Lack of the burst firing of thalamocortical relay neurons and resistance to absence seizures in mice lacking alpha(1G) T-type Ca(2+) channels.
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A specific tryptophan in the I-II linker is a key determinant of beta-subunit binding and modulation in Ca(V)2.3 calcium channelsSpecific contribution of human T-type calcium channel isotypes (alpha(1G), alpha(1H) and alpha(1I)) to neuronal excitabilityMolecular targets for antiepileptic drug developmentThe Physiology, Pathology, and Pharmacology of Voltage-Gated Calcium Channels and Their Future Therapeutic PotentialSignal processing by T-type calcium channel interactions in the cerebellumCaV3.2 calcium channels control NMDA receptor-mediated transmission: a new mechanism for absence epilepsy.A Recurrent Mutation in CACNA1G Alters Cav3.1 T-Type Calcium-Channel Conduction and Causes Autosomal-Dominant Cerebellar AtaxiaA mutation in the low voltage-gated calcium channel CACNA1G alters the physiological properties of the channel, causing spinocerebellar ataxiaGating effects of mutations in the Cav3.2 T-type calcium channel associated with childhood absence epilepsyVoltage-gated calcium channelsNeuronal T-type calcium channels: what's new? Iftinca: T-type channel regulationThe neurobiology of antiepileptic drugsMolecular basis of R-type calcium channels in central amygdala neurons of the mouseEnhanced expression of L-type Cav1.3 calcium channels in murine embryonic hearts from Cav1.2-deficient micePresence of a Chaotic Region at the Sleep-Wake Transition in a Simplified Thalamocortical Circuit ModelContrasting the roles of the I-II loop gating brake in CaV3.1 and CaV3.3 calcium channels.T-type calcium channel blockers as neuroprotective agents.T-type voltage-gated calcium channels as targets for the development of novel pain therapies.The voltage dependence of gating currents of the neuronal CA(v)3.3 channel is determined by the gating brake in the I-II loopMechanisms of inhibition of CaV3.1 T-type calcium current by aliphatic alcoholsAlternative splicing within the I-II loop controls surface expression of T-type Ca(v)3.1 calcium channels.The potential roles of T-type Ca2+ channels in motor coordination.Thalamic Cav3.1 T-type Ca2+ channel plays a crucial role in stabilizing sleepAbsence seizures in C3H/HeJ and knockout mice caused by mutation of the AMPA receptor subunit Gria4Genetic enhancement of thalamocortical network activity by elevating alpha 1g-mediated low-voltage-activated calcium current induces pure absence epilepsy.Heterozygous mutations of the voltage-gated sodium channel SCN8A are associated with spike-wave discharges and absence epilepsy in mice.PICK1 uncoupling from mGluR7a causes absence-like seizures.Deletion of phospholipase C beta4 in thalamocortical relay nucleus leads to absence seizures.Ca(V)3.1 is a tremor rhythm pacemaker in the inferior oliveAlpha1G T-type calcium channel selectively regulates P-selectin surface expression in pulmonary capillary endothelium.Confirmation of an epilepsy modifier locus on mouse chromosome 11 and candidate gene analysis by RNA-Seq.T-type channel blockade impairs long-term potentiation at the parallel fiber-Purkinje cell synapse and cerebellar learning.Characterization and regulation of T-type Ca2+ channels in embryonic stem cell-derived cardiomyocytes.Childhood absence epilepsy: genes, channels, neurons and networks.Cingulate seizure-like activity reveals neuronal avalanche regulated by network excitability and thalamic inputs.Lack of delta waves and sleep disturbances during non-rapid eye movement sleep in mice lacking alpha1G-subunit of T-type calcium channels.17Beta-estradiol regulation of the mRNA expression of T-type calcium channel subunits: role of estrogen receptor alpha and estrogen receptor beta.Transient and big are key features of an invertebrate T-type channel (LCav3) from the central nervous system of Lymnaea stagnalisActivity of cortical and thalamic neurons during the slow (<1 Hz) rhythm in the mouse in vivo.Inhibition of the activation pathway of the T-type calcium channel Ca(V)3.1 by ProTxII.
P2860
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P2860
Lack of the burst firing of thalamocortical relay neurons and resistance to absence seizures in mice lacking alpha(1G) T-type Ca(2+) channels.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
Lack of the burst firing of th ...... seizures in mice lacking alpha
@nl
Lack of the burst firing of th ...... ha(1G) T-type Ca(2+) channels.
@en
type
label
Lack of the burst firing of th ...... seizures in mice lacking alpha
@nl
Lack of the burst firing of th ...... ha(1G) T-type Ca(2+) channels.
@en
prefLabel
Lack of the burst firing of th ...... seizures in mice lacking alpha
@nl
Lack of the burst firing of th ...... ha(1G) T-type Ca(2+) channels.
@en
P2093
P1433
P1476
Lack of the burst firing of th ...... ha(1G) T-type Ca(2+) channels.
@en
P2093
McEnery MW
P356
10.1016/S0896-6273(01)00343-9
P407
P577
2001-07-01T00:00:00Z