Neuronal and glial coexpression of argininosuccinate synthetase and inducible nitric oxide synthase in Alzheimer disease. - Wikidata - awgldk - TriplyDB

Neuronal and glial coexpression of argininosuccinate synthetase and inducible nitric oxide synthase in Alzheimer disease.

Neuronal and glial coexpression of argininosuccinate synthetase and inducible nitric oxide synthase in Alzheimer disease.

http://www.wikidata.org/entity/Q43736972

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Apolipoprotein J (clusterin) activates rodent microglia in vivo and in vitro Glycogen synthase kinase-3 (GSK3): inflammation, diseases, and therapeutics Glial Tau Pathology in Tauopathies: Functional Consequences Innate Immune Response in Brain, NF-Kappa B Signaling and Cystatins Glial cells in (patho)physiology Age-dependent neuroinflammation and cognitive decline in a novel Ala152Thr-Tau transgenic mouse model of PSP and AD.Alzheimer Disease: Crosstalk between the Canonical Wnt/Beta-Catenin Pathway and PPARs Alpha and Gamma Argininosuccinate synthetase from the urea cycle to the citrulline-NO cycle The Potassium Channel KCa3.1 Represents a Valid Pharmacological Target for Astrogliosis-Induced Neuronal Impairment in a Mouse Model of Alzheimer’s Disease Activated microglia modulate astroglial enzymes involved in oxidative and inflammatory stress and increase the resistance of astrocytes to oxidative stress in vitro CAT2 arginine transporter deficiency significantly reduces iNOS-mediated NO production in astrocytes Uncovering molecular biomarkers that correlate cognitive decline with the changes of hippocampus' gene expression profiles in Alzheimer's disease.Detailed immunohistochemical characterization of temporal and spatial progression of Alzheimer's disease-related pathologies in male triple-transgenic mice Pleiotropic effects in Eya3 knockout mice.MRP14 (S100A9) protein interacts with Alzheimer beta-amyloid peptide and induces its fibrillization Nitric oxide activity and isoenzyme expression in the senescence-accelerated mouse p8 model of Alzheimer's disease: effects of anti-amyloid antibody and antisense treatments Expression profile and down-regulation of argininosuccinate synthetase in hepatocellular carcinoma in a transgenic mouse model Radix Scrophulariae extracts (harpagoside) suppresses hypoxia-induced microglial activation and neurotoxicity.Vascular oxidative stress in Alzheimer disease Oxidative and nitrative injury in periventricular leukomalacia: a review.Inflammatory changes parallel the early stages of Alzheimer disease.Protection from Alzheimer's-like disease in the mouse by genetic ablation of inducible nitric oxide synthase.Amyloid-β peptide-induced extracellular S100A9 depletion is associated with decrease of antimicrobial peptide activity in human THP-1 monocytes.Evidence of endothelial dysfunction in the development of Alzheimer's disease: Is Alzheimer's a vascular disorder?SUMO and Alzheimer's disease.Alzheimer's mechanisms in ischemic brain degeneration.The Influence of Vitamin D Treatment on the Inducible Nitric Oxide Synthase (INOS) Expression in Primary Hippocampal Neurons.Neuroinflammatory processes in Alzheimer's disease.Neuroprotective mechanisms of peroxisome proliferator-activated receptor agonists in Alzheimer's disease.L-arginine and nitric oxide in CNS function and neurodegenerative diseases.Innate immune activation in neurodegenerative disease.Truncated and modified amyloid-beta species.Inflammation in Alzheimer's Disease and Molecular Genetics: Recent Update.The Brain NO Levels and NOS Activities Ascended in the Early and Middle Stages and Descended in the Terminal Stage in Scrapie-Infected Animal Models.PPARs in Alzheimer's Disease.Impact and Therapeutic Potential of PPARs in Alzheimer's Disease.Modulation of inducible nitric oxide synthase expression by sumoylation.Accelerated neurodegeneration and neuroinflammation in transgenic mice expressing P301L tau mutant and tau-tubulin kinase 1.Immunocytochemical characterization of Alzheimer disease hallmarks in APP/PS1 transgenic mice treated with a new anti-amyloid-β vaccine.Noradrenergic depletion increases inflammatory responses in brain: effects on IkappaB and HSP70 expression.

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P2860

Neuronal and glial coexpression of argininosuccinate synthetase and inducible nitric oxide synthase in Alzheimer disease.

http://www.wikidata.org/entity/Q43736972

description

2001 nî lūn-bûn

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2001年の論文

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年學術文章

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2001年學術文章

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2001年學術文章

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name

Neuronal and glial coexpressio ...... synthase in Alzheimer disease.

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Neuronal and glial coexpressio ...... synthase in Alzheimer disease.

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type

label

Neuronal and glial coexpressio ...... synthase in Alzheimer disease.

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Neuronal and glial coexpressio ...... synthase in Alzheimer disease.

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prefLabel

Neuronal and glial coexpressio ...... synthase in Alzheimer disease.

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Neuronal and glial coexpressio ...... synthase in Alzheimer disease.

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Journal of Neuropathology & Experimental Neurology

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Neuronal and glial coexpressio ...... synthase in Alzheimer disease.

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P2093

Dumitrescu-Ozimek L

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Feinstein DL

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Heneka MT

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Klockgether T

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Riederer P

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Wiesinger H

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P2860

Neuropathological stageing of Alzheimer-related changes

Inflammation and Alzheimer's disease

Induction of astrocyte argininosuccinate synthetase and argininosuccinate lyase by dibutyryl cyclic AMP and dexamethasone

Neuroinflammatory processes are important in neurodegenerative diseases: an hypothesis to explain the increased formation of reactive oxygen and nitrogen species as major factors involved in neurodegenerative disease development.

Brain interleukin 1 and S-100 immunoreactivity are elevated in Down syndrome and Alzheimer disease

Regulation of citrulline recycling in nitric oxide-dependent neurotransmission in the murine proximal colon.

Amyloid beta-peptide stimulates nitric oxide production in astrocytes through an NFkappaB-dependent mechanism.

Inducible nitric oxide synthase in tangle-bearing neurons of patients with Alzheimer's disease.

Presence of argininosuccinate synthetase in glial cells as revealed by peptide-specific antisera.

Tetrahydrobiopterin biosynthesis in C6 glioma cells: induction of GTP cyclohydrolase I gene expression by lipopolysaccharide and cytokine treatment.

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Alzheimer's disease