Arginase-1 expression in granulomas of tuberculosis patients.
about
Nitric oxide synthase: non-canonical expression patternsIL-4Rα-dependent alternative activation of macrophages is not decisive for Mycobacterium tuberculosis pathology and bacterial burden in miceTH1/TH2 paradigm extended: macrophage polarization as an unappreciated pathogen-driven escape mechanism?Gr1(int)CD11b+ myeloid-derived suppressor cells in Mycobacterium tuberculosis infectionGrowth factor and Th2 cytokine signaling pathways converge at STAT6 to promote arginase expression in progressive experimental visceral leishmaniasisMacrophage arginase-1 controls bacterial growth and pathology in hypoxic tuberculosis granulomas.Manipulation of the mononuclear phagocyte system by Mycobacterium tuberculosis.L-arginine and vitamin D adjunctive therapies in pulmonary tuberculosis: a randomised, double-blind, placebo-controlled trial.Immunology studies in non-human primate models of tuberculosisIn Situ Characterization of Splenic Brucella melitensis Reservoir Cells during the Chronic Phase of Infection in Susceptible MiceHelminth-induced arginase-1 exacerbates lung inflammation and disease severity in tuberculosisImpaired pulmonary nitric oxide bioavailability in pulmonary tuberculosis: association with disease severity and delayed mycobacterial clearance with treatment.Microenvironments in tuberculous granulomas are delineated by distinct populations of macrophage subsets and expression of nitric oxide synthase and arginase isoforms.RON receptor tyrosine kinase, a negative regulator of inflammation, is decreased during simian immunodeficiency virus-associated central nervous system diseaseType I IFN Inhibits Alternative Macrophage Activation during Mycobacterium tuberculosis Infection and Leads to Enhanced Protection in the Absence of IFN-γ Signaling.Shortening the 'short-course' therapy- insights into host immunity may contribute to new treatment strategies for tuberculosis.Immunometabolism within the tuberculosis granuloma: amino acids, hypoxia, and cellular respiration.Emerging trends in the formation and function of tuberculosis granulomas.Phenotypically resembling myeloid derived suppressor cells are increased in children with HIV and exposed/infected with Mycobacterium tuberculosis.Tuberculosis State Is Associated with Expression of Toll-Like Receptor 2 in Sputum Macrophages.Virulent Factor ESAT-6 Drives Macrophage Differentiation Toward the Pro-inflammatory M1 Phenotype and Subsequently Switches It to the Anti-inflammatory M2 PhenotypeModulating Iron for Metabolic Support of TB Host Defense
P2860
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P2860
Arginase-1 expression in granulomas of tuberculosis patients.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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2012年學術文章
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name
Arginase-1 expression in granulomas of tuberculosis patients.
@en
Arginase-1 expression in granulomas of tuberculosis patients.
@nl
type
label
Arginase-1 expression in granulomas of tuberculosis patients.
@en
Arginase-1 expression in granulomas of tuberculosis patients.
@nl
prefLabel
Arginase-1 expression in granulomas of tuberculosis patients.
@en
Arginase-1 expression in granulomas of tuberculosis patients.
@nl
P2093
P2860
P1476
Arginase-1 expression in granulomas of tuberculosis patients
@en
P2093
Albanita Vianna
Ana L Mattos-Guaraldi
Ana P Pessanha
Lilian O Moreira
P2860
P304
P356
10.1111/J.1574-695X.2012.01012.X
P577
2012-08-21T00:00:00Z