Upregulation of a T-type Ca2+ channel causes a long-lasting modification of neuronal firing mode after status epilepticus.
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The 'window' T-type calcium current in brain dynamics of different behavioural statesAcute alterations of somatodendritic action potential dynamics in hippocampal CA1 pyramidal cells after kainate-induced status epilepticus in miceZinc regulates a key transcriptional pathway for epileptogenesis via metal-regulatory transcription factor 1.Ion channels in genetic and acquired forms of epilepsy.Homeostasis or channelopathy? Acquired cell type-specific ion channel changes in temporal lobe epilepsy and their antiepileptic potentialNeuronal T-type calcium channels: what's new? Iftinca: T-type channel regulationZ944, a Novel Selective T-Type Calcium Channel Antagonist Delays the Progression of Seizures in the Amygdala Kindling ModelRole of voltage-gated calcium channels in epilepsy.Characterization of the gating brake in the I-II loop of CaV3 T-type calcium channels.I-II loop structural determinants in the gating and surface expression of low voltage-activated calcium channels.Massive and specific dysregulation of direct cortical input to the hippocampus in temporal lobe epilepsy.Calcium channel dysfunction in inferior colliculus neurons of the genetically epilepsy-prone ratConcepts of connectivity and human epileptic activityACTH induces Cav3.2 current and mRNA by cAMP-dependent and cAMP-independent mechanisms.Presynaptic inhibitory terminals are functionally abnormal in a rat model of posttraumatic epilepsyWONOEP appraisal: new genetic approaches to study epilepsyAltered expression and function of small-conductance (SK) Ca(2+)-activated K+ channels in pilocarpine-treated epileptic ratsOptogenetic and potassium channel gene therapy in a rodent model of focal neocortical epilepsy.Mechanisms of epileptogenesis: a convergence on neural circuit dysfunction.Blockade of the sodium calcium exchanger exhibits anticonvulsant activity in a pilocarpine model of acute seizures in ratsProgressive dendritic HCN channelopathy during epileptogenesis in the rat pilocarpine model of epilepsy.Intrinsic neurophysiological properties of hilar ectopic and normotopic dentate granule cells in human temporal lobe epilepsy and a rat model.Cortical up state activity is enhanced after seizures: a quantitative analysisEffects of eslicarbazepine acetate on acute and chronic latrunculin A-induced seizures and extracellular amino acid levels in the mouse hippocampusEslicarbazepine acetate for the treatment of focal epilepsy: an update on its proposed mechanisms of action.Dendritic ion channelopathy in acquired epilepsyIonic mechanism underlying recovery of rhythmic activity in adult isolated neuronsA seizure-induced gain-of-function in BK channels is associated with elevated firing activity in neocortical pyramidal neurons.Persistent sodium current drives conditional pacemaking in CA1 pyramidal neurons under muscarinic stimulationAn acquired channelopathy involving thalamic T-type Ca2+ channels after status epilepticus.Double Trouble? Potential for Hyperexcitability Following Both Channelopathic up- and Downregulation of I(h) in Epilepsy.Serotonin Regulates the Firing of Principal Cells of the Subiculum by Inhibiting a T-type Ca2+ Current.Contributions of T-type calcium channel isoforms to neuronal firing.High-frequency oscillations and other electrophysiological biomarkers of epilepsy: underlying mechanismsCortical inhibition, pH and cell excitability in epilepsy: what are optimal targets for antiepileptic interventions?Low threshold T-type calcium channels as targets for novel epilepsy treatments.The Role of Calcium Channels in Epilepsy.Transcriptional regulation of T-type calcium channel CaV3.2: bi-directionality by early growth response 1 (Egr1) and repressor element 1 (RE-1) protein-silencing transcription factor (REST).A Cav3.2/Stac1 molecular complex controls T-type channel expression at the plasma membrane.T-type Ca2+ channels encode prior neuronal activity as modulated recovery rates.
P2860
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P2860
Upregulation of a T-type Ca2+ channel causes a long-lasting modification of neuronal firing mode after status epilepticus.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Upregulation of a T-type Ca2+ ...... mode after status epilepticus.
@en
Upregulation of a T-type Ca2+ ...... mode after status epilepticus.
@nl
type
label
Upregulation of a T-type Ca2+ ...... mode after status epilepticus.
@en
Upregulation of a T-type Ca2+ ...... mode after status epilepticus.
@nl
prefLabel
Upregulation of a T-type Ca2+ ...... mode after status epilepticus.
@en
Upregulation of a T-type Ca2+ ...... mode after status epilepticus.
@nl
P2093
P1476
Upregulation of a T-type Ca2+ ...... mode after status epilepticus
@en
P2093
Albert Becker
Hailing Su
Heinz Beck
Yanwen Jiang
Yoel Yaari
P304
P356
10.1523/JNEUROSCI.22-09-03645.2002
P407
P577
2002-05-01T00:00:00Z