development and maintenance of the mononuclear phagocyte system of the chick is controlled by signals from the macrophage colony-stimulating factor receptorMacrophage gene expression associated with remodeling of the prepartum rat cervix: microarray and pathway analyses.Myocardial infarction: a critical role of macrophages in cardiac remodeling.Cytokine profiling of docetaxel-resistant castration-resistant prostate cancer.Characterisation of a novel Fc conjugate of macrophage colony-stimulating factor.Much More than M1 and M2 Macrophages, There are also CD169(+) and TCR(+) Macrophages.The adult murine heart has a sparse, phagocytically active macrophage population that expands through monocyte recruitment and adopts an 'M2' phenotype in response to Th2 immunologic challenge.Thrombospondin-1 (TSP1) contributes to the development of vascular inflammation by regulating monocytic cell motility in mouse models of abdominal aortic aneurysm.Development of patent Litomosoides sigmodontis infections in semi-susceptible C57BL/6 mice in the absence of adaptive immune responses.Redefining the transcriptional regulatory dynamics of classically and alternatively activated macrophages by deepCAGE transcriptomics.A unique role for p53 in the regulation of M2 macrophage polarizationMacrophage Colony Stimulating Factor Derived from CD4+ T Cells Contributes to Control of a Blood-Borne InfectionAlternatively activated macrophages promote pancreatic fibrosis in chronic pancreatitis.
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IL-4 directly signals tissue-resident macrophages to proliferate beyond homeostatic levels controlled by CSF-1
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 07 October 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
IL-4 directly signals tissue-r ...... tic levels controlled by CSF-1
@en
IL-4 directly signals tissue-r ...... ic levels controlled by CSF-1.
@nl
type
label
IL-4 directly signals tissue-r ...... tic levels controlled by CSF-1
@en
IL-4 directly signals tissue-r ...... ic levels controlled by CSF-1.
@nl
prefLabel
IL-4 directly signals tissue-r ...... tic levels controlled by CSF-1
@en
IL-4 directly signals tissue-r ...... ic levels controlled by CSF-1.
@nl
P2093
P2860
P50
P356
P1476
IL-4 directly signals tissue-r ...... tic levels controlled by CSF-1
@en
P2093
Frank Brombacher
Rick M Maizels
Sheelagh Duncan
P2860
P304
P356
10.1084/JEM.20121999
P407
P577
2013-10-07T00:00:00Z