Inflammatory changes parallel the early stages of Alzheimer disease.
about
Brain Insulin Resistance at the Crossroads of Metabolic and Cognitive Disorders in HumansDocosahexaenoic Acid and Cognition throughout the LifespanHerpes Simplex Virus Type 1 and Other Pathogens are Key Causative Factors in Sporadic Alzheimer's DiseaseGene expression parallels synaptic excitability and plasticity changes in Alzheimer's diseaseThe airbag problem-a potential culprit for bench-to-bedside translational efforts: relevance for Alzheimer's diseaseCommon transcriptional signatures in brain tissue from patients with HIV-associated neurocognitive disorders, Alzheimer's disease, and Multiple SclerosisReduced β-amyloid pathology in an APP transgenic mouse model of Alzheimer's disease lacking functional B and T cellsMolecular mechanisms of cognitive dysfunction following traumatic brain injuryGain in brain immunity in the oldest-old differentiates cognitively normal from demented individualsLoss of MAP function leads to hippocampal synapse loss and deficits in the Morris Water Maze with agingAging effects on DNA methylation modules in human brain and blood tissue.Blocking IL-1 signaling rescues cognition, attenuates tau pathology, and restores neuronal β-catenin pathway function in an Alzheimer's disease model.Effects of long-term pioglitazone treatment on peripheral and central markers of aging.Reductions in amyloid-beta-derived neuroinflammation, with minocycline, restore cognition but do not significantly affect tau hyperphosphorylation.Presenilin/gamma-Secretase and Inflammation.Late-onset Alzheimer's disease, heating up and foxed by several proteins: pathomolecular effects of the aging process.Altered expression of diabetes-related genes in Alzheimer's disease brains: the Hisayama study.Gene expression reveals overlap between normal aging and Alzheimer's disease genesNeuroimmune mechanisms of cytokine-induced depression: current theories and novel treatment strategies.Transcriptome meta-analysis reveals a central role for sex steroids in the degeneration of hippocampal neurons in Alzheimer's diseaseThe inflammatory response in Alzheimer's disease.Aging-related gene expression in hippocampus proper compared with dentate gyrus is selectively associated with metabolic syndrome variables in rhesus monkeys.Cattle encephalon glycoside and ignotin injection improves cognitive impairment in APPswe/PS1dE9 mice used as multitarget anti-Alzheimer's drug candidatesAstrocytes are important mediators of Aβ-induced neurotoxicity and tau phosphorylation in primary culture.Ablation of TNF-RI/RII expression in Alzheimer's disease mice leads to an unexpected enhancement of pathology: implications for chronic pan-TNF-α suppressive therapeutic strategies in the brain.The triggering receptor expressed on myeloid cells 2 (TREM2) is associated with enhanced inflammation, neuropathological lesions and increased risk for Alzheimer's dementiaMHC class I limits hippocampal synapse density by inhibiting neuronal insulin receptor signalingAmyloid-β oligomers induce differential gene expression in adult human brain slices.Reelin-mediated Signaling during Normal and Pathological Forms of AgingMinocycline corrects early, pre-plaque neuroinflammation and inhibits BACE-1 in a transgenic model of Alzheimer's disease-like amyloid pathology.Arizona Study of Aging and Neurodegenerative Disorders and Brain and Body Donation Program.Indomethacin-loaded lipid-core nanocapsules reduce the damage triggered by Aβ1-42 in Alzheimer's disease models.Transcriptional alterations related to neuropathology and clinical manifestation of Alzheimer's disease.Expression of CD74 is increased in neurofibrillary tangles in Alzheimer's disease.Inflammation and clinical presentation in neurodegenerative disease: a volatile relationship.Biomarkers for early diagnosis of Alzheimer disease: 'ALZheimer ASsociated gene'--a new blood biomarker?Transcriptional vulnerability of brain regions in Alzheimer's disease and dementiaRole of Mitochondria-Associated Endoplasmic Reticulum Membrane in Inflammation-Mediated Metabolic DiseasesInflammation in Alzheimer's disease: relevance to pathogenesis and therapyChloride intracellular channel 1 (CLIC1): Sensor and effector during oxidative stress.
P2860
Q26739671-B3E92618-6E90-463D-BE50-67C687FF7C25Q26768442-3EC1F319-F7F6-48AF-9FD0-C24B931EF8B9Q26781578-B14DF3AC-3B65-47AC-AB62-7267519511B0Q26785375-708A2383-BDDC-4C69-995F-3810496A0626Q26991654-60CC35F8-F769-4F5F-9B86-74CADE9464FDQ27007041-55EF3002-3C50-4370-822D-EEAB9391AB1CQ27317159-DB1AD7ED-7B2A-48D7-B97C-1F6946FA1C5AQ28385324-9137FEF6-6364-49E7-9D26-6781E412C895Q33513475-FB31B98C-01CD-4484-AC4C-459756E1E677Q33639450-D0A60F5C-BD31-44B4-BF6E-46EA6B381800Q33741734-F4778C58-D940-486D-A4D0-89DAAF0DA72AQ33810933-6A8B8890-E5AC-42D3-B637-A2A59FC32674Q33818388-4B5E7B34-F171-4397-91B2-1670A813AE62Q33856674-B7DF6179-A9C4-4A9E-8CC9-8F985B3C4C31Q33918695-51E18C2E-3355-4FDE-9A53-7433FE507170Q34020079-9A2CD12E-4A17-4FD8-B2C8-71E59C9DF384Q34027810-35DDE24C-61AC-4295-B452-B9FFAB85C1E6Q34154632-9D69F798-531B-450F-A699-95F73B563ECDQ34369407-24428BC1-B48F-4C0D-9B4E-03E16A892EAEQ34784966-50CA75D9-2466-416C-B1C8-4929D9FB0154Q34803247-8AA26084-3E87-413B-B3A6-20B22C67F10BQ35161815-5A4FE3A7-3744-48A8-8DF9-4C75EC8351B2Q35167977-18B04E8E-DCEF-4663-B943-CBCBA8FFC3A6Q35202286-D1D23126-B01A-4941-AE26-2E00CA8EDC6DQ35235768-0DEE8C0D-B8A2-451D-A043-01492BB41866Q35706990-5B0582EF-A66E-4879-A50C-D75434EF2F4AQ35744568-72F0E110-F2E0-419A-96C7-8592D43A7119Q35802078-70C327D2-4660-467C-B752-2854D75677B6Q35806690-B3C6AD3D-ECEA-4A10-A039-01CA898934E7Q35959633-DA3AD540-0BF9-43B6-99FB-4D0F6B479B0BQ36121377-E28AA9D1-C232-41EB-B226-EC324C501EA6Q36247082-A131A743-EF41-4D16-A8E8-C6A978E30574Q36381106-CE63E0C8-B6B2-42CB-8E79-1FCEDF63F18BQ36932516-E261664A-3B2C-47B4-8DD5-26E5832BC64AQ37069901-0F44D7A9-D99E-46EA-BC71-E3A6658CDCEDQ37118083-47C03ABA-9132-4019-B7A6-A50FA887824DQ37351485-40B3D469-C786-4DEB-AEFA-015BBC4F12D0Q37542677-FFB3784F-B2FA-435A-9F8E-9C24F95BC603Q37685285-14176184-D910-4327-95E1-7533071424E4Q37729392-2FF38E15-9CB0-4B3D-BEFE-5D6E5E3D770E
P2860
Inflammatory changes parallel the early stages of Alzheimer disease.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
Inflammatory changes parallel the early stages of Alzheimer disease.
@ast
Inflammatory changes parallel the early stages of Alzheimer disease.
@en
type
label
Inflammatory changes parallel the early stages of Alzheimer disease.
@ast
Inflammatory changes parallel the early stages of Alzheimer disease.
@en
prefLabel
Inflammatory changes parallel the early stages of Alzheimer disease.
@ast
Inflammatory changes parallel the early stages of Alzheimer disease.
@en
P2093
P2860
P1476
Inflammatory changes parallel the early stages of Alzheimer disease.
@en
P2093
A Parachikova
C W Cotman
D H Cribbs
M Blurton-Jones
M G Agadjanyan
P2860
P304
P356
10.1016/J.NEUROBIOLAGING.2006.08.014
P577
2006-10-18T00:00:00Z